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Created by Evian Chai
about 4 years ago
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Electrical signals transmit through the heart from the ....cardium to the ...cardium
What is the conduction pathway through the heart, starting from the SA node?
Excitation Contraction coupling refers specifically to
When an AP arrives at the T-tubule, which channels open? What happens after?
How is Ca2+ reuptaken?
In the first phase of depolarisation in the pacemaker potential (SA node), why is depolarisation so slow?
What initiates spontaneous depolarisation in the SA node?
What channel is responsible for depolarisation (phase 0) of Pacemaker potential?
What channel is responsible for repolarisation (phase 3) of Pacemaker potential?
What causes depolarization (phase 0) of the ventricular action potential?
After depolarisation (phase 0) in the ventricular action potential, why is there a partial repolarisation?
After partial repolarisation (phase 1) in the ventricular action potential, why is there a plateau (phase 2)?
What eventually causes repolarisation (phase 3) in the ventricular action potential?
What is phase 4?
What is the ventricular action potential?
What is the unique feature about its shape and why is it important?
What is the rate of distribution of AP in the heart determined by? (2)
How does AP distribute through myocytes?
How do ionotropic factors (eg. noradernaline, B agonist) affect HR?
How does noradernaline affect HR?
How do B-adrenergic agonists impact HR?
How does ischemia hypoxia impact the heart?
What do chronotropic drugs do?
What is an example of a sympathetic drug?
What about examples of parasympathetic drugs?
How do sympathetic chronotropic drugs increase HR?
What is the Staircase/Trepple Effect?
How do parasympathetic chronotropic drugs decrease HR?
