Karteikarten
Stroke
Karten in diesem Stapel (25)
- 1Blood flow to the brain750mL/min
20% of blood volume circulates through the brain - 2Brain regulating mechanisms we can influenceCarbon dioxide concentration - too low causes vasoconstriction
Oxygen concentration - hyperoxygenation causes oxidative stress, free radicals, - 3Define embolic/ischaemic strokeAn episode of neurological dysfunction caused by cerebral, spinal or retinal infarction
- 4Most commonly affected vessels1. Middle cerebral artery
2. Posterior cerebral artery
3. Basilar artery
4. Vertebral artery
The larger the artery affected, the greater the affected area and associated deficit - 5Pathophysiology of ischaemic stroke1. Plaque ruptures, or tumour, fat globules, air or amniotic fluid
2. Thrombus formation, typically at origin or bifurcation of arteries
3. Plaque formation with fibrous fab -> degradation of cap by macrophages, initiation of thrombus formation - 6Causes of embolism- Vascular stasis
- Stenotic vessels
- AF
- AMI
- Aneurysms
- Thromboembolism
- Gas embolism
- Amniotic fluid embolism during child birth
- Fat embolism in major fractures - 7Virchow's triad- Hypercoagulability
- Haemodynamic changes - stasis, turbulence
- Endothelial injury/dysfunction - 8Large vessel occlusion, vessels affected- <20% of AIS
- Most severe stroke with worst prognosis
- Middle cerebral artery
- Internal carotid artery
- Basilar arteries - 9Arterial dissection facts- 20% of AIS <25 years
- Females 3x more likely than males
- Chiropractors and hairdressers dangerous
- Neurological symptoms and a sore neck -> arterial dissection until proven otherwise - 10Risk factors for embolic stroke (7)- AF
- Endocarditis
- Dilated cardiomyopathy
- Recent MI
- Heart valve replacement
- Rheumatic heart disease
- Patent foramen ovale - 11Risk factors for thrombotic stroke (7)- Hypertension
- Hyperlipidaemia
- Diabetes
- Smoking
- Obesity
- Systemic atherosclerosis
- Hypercoagulable state - 12Intracerebral haemorrhagic stroke pathophysiologyRapidly developing clinical signs of neurological dysfunction attributable to a focal collection of blood within the brain parenchyma or ventricular system that is not caused by trauma
- 13Subarachnoid haemorrhagic stroke pathophysiologyRapidly developing clinical signs of neurological dysfunction and/or headache because of bleeding into the subarachnoid space, which is not caused by trauma
- 14Haemorrhagic stroke presentation- Severe sudden headache in 95% of victims, often occipital lobe
- 50% have a warning leak, mild headache
- Upper neck pain -> inflammation of CSF
- Photophobia
- Nausea and vomiting
- Hypertension
- Bradycardia
- Herniation - late stage - 15Other "stroke"An episode of acute neurological dysfunction presumed to be caused by ischaemia or haemorrhage persisting >24 hours or until death, but without sufficient evidence to be classified as one of the above
- 16Transient ischaemic attack (TIA), pathophysiology and relation to stroke predictionNeurological deficit that resolves within 24 hours
1 in 2 TIAs will progress to stroke. 1/2 within 12 months - 17Stroke epidemiology- 80% of strokes can be prevented
- one occurs every 10 mins in Aus
- One of Australia's biggest killers and cause of disability - 18Non-modifiable risk factors for stroke- Age
- Gender
- Family history of stroke - 19Modifiable risk factors for strokeTIA
Hypertension
Smoking
High cholesterol
Poor diet and lack of exercise
Obesity
Diabetes
Alcohol
AF - 20Lateral pointine syndrome- Presents scarily similar to narcotic overdose
- Basilar artery
- Pinpoint pupils
- Respiratory depression
- No response to naloxone - 21Locked in syndrome- Basilar artery
- 22Five aspects of neurological assessmentLOC
Motor function and tone
Sensory function
Speech
Pupillary response - 23Differential diagnoses of strokeIntracranial space-occupying lesion
Subdural haematoma
Multiple sclerosis
Head injury
Hypoglycaemia
Seizure disorders – Todds paresis (one sided paralysis -> resolves)
Complex migraine - 24Endovascular clot retrievalMost benefit for large vessel occlusion
Up to six hours post onset of symptoms
Posterior circulation stroke up to 24 hours due to poor prognosis - 25Pre-hospital stroke caresOxygen - causes oxidative stress
Head position - semi-recumbent is haemorrhagic, supine is ischaemic
Aspirin - can't tell if clot
Magnesium - no benefit found
Informationen
- Beschreibung
- Paramedics (CSB335) Karteikarten am Stroke, erstellt von Amelia Tuffley am 11/11/2018.
- Erstellt von
- Amelia Tuffley
- Aufrufe
- 6
- Tags
- Bewertung
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