Karteikarten
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Karten in diesem Stapel (27)
- 1Internal Derangement-Abnormal positional relationship of the disk relative to the mandibular condyle and the articular eminence
-Continuous pain localized to the TMJ during jaw movement
-Disc may be displaced anteriorly and return to normal position on opening of mouth or disc may act as an obstacle to the sliding condyle creating intermittent jaw locking limited opening and restrictive lateral excusrion-Morning jaw stiffness from bruxing or provoked by gum chewing - 2Hypermobility DisorderWhen one or both condyles translate anterior to the articular eminence resulting in locking.
-Can occur spontaneously or due to eating, yawning or during dental treatment. - 3DisloctationCorrected by placing downward and posterior pressure on the mandible to relocate the condyle within the fossa.
-Prolonged dislocation may require muscle relaxants or general anaesthetic - 4SubluxationHypermobility in which the patient can relocate the mandible backward into the glenoid fossa.
- 5AnkylosisImmobility of the condyle because of fibrous or bony fusion (complete or incomplete) between articulating structure of the joint. Joint infection after trauma accounts for 50% of ankylossed cases-most common in children because of greater osteogenic (bone producing) potential and undeveloped meniscus joint.
- 6NeoplasiaTumors are rare. Most common tumors are osteochondroma, osteoblastoma, chondroblastoma, osteoma. Symptoms include degenerative changes similar to osteoarthritis, swelling, pain, limited movement. Radiolucent or radiopaque findings and variable widening or loss of joint space.
-Malignant tumor=Osteosarcoma-Rare - 7Burning Mouth Disorder-Unexplained and continuous burning sensation of oral soft tissue
-Likely neuropathic etiology
-Onset is spontaneous
-Evaluation involves a careful work up
-Lab testing may reveal undiagnosed systemic disease like anemia and diabetes - 8Clinical features of Burning Mouth-Patient complains of burning and painful tongue
-Dorsal anterior and middle third of the tongue are particularly uncomfortable
-pain progresses throughout the day, worse at night
-2/3 of patients report altered taste and xerostomia - 9Trigeminal Neuraglia-Pathologic condition involving the fifth cranial nerve
-Characterized by unilateral sharp, shooting, knifelike, or electric shock-type pain
-Unknown etiology - 10Clinical Features of Trigeminal Neuraglia-50% of patients have trigger zone that will cause an episode if touched or temp change
-Pain lasts less than 2 minutes but patient experiences recurrent episodes of shooting, excrutiating pain
-Refractory period after episode where trigger cannot cause another episode
-More common in females mid 50's to 70's - 11Bell's Palsy-Acute, self-limiting loss of muscle control on one side of the face
-mostyl 15-45 year olds, women more - 12Clinical Feature-Presentation reflects the anatomic distribution of 7th cranial (facial) nerve
-May be unable to smile, close eye, raise eyebrow, corner of mouth droops causing drooling
-Speech affected
-Neck, mastoid and/or ear pain
-Dysguesia (taste change), hyperacusia (hearing disorder causing sensitivity to certain frequencies and volumes), altered facial sensation - 13Cyclic Neutropenia (Si-kulick)-cyclic decrease in number of circulating neutrophilic leukocytes (neutrophils) called neutropenia
-21-27 day cycles but may extend to several months
-2-3 day episodes
-Fever, fatigue, malaise, sore throat, infection
-Severe ulcerative Gingivitis/gingivostomatitis, ulcers on tongue and mucosa
-Ulcers vary in size, painful crater like, bleeding base, frequent secondary infections that improve when neutrophils return
-Eventually lead to severe perio disease and bone loss, mobility and premature tooth loss
-Determine cycle frequency via neutrophil counts and antibiotics for secondary infections
-Only treat when neutrophil count is normal
-ELA-2 gene - 14Chronic Neutropenia (Kostmann Syndrome)-Inherited autosomal recessive condition
-Not continuous or cyclic
-Same symptoms as cyclic neutropenia except constant and require systemic treatment
-G-CSF - 15Papillon-Lefevre Syndrome-Rare genetic disorder
-Marked destruction of periodontal tissue with premature tooth loss, hyperkeratosis of palms and soles of feet
-Palmar-plantar keratossi=scaly red lesions
-abnormal nails, excessive sweat, chronic, recurrent infections (colds, ear infections and skininfections)
-Normal at birth except scaly palms and soles
-Teeth erupt in normal sequence at 2 but gingivoperiodontal inflammatory process develops
-Edema, bleeding, alveolar bone resorption, mobile teeth, with consequent tooth loss (same sequence as eruption
-All teeth lost by 5
-Permanent teeth erupt as normal
-At 8-9 year gingivoperiodontal destruction repeats
-all teeth lost at 14 and gingiva appears normal again
-Marked bone loss and vertical pockets - 16Gingival Fibromatosis-Develops early in life, within few years teeth completely covered
-Firm, granular corrugated surface
-Fibrous connective tissue, paler i colour
-Causes lip protrusion
-Frequent gingivectomy/gingivoplasty
-Frequent visits to reduce inflammation and secondary infection
