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Karteikarten

Stroke, Cerebral Palsy and Epilepsy

Morgan Morgan Medicine, Surgery & Radiology University Englisch 44 Karten

Karten in diesem Stapel (44)

  1. 13 main types?Thromb-embolic
    Cerebral and celebellar haemorrhage
    Subarachnoid haemorrhage
    Hemiplegia following middle cerebral arterial thromboembolism is the typical example
  2. 2DefinitionA syndrome of rapid onset of cerebral defecit (usually focal) lasting > 24hours or leading to death, with no cause apparent other than a vascular one
  3. 3Completed StrokeThe defecit has become maximal – usually within 6 hours
  4. 4Stroke in evolutionDescribes progression during initial 24 hours
  5. 5Minor StrokePatients normally recover within 7 days, with no significant defecit
  6. 6Transient Ischaemic AttackA sudden focal deficit lasting from seconds to 24 hours with complete recovery
  7. 7Causes (3)Arterial embolism
    Arterial thrombosis
    Both classified as “Ischaemic”
    Haemorrhage
  8. 8Thromboembolic Strokes are the most common and account for what %?80%
  9. 9ThrombusA thrombus is a blood clot that forms in a vessel and remains there
  10. 10EmbolismAn embolism is a clot that travels from the site where it formed to another location in the body.
  11. 11Risk FactorsHypertension
    Smoking
    Lifestyle
    Alcohol
    High Cholesterol
    Raised haemocrit
    Atrial fibrillation
    Obesity
    Diabetes
    Severe carotid stenosis
    Sleep apnoea
  12. 12Hypertensionincreased pressure in arteries – tunica intima damage – atherosclerosis – thrombus – embolism risk
    Managed by anti hypertensive drugs
  13. 13Diagnosis 7Physical symptoms
    CT Scan or MRI
    Catheter ultrasound – carotid arteries
    Swallow Test
    Blood Tests
    Chest X-Ray
    ECG
  14. 14Physical symptoms 6speech, limb weakness, hemiplegia, hemiparesis , facial problems. Limbs initially flacid and are flexic – over time – several days – reflexes return – spastic muscle paralysis
  15. 15Hemiplegia(paralysis of arm/leg one body side)
  16. 16hemiparesis(weakening – less severe than hemiplegia),
  17. 17Ct and MRIshow type of stroke – area of brain – extent of damage
    Very important for – those who would benefit from thrombyolytic treatment – already on anti-coags – low level of consciousness
  18. 18Swallow1/3 stroke victims difficulty swallowing
  19. 19Immediate managementAdmit to multidisciplinary stroke unit
    General medical measures
    Airway: confirm patency and monitor
    Continue care of the unconscious or stuporose patient
    Oxygen by mask
    Monitor BP
    Look for source of emboli
    Assess swallowing
  20. 20Is thrombolysis appropriate?If so immediate brain imaging is essential.
  21. 21Further management (4) drugsAnti-hypertensives
    Anti-platelet agents
    Endartectomy
    Possible anticoagulants
  22. 22Further management 5Speech therapy
    Dysphagia care
    Physiotherapy
    Occupational therapy
    Specific issues
    Epilepsy
    Pain
    Incontinence
  23. 23Treatment - Haemorrhagic Stroke 4 H,T,H,RHaematoma evacuation
    Thrombolysis
    Haemostatic therapies
    Reducing inter-cranial pressure
  24. 24Prognosis Ischaemic? Haemorrhagic?Ischaemic
    Early mortality – 20%
    Independent @ 6mths – 60%
    Recurrance @ 1 mth – 10%
    Recurrance @ 3 mths – 18%
    Haemorrhagic
    Early mortality – 60%
    Independent @ 6mths – 20%
    Recurrance – 2% per annum
  25. 25Cerebral PalsyChronic disorder of posture and movement caused by a non progressive lesion sustained by the developing brain.
    The damage is irreversible and produces life long disabilities which can be progressive
  26. 26It is not one single entity but is a collection of disorders with a common presentation Often linked with ........ incapacity (?%) and seizures Prevalence is 2 per 1000 childrenMental (35%)
