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Week 8

Dolu Falowo Foundations 2 Inglés 50 fichas

Week 8

Fichas del conjunto (50)

  1. 1What is necrotizing fasciitis?-Bacterium feeds on fascia
    -Damage is spread along fascial planes
    -Can consume an entire person in hours
  2. 2The neck is divided into fascial compartments. Why?-Ease of movement during movement, swallowing etc.
    -Forming natural planes of tissue packing
    -Determine the spread of infection
  3. 3What happens if there are infections...? -Between the investing and pretracheal fascia -In the retropharyngeal space -In the head What prevents abscess spread?-Spread to thoracic cavity
    -Spread to superior mediastinum
    -Inferior spread, posterior to oesophagus
    Investing layer helps prevent abscess spread
  4. 4What are the 3 compartments of the thigh? Describe themAnterior: knee extensors. Contain quadriceps
    Posterior: knee flexors and hip extensors. Contain hamstrings
    Medial: hip adductors. Contain adductors
  5. 5Describe the innervation of the thigh muscular compartmentsAnterior: femoral nerve (L2-4)
    Medial: obturator nerve (L2-4)
    Posterior: Sciatic nerve (L4-S3)
  6. 6Describe the arterial supply of the high muscular compartmentsAnterior: femoral artery
    Medial: obturator artery
    Posterior: perforating branