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Quiz

203: LECT 8

Kaaj Pala 11 Fragen

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  1. 1Which of the following is not associated with the cardinal signs of inflammation when responding to injury or infection?
    1. Aredness
    2. Bheat
    3. Cswelling
    4. Dpain
    5. Eincreased pH
  2. 2Inflammation provides the cell in many ways:
    -delivering nutrients and [blank_start]oxygen[blank_end] to injured sites.
    -generates an exudate (increased tissue fluid) that [blank_start]carries toxins[blank_end] from affected area via [blank_start]lymphatic drainage[blank_end] to local lymph node where they stimulate an [blank_start]immune response[blank_end]
    -generates exudate that carries [blank_start]antibodies[blank_end] and other substances to affected areas to neutralise harmful agents
    -work force to remove [blank_start]debris[blank_end] after injury
    -[blank_start]defence[blank_end] force to remove harmful agents
    -limits spread of harmful agents via [blank_start]fibrin mesh[blank_end] and abscess walls
    -provides [blank_start]hydrolases[blank_end] to digest inflammatory exudates after crisis
    -initiates repair
    1. Aoxygen
    2. BCO2
    3. Ciron
    4. Dcarries toxins
    5. Ebacteria removal
    6. Flysosomes
    7. Glymphatic drainage
    8. Hblood vessels
    9. Iantibody pathway
    10. Jimmune response
    11. Kacute response
    12. Lelongated antibody response
    13. Mantibodies
    14. Npathogens
    15. Otoxins
    16. Plymphocytes
    17. Qdebris
    18. Rextra contents
    19. SICF fluid
    20. Tdefence
    21. Uattack
    22. Vteam
    23. Wfibrin mesh
    24. Xantibody barriers
    25. Yimmune activity
    26. Zhydrolases
    27. AAlysosome
    28. ABantibody
  3. 3Which of the following is not associated with tissue damage signals that induce inflammation?
    1. ANeurons release bioactive peptides in response to pain
    2. BBroken cells release intracellular molecules that signal presence of damage
    3. CMicrobial products i.e. endotoxin/LPS recognised as PAMPs by PRRs
    4. DLipid derived signals
  4. 4Is it true that PRR's are not soluble and are not cell bound (TLRs)?
    1. ARichtig
    2. BFalsch
  5. 5Tissue resident [blank_start]mast cells[blank_end] (or basophils) and [blank_start]macrophages[blank_end] rapidly respond to these signals. Mast cells are full of [blank_start]granules[blank_end] (storage organelles) and initiate inflammation by released mediators such as histamine, proteases (tryptases), lipid derived signals and [blank_start]cytokines[blank_end] such as TNF. These signals cause rapid microvascular change and the 4 cardinal sign process -redness-heat-swelling-pain response.
    1. Amast cells
    2. Bneutrophils
    3. Cleukocytes
    4. Dmacrophages
    5. Elymphocytes
    6. Fsatellite cells
    7. Ggranules
    8. HICF contents
    9. Iamino acids
    10. Jcytokines
    11. Kneurons
    12. Llymph nodes
  6. 6Hyperaemia is a result of vasodilation?
    1. ARichtig
    2. BFalsch
  7. 7Is it true that most blood vessel are lined by endothelial cells?
    1. ARichtig
    2. BFalsch
  8. 8Vascular permeability [blank_start](leakiness)[blank_end] occurs when the endothelial cells (ECs) retract to create gaps .
    -[blank_start]Mild injury[blank_end] i.e. insect bites, inflammatory signals cause venular ECs to [blank_start]rapidle and reversibly retract[blank_end], creating gaps between cells- where protein rich fluid (inflammatory exudate) can pass.
    -Serious injury leads to EC damage, [blank_start]detachment from basement membrane[blank_end], increases in vascular permeability.
    These may include delayed (sunburn, bacterial toxins) with leakage from capillaries and venules
    OR immediate (burns, trauma) with leakage from [blank_start]all types of vessels[blank_end] until vessel is blocked with clot or repaired.
    1. Aleakiness
    2. Bresistance
    3. Cpressue
    4. DMild injury
    5. ETrauma
    6. FNeuro disease
    7. Grapidly and reversibly retract
    8. Hincrease vessel pressue
    9. Idecrease in diameter
    10. Jdetachment from basement membrane
    11. Kincreased attachment to the ECM
    12. Lrelease of toxins
    13. Mall types of vessels
    14. Njust venules
    15. Oonly capillaries
  9. 9As a result of vascular permeability (microvascular leakage) inflammation occurs:
    Fluid is forced out of capillaries from [blank_start]arteriolar side[blank_end] (by hydrostatic pressure) and re enters at venular side (due to [blank_start]plasma proteins bind to water[blank_end]). During inflammation:
    -Increased hydrostatic pressure and increased water binding capacity of proteins in exudates leads to [blank_start]retention of fluid[blank_end] in the interstitium (extravascular space between cells).
    -Accumulated fluid causes [blank_start]swelling or oedema[blank_end] (build up of fluid in body)
    -Stretching of tissue with [blank_start]chemical mediators[blank_end] causes pain.
    1. Aarteriolar side
    2. Bvenule
    3. Cby joint
    4. Dplasma proteins bind to water
    5. Eexcess fluid build up
    6. Fmore leakage occurs
    7. Gretention of fluid
    8. Hloss of fluid
    9. Iincreased blood pressure
    10. Jswelling or oedema
    11. Khead injury
    12. Ltrauma
    13. Mchemical mediators
    14. Nreceptors
    15. Otoxin
  10. 10Signals involved in controlling blood vessel function during inflammation are not secreted by cells but are ONLY derived from plasma proteins?
    1. ARichtig
    2. BFalsch

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Beschreibung
Acute Inflammation: Blood flow
Sprache
English
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