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When you increase CHO what happens to muscle glycogen throughout exercise?
What is the difference between trained athletes and untrained athletes with regards to glycogen.
How do trained individuals replenish glycogen quicker?
What is GLUT-4?
How does pre exercise muscle glycogen content and exercise intensity effect fuel metabolism?
Describe the testing.
How does pre exercise muscle glycogen content and exercise intensity effect fuel metabolism?
What were the findings?
What are the potential underlying mechanisms?
How does the regulation of glycogen phosphorylation work?
How does maintaining a low CHO diet work?
What is exercise induced activation of PSH attenuated with?
What effect does glycemic index have?
Why does high GI have that effect?
Why does low GI have that effect?
What do reduced plasma FFA and glycerol suggest is happening to lipolysis?
Lipolytic suppression following CHO ingestion limits fat oxidation during exercise.
What happens when we increase suppression of lipolysis
Lipolytic suppression following CHO ingestion limits fat oxidation during exercise.
What are happens to levels of glucose and fructose?
Lipolytic suppression following CHO ingestion limits fat oxidation during exercise.
When fasted what happened?
When fed what happened?
Lipolytic suppression following CHO ingestion limits fat oxidation during exercise.
When fed and lipids are infused what happens?
In the fructose trial what happened?
If lipid infusion does not restore fat oxidation rates to those that occur in the fasted state, where else can that attenuation occur?
What does reduced carnitine availability, caused by high PDH flux, limit?
What does an increase in acetyl-carnitine lead to?
What does a decrease in free carnitine lead to?
What happens when the body is subjected to a constant high fat diet?
When a time trial was completed, what was the difference between the fat adapted trial and the high CHO conditions.
During 2 hour exercise, what happens to fat oxidation compared to CHO or fat adapted conditions?
What does increasing CHO do to the flux of PDH?
What is endogenous carbohydrate availability referring to?
What is exogenous carbohydrate availability referring to?
Can the interaction of endogenous & exogenous CHO availability affect training-induced skeletal muscle adaptations to exercise?
Is breakfast before training really a good thing?
What is citrate synthase?
Is breakfast before training really a good thing?
What did measuring citrate synthase and beta-HAD show?
What does training low allow us to do?
What does competing high allow us to do?
Does exercise increase p53 phosphorylation?
Does reduced CHO availability affect the exercise-induced increase in p53 phosphorylation?
How?
What does upstream AMPK signalling lead to?
CHO availability can modulate fatigue. What can deliberate training in conditions of reduced CHO availability allow?
What does strategically periodising out training and nutrition allow us to do?
