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Ophtalmology Mind Map on Neuro-opthalmology, created by LewisLewis on 07/10/2014.

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Created by LewisLewis over 5 years ago
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1 Visual loss
1.1 Amsler grid
1.1.1 In maculopathies the lines appear curved or broken (metamorphopsia)
1.2 Pinhole is used to test visual acuity
1.2.1 Cataract and keratoconus improve with the pinhole, while optic neuritis doesn't
1.3 Color vision test helps you identify lesions at the level of the macula, of the optic nerve or at the chiasm
1.4 Causes
1.4.1 Optic neuropathy
1.4.2 Eye disease
1.4.3 Lesion in the intracranial visual pathway
1.5 Monocular visual loss results from lesions anterior to the chiasm, so to the optic nerve or to the eye itself; binocular visual loss results from lesions at the level of the chiasm or posterior to the chiasm
1.6 Visual field testing is helpful to localize and identify diseases affecting the visual pathways
1.6.1 You can test the visual field very simply with confrontation in the office or with automated stated perimetry
1.7 The relationship between the retina and the visual field is opposite and reverse
1.8 Span of the visual field
1.8.1 60° superiorly, 75° inferiorly
1.8.2 60° on the nasal side, 100° on the temporal side
1.9 Automated static perimetry
1.9.1 The most quantitative and sensible and reproducible technique to detect visual field deficits
1.10 4 rules to help us to understand better the location of the defects
1.10.1 The more congruous the defect, the more posterior the lesion
1.10.2 In chiasmatic lesions you always have bitemporal visual defects, also know as tunnel vision
1.10.3 In posterior lesions you can pretend your back head is against retina in order to locate the lesion
1.10.4 Occipital lesions spare the macula Occipital part in fact presents 2 different vascular supplies: posterior and medial cerebral artery
1.11 Monocular lesions anterior to the chiasm do not respect the vertical meridian
1.12 Binocular lesions instead respect the vertical meridian
1.13 Tools needed for a neuro-opthalmic examination at bedside
1.13.1 Near card to check visual acuity
1.13.2 A pair of reading glasses
1.13.3 A pinhole
1.13.4 A red object
1.13.5 A striped ribbon or paper to test optokinetic nystagmus
1.13.6 An Amsler grid
1.13.7 Short-lasting dilating drops
1.13.8 A direct opthalmoscope
2 Diplopia
2.1 Due to alterations in the movement of the eye
2.1.1 Maddox rod test is used to recognize small differences in motility
2.2 Monocular diplopia is not related to neurological disorders, but to optical problems
2.3 Binocular diplopia could result from
2.3.1 Extraocular muscle disordes CN lesion (III, IV, VI) Thyroidal disease can involve these muscles Others Inflammatory disorders Tumors Infections Orbital venous congestion Trauma Giant cell arteritis (Horton's) Progressive myopathies
2.3.2 Neuromuscular junction disease Myasthenia gravis 50% of patients present with diplopia or ptosis You can test with the rest test or eye-pack test Pupils are never involved
2.3.3 Inter/supra-nuclear pathways disease
3 CN III palsy
3.1 Etiology
3.1.1 Ischemia Check if patient is diabetic or has hypertension It occurs in the deeper part of the nerve, sparing the superficial parasympathetic fibers If we find an enlarged pupil, ask for MRI or angiography to identify compressive site If no pupil enlargement, check the BP or the glucose
3.1.2 Aneurysmatic compression
4 CN VI palsy
4.1 Always check intracranial pressure
4.1.1 The nerve is very susceptible when it enters the cavernous sinus, where it tilts of 90°
4.2 Patients usually tilt the head in order to avoid double vision
5 CN IV palsy
5.1 Typically patients, to compensate for the diplopia, tilt the head away from the lesion
5.1.1 Hardest palsy to recognize
5.2 Causes
5.2.1 1/3 ischemical
5.2.2 1/3 congenital Especially in children
