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Developmental Abnormalities (path 2)

Katie Hayes SP D2 Englisch 38 Karten

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  1. 1febrile illnessbilateral
    symmetric
    horiz zones of hypoplasia at chronological time of illness
  2. 2turners hypoplasiadefects in adult dentition due to localized (non-systemic) disruption due to inflammation or trauma of primary dentition
    typically affects adult PM
    trauma-->opacity
    inflamm-->hypoplasia
  3. 3radicular hypoplasiasig root resorption due to radiation tx
    also assoc with xersotomia and caries
  4. 4fluorosisexcessive fl during development-->white, chalky hypomineralized areas
    retained amelogenin (enamel matrix prot)
    incr caries resistance
  5. 5congenital syphillisblindness, deafness, dental hypoplasia (CN8 affected)
    hutchinson's incisors and mulberry molars
  6. 6attritionloss of tooth structure due to tooth-tooth contact during occlusion
    flattened squared off look
  7. 7abrasionloss of tooth structure due to action of external agent (non masticatory)
    ie. toothbrush
  8. 8abfractionfracture of tooth structure due to occlusal stresses causing tooth flexure
    deep narrow, v-shaped lesions in cervical areas of lesions
    may resemble abrasion/erosion
  9. 9erosionnon-bact chem dissolution (food, drinks, meds, environ, gerd) of tooth structure
    includes perimolysis on linguals of max ant teeth
  10. 10internal root resorptionbegins in dental pulp
    usually asymptomatic
    granulation tissue due to necrotic tissue--> pink tooth of mummery
  11. 11external resorptionbegins in PDL
    usually asymptomatic
    due to trauma, inflamm, cysts, neoplasms, idiopathic, ortho
  12. 12impactioneruption ceases before emergence
    soft tissue or bony
  13. 13ankylosisfusion of tooth with alveolar bone
    cessation of eruption after emergence
    retained primary "submerged" teeth
  14. 14natal teethpresent at birth or first 30 days of life
    mand incisors most freq
    prematurely erup deciduous teeth (not supernumerary)
    retain if stable, remove if aspirate
  15. 15hypohydrotic ectodermal dysplasiamost common type of ED
    x-linked inheritance
    defective epidermal structures: hypodontia, hypotrichosis, hypohidrosis
    abnormal, conical teeth
    faint hair, midface hypoplasia
    possible anodontia
  16. 16congentially missing teethrare in primary dentition (mand incisors)
    common in perm dentition (3M, 2PM, max LI)
    least common: max CI, M1
  17. 17supernumerary teethmost common: max incisors, max 4M, mand PM
    mesiodens
    distomolar (4M)
    or paramolar (B or L to molar)
    seen in gardners and CCD
  18. 18Gardner SyndromeAD
    supernumerary teeth
    osteomas and jaw densities
    intestinal polyps with 100% chance of malig transformation
  19. 19microdontiageneralized: rare (pituitary dwarfism, Down Syn)
    localized: common (peg lateral, 3M)
  20. 20macrodontiageneralized: rare (pit gigantism, hemifacial hyperplasia)
    localised: rare (mand 3M)
  21. 21relative macrodontia/microdontianormal teeth in small jaw causing crowding
    OR
    normal teeth in large jaw causing spacing
  22. 22geminationdevelops as one tooth than splits into 2 during devl
    tooth count is normal
  23. 23fusion2 teeth develop fused together
    tooth count is missing one
  24. 24concrescenceunion by cementum alone
    no confluence of dentin
    may be due to devl or inflamm
    may not be obvious on radio
  25. 25types of accessory cuspscusp of carabelli (ML cusp of max M1)
    dens evaginatus
    talon cusp
  26. 26dens invaginatus (dens in dente)deep surface invagination lined by enamel
  27. 27enamel pearlectopic enamel forming nodule
    may have dentin and pulp or just enamel
    causes PD and inflamm issues
  28. 28cervical enamel extensionextension of ectopic enamel on buccal surface of mand molars
    may cause buccal bifurcation cysts
  29. 29taurondontismelongated pulp chamber and shallow furcation
    can affect primary and perm
    sometimes seen in: AI, Ectodermal Dys, hypophosphatasia, Klinefelter, Down
  30. 30hypercementosisexcess deposition of secondary cementum
    more cellular than typical cementum (more like bone)
    may cause anklyosis
    localized due to inflamm or occlusal trauma
    generalized: paget's ds of bone
    can follow PDL all the way around
  31. 31dilacerationangulation or bend in a tooth root caused by displacement of tooth germ
  32. 32supernumerary rootsfound in 3M, mand C, mand PM most commonly
  33. 33AIhereditary ds with defective enamel formation
    14 subtypes including hypoplastic (most common), hypocalcified, hypomaturation
    mostly AD, some X-lined
  34. 34DIAD defective foramtion of dentin
    may be assoc with OI
    oaplescent teeth, soft dentin
    radio: cervical constriction, obliteration of pulp
    dentin next to enamel is normal, but remaining dentin has disoriented, irregular, and widely spaced tubules
  35. 35type I dentin dysplasiaAD
    radicular dentin dysplasia
    pulp obliteration with chevron shaped pulp chambers
    short and thin roots
    PARLs
    pulp chamber histo: 'water flowing around boulders'
  36. 36type II dentin dysplasiaAD
    coronal dentin dysplasia
    normal roots
    "thistle tube" pulp chambers and stones in coronal portion
  37. 37regional odontodysplasia"ghost teeth"
    localized devl abnormality that involved enamel, dentin, pulp
    idiopathic, poss due to alt in vascular supply
    primary or perm teeth
    affects a segment of the jaw, usually ant
    pale, whispy teeth thin enamel and dentin surr large pulp
    adjacent tissue filled with dystrophic calcs
  38. 38segmental odontomaxillary dysplasiadevl disorder
    unilateral enlargement of bone + gingiva of post maxilla with missing or abnormal teeth (PM missing or abnormal primary M)
    delayed eruption
    granular, RO bone without trabeculae

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SP D2 Karteikarten am Developmental Abnormalities (path 2), erstellt von Katie Hayes am 14/04/2020.
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Katie Hayes
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