How does disinhibition relate to aura and hallucinatory experience?

What factors may predispose someone to experience neural disinhibition and aura?

Not all instances of disinhibition result in sensory hallucination – why?

What is stable processing and what can disrupt this?

What are the two types of sensory hallucination as defined by Kluver (1966) and Siegal (1977) ?

What is Cowan's computational model of form constants and what did it attempt to address? What are its strengths and limitations?

What is the difference between sensations and perception?

How does Emmert's Law (1881) show how perception goes beyond sensation in the context of afterimages?

What is the difference between illusions, hallucinations and delusions?

What is the Spreading Cortical Depression Model? MRI evidence? What clinical cases studies are associated with this?

What evidence is there for less-inhibited brains in migraine?

What is Pattern-Glare/Visual stress and how can it be treated?

What are the different types of seizures? And what are the most seizure-prone brain areas?

What is the evidence for the association between complex seizures and high order hallucinations?

What is Charles-Bonnet Syndrome and how do brain-imaging studies show that patients are 'truly seeing'?

What are the two theories for CBS?

What is the medical model view of hallucinations? What are the problems with this?

What is the continuum view of hallucination? Examples of continuums?

Strengths and limitations of continuum view?

What is the Attributional theory of H/D (Bentall, 1990; Slade & Bentall, 1988)? Three main pieces of evidence?
*Limitations

How does source monitoring and reality monitoring link? What are two examples of failures in source monitoring?