5.2.3 1/3 due to injuries or tumor compression Especially in elderly
6 Multiple nerve palsy
6.1 Typically in cavernous sinus and orbital apex
6.1.1 Cavernous sinus syndrome Opthalmoplegia (multiple cranial nerve palsies) Horner Sd (sympathetic) Pain (trigeminal nerve) Proptosis and periorbital edema (if venous hypertension)
6.1.2 Orbital apex syndrome Ophthalmoplegia (multiple cranial nerve palsies) Horner Sd Pain (trigeminal nerve) Visual loss (optic neuropathy) – not present in cavernous sinus syndrome)
7 Anisocoria
7.1 Unequal size of the pupils
7.2 It can reveal a serious problem, like aneurysmatic compression in CN III palsy
7.3 We should always check the pupils in the light, in the dark, near and at a distance
7.4 Pupils are controlled by a steady balance between parasympathetic (CN III) constriction and sympathetic dilation
7.5 20% of people have physiologic anisocoria
7.5.1 Always the same both in light and in the dark and it may switch side or go away
8 Myosis
8.1 Sympathetic defect
8.2 Horner syndrome
8.2.1 Carotid dissection is the most important cause
8.2.2 Traumatic causes (severe neck injury)
8.2.3 Spontaneous (trivial events)
8.2.4 Most frequent cause of myosis, if you can exclude ocular problems or pharmacological problems
8.2.5 Characterized by unilateral myosis with dilation lag in the dark, mild ptosis due to Muller muscles paralysis and by pseudoenophthalmos
8.3 Pharmacological test can be done to localize the lesion (not to make the diagnosis)
8.3.1 Apraclonidine helps to differentiate preganglionic from post-ganglionic lesions In post-ganglionic lesions it dilates the normal pupil, while it doesn't affect the damaged pupil In pre-ganglionic lesions it creates an inverted anisocoria where the affected pupil dilates more than the normal one
9 Mydriasis
9.1 Most common causes
9.1.1 Tonic pupil (Adie pupil) Most common parasympathetic palsy It features acute denervation (injury to short ciliary nerve, pupil and accommodation fibers) and aberrant reinnervation (accommodative fibers innervate iris sphincter)
9.1.2 CN III palsy
9.2 Associated situations
9.2.1 Unilateral mydriasis
9.2.2 Loss of accommodation
9.2.3 Better constriction at near
9.2.4 Sectorial palsy of iris sphincter
9.2.5 Slow tonic redilation
9.2.6 Supersensitivity to pilocarpine (parasympathomimetic) (0.1%) We can exploit this for diagnostic purposes If myosis at 0.1: Adie pupil If myosis at 1: 3rd nerve palsy If myosis at 2.5: pharmacological midriasis (e.g. cocaine)
10 The blind spot is in the temporal zone
11 Optic neuritis
11.1 Inflammatory, infective or demyelinating process affecting the optic nerve
11.2 Classification
11.2.1 Opthalmoscopic classification Retrobulbar neuritis Most frequent type in adults and frequently associated with MS Papillitis Most common type in children Neuroretinitis Painless unilateral visual impairment which starts gradually and then becomes severe after about a week
11.2.2 Etiological classification Demyelinating Causes Isolated optic neuritis MS (most common) Devic disease (neuromyelitis optics) Schilder disease Treatment Intravenous methylprednisolone Intramuscular interferon beta-1a Most common Parainfectious Optic neuritis may be associated with various viral infections Infectious Sinus-related Cat-scratch fever Syphilis Lime disease Cryptococcal meningitis VZV Non-infectious Sarcoid Autoimmune
11.3 Non-arteritic anterior ischemic optic neuropathy
11.3.1 Caused by occlusion of the short posterior ciliary arteries resulting in partial or total infarction of the optic nerve head
11.4 Arteritic aterior ischemic optic neuropathy
11.4.1 Caused by giant cell arteritis Granulomatous necrotizing arteritis with a predilection for large and medium-sized arteries
11.4.2 Presentation with sudden unilateral visual loss which may be accompanied by periocular pain
11.4.3 Treatment Methylprednisolone Antiplatelet therapy Immunosuppressives