  27. 27Risk factors – pre-natal (5) S, A, M, P, GSmoking
    Alcohol and drugs
    Maternal infection
    Placental insufficiency
    Genetic issues
  28. 28Risk factors – peri-natal (4) P,F,H,IPremature and low birth weight babies
    Foetal distress
    Hypoglycaemia
    Infections - meningitis
  29. 29Risk factors – post-natal 2 T, ITrauma – intra-ventricular haemorrhage
    Infection – meningitis
    10 % of all CP cases
  30. 30Cerebral Palsy - classification (3)Spastic
    Athetoid
    Ataxic
  31. 31Spastic - 4 further classificationsTopographically – by the area affected
    Hemiplegia
    Diplegia
    Quadriplegia
  32. 32The most common form of CP is spastic, where the muscles are? There is ..........muscle tone and..........reflexes. It is usually detected after? What may occur?Tense and contracted
    Increased muscle tone and spinal reflexes
    Usually detected after the 1st year
    Spasticity does not resolve
    Tremors may occur
  33. 33Athetoid CP Coordination for ............... is poor Worse during periods of ?constant, uncontrolled movement of the limbs, head and eyes.
    Coordination for speech is poor
    Worse during periods of stress
  34. 34Ataxic CP - 4Affects balance and depth perception
    Poor coordination, often causing falls
    Intention tremor
    Difficulty with quick or precise movements
  35. 35CP common features - 4Half are of normal or above average intelligence
    One third have epilepsy
    25 – 80% have speech problems
    A quarter will be fully independent adults
  36. 36Outcomes 4Almost all hemiplegic children walk
    75% of diplegic children walk
    Children who can sit at 2 years will probably walk
    Not walking by 7 years is a poor prognosis.
  37. 37Equinus Gait in CPPatient walks on tip toes.
  38. 38Epileptic seizureAn intermittent convulsion or transient event due to sudden activity of cerebral neurones.
    All cease spontaneously
    Epilepsy is a continuing
    tendency to have such
    seizures – even if long
    time intervals occur
  39. 39Seizures occur because of a disturbance in the brain that gives rise to: 4 M, S, B,Caltered movement
    disturbance of sensory function
    altered behaviour
    altered consciousness
  40. 40Aetiology - 8Birth injury
    Cerebral tumours
    Trauma
    Cerebral Malformation
    HIV encephalopathy
    CVI
    Infection
    Metabolic disorder
    Idiopathic
  41. 41Classification Partial - 2 Generalised - 3Partial:
    Simple partial (Jacksonian motor epilepsy)
    Complex partial
    Generalised:
    Typical absence (Petit Mal)
    Tonic-clonic (Grand Mal)
    Atonic (Drop attacks)
  42. 42Management 2Long term anticonvulsant drugs
    E.g. Phenytonin, Carbamazepine, Sodium valproate, Vigabatrin, Gabapentin
    Surgical management is sometimes possible:
    Removal of the anterior temporal lobe
  43. 43Emergency TreatmentStay calm & reassure
    Ensure the patient harms themselves as little as possible.
    Maintain an open airway if possible
    Cushion the head
    Loosen restrictive clothing around the neck
    Place in the recovery position when possible
    If hypoglycaemia is suspected give glucose
    If possible let them sleep
  44. 44Status EpilepticusChain of seizures following one another without recovery
    Epileptic attack maintained for more than 60 mins in an adult and 30 min in a child
    Medical emergency
    Mortality 10 – 15%
    (cardio-respiratory failure)

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Beschreibung
University Medicine, Surgery & Radiology Karteikarten am Stroke, Cerebral Palsy and Epilepsy, erstellt von Morgan Morgan am 28/12/2014.
Erstellt von
Morgan Morgan
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