-component of other syndromes - 17Gingival Fibromatossi with Hypertrichosis-Excessive hair growth of eyebrows, extremities, genitals and sacral regions
-Epilepsy, MR syndrome - 18Gingival Fibromatosis with multiple Hyaline Fibromas-Gingival fibromatosis, hypertrophy of nail beds, multiple hyaline fibromas on nose, chin, head, back, fingers, thighs, and legs
- 19Cherubism-Autosomal dominant inheritance
-Excessive bone degredation of upper and lower jaws followed by fibrous masses forming which cause facial swelling
-Bone degradation begins around 3
-Multiple symmetrical cysts on max and mand filled with fibrous tissue consisting of stromal cells (CT) and osteoclast-like cells which cause swelling
-Initially present with progressive, bilateral facial swelling at 1-24 years
-Mandible most commonly affected
-When in max, infraorbital bones enlarge, eyes appear displaced and tilt upward
-Greater distance between eyes
-Jaws show soap bubble radiolucencies near molar/premolar or entire jaw-condyle if severe
-At 20-30 years bone looks normal in rads but microscopically appears as central giant cell granuloma
-Pseudodontia (teeth falsely appear missing)-delayed molar and pre-molar eruption
-Jaw increases rapidly until puberty but deformed for life - 20Ellis-Van Creveld Syndrome-Autosomal-recessive inheritance
-Dwarfism-shortened extremities
-1/3 mildly mentally disabled
-Polydactyl- extra digits on ulnar side
-Deformed nails
-Skeletal deformaties like curved legs
-50% have congenital heath defect
-"V-notch" appearance in midline of upper lip
-anterior max vestibule is lacking, lower anterior alveolar ridge thickened, central incisors lacking, conical teeth and enamel hypoplasia
-50% of neborns have natal teeth - 21Cleidocranial Dysplasia-Cliedo=collar bone, cranial=head, dysplasia=abnormal forming
-Short stature, narrow features
-Cranium develops a mushroom shape because fontanelles remain open making face seem small
-Frontal, parietal and occipital enlargement, underdeveloped paranasal sinuses, nose is flat and wide-lacks bridge, neck long and narrow, partial or absent clavicles so can fold shoulder in
-Multiole supernumerary teeth that delay eruption, crowding, pseudodontia, multiple cysts, narrow, high vaulted palate, cleft palate or lip in 1% affected patients
-May need ortho, dentures, extractions, corrective surgery - 22Gardner Syndrome-Facial and Oral Osteomas and Odontomas-Familial Colorectal Polyposis
-Rare dominant inheritance in which Adenomatous Polyposis Coli Gene is responsible
-Multiple adenomatous intestinal polyps malignant at 30
-Polyps are glandular and dangerous in stomach and small intestine
-Colon and rectum affected after puberty
-Polyps transform into adenocarcinomas with age
-Osteomas in various bone-when in facial skeleton expand to obliterate sinuses and create facial asymmetry
-Odontomas in jawbone, especially mandible
-tooth hyper cementosis and failure to erupt
-Multiple sebaceous cysts on scalp - 23Nevoid Basal Cell Carcinoma Syndrome-Gorlin Syndrome
-Autosomal Dominant causing oral, systemic and skeletal anomalies
-Nevoid basal cell carcinoma, jaw cysts, congenital skeletal anomalies, ectopic calcification, palmar and plantar pits
-Mild hypertelorism (increased eye distance), Pronathism (prominent mandible), frontal and parietal enlargement, broad nasal root
-Nevi (basal cell carcinomas) over eyelids, nose, cheeks, neck, arms and trunk-flesh coloured or plate brown singly or in clusters
-Palms and soles show small pits that fill with dirt and appear as dark spots
-Multiple Odontogenic Keratocysts which can move teeth and resorb them, develop at 5-6 years, recur after surgical removal
-Frontal bossing, cleft lip or palate, falx cerebri calcification, rib abnormalities, shortened metacarpals, spina bifida, medulloblastoma - 24Mandibulofacial Dysostosis-Treacher Collin SyndromeAutosomal dominant
-More severe as passed along
-60% spontaneous mutation
-eyes sloped down, lower lid notched, missing lashes, zygomatic arch is hypoplastic
-ears low set, malformed or missing, ear tags
-fish like mouth, downward commissure slope
-Hypoplastic mandible, flattened condyle, coronoid process, high palatal vault, 30% cleft lip/palate, severe malocclusion, open bite, wide interprox, displaced teeth, gingival disease
-Surgery ortho and implants - 25Osteogenesis Imperfecta-30% have family history, 70% represent sporadic cases so may be autosomal recessive
-Abnormal bone formation that fracture easily
-may have fractures at birth-4 types - 26Osteogenesis Imperfecta Type 1-Most common and mild
-Collagen improperly formed
-Blue sclera, 70% hearing impaired
-Normal or near normal stature-most most fractures occur before puberty
-Loose joint and weakened muscles, triangular face, spinal curvature, brittle teeth, dentinogenesis imperfecta
-80% primary teeth affected, 35% permanent teeth affected
-Crowns, roots, pulp chambers smaller than normal, teeth appear opalescent or translucent at eruption and then darken with age, enamel lost due to inadequate dentin support
-Normal life span - 27—
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- Karteikarten am Ohne Titel, erstellt von Hayden Mcbennett am 02/11/2020.
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- Hayden Mcbennett
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