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Erstellt von gina_evans0312
vor mehr als 12 Jahre
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| Frage | Antworten |
| Family Hypercholesterolemia- Fault | Faulty ApoB/E receptor gene |
| Family Hypercholesterolemia- Effect on Lipoproteins | Inc LDLs and Remnant particles |
| Family Hypercholesterolemia- Effect on Systems | Effects fuel transport and the overflow pathway |
| Family Hypercholesterolemia- Effect on body | Inc risk of cardiovascular disease |
| Family Combined Hyperlipidemia- Fault | Overproduction of ApoB100 & reduced LPL function |
| Family Combined Hyperlipidemia- Effect on Lipoproteins | Inc VLDL & LDL production |
| Family Combined Hyperlipidemia- Effect on Systems | Overloads fuel transport and overflow pathway |
| Family Combined Hyperlipidemia- Effect on Body | Inc risk of heart problems |
| Family Dysbetalypoproteinemia- Fault | Caused by ApoE mutation with low ApoE/B receptor affinity |
| Family Disbetalypoproteinemia- Effect on Lipoproteins | Remnant particles accumulate |
| Family Disbetalypoproteinemia- Effect on body | Risk of early coronary diseae |
| Type II Diabetes- Fault | Effects fuel transport proteins |
| Type II Diabetes- Effect on Lipoproteins | Inc. VLDL synthesis & inc Remnant particles, dec HDL cholesterol |
| Type II Diabetes- LDL Cholesterol | Usually normal |
| Metabolic Disorder- Fault | Glucose intolerance and low HDL levels |
| Metabolic Disorder- Effect on Body | Abdominal obesity, hypertension & coronary disease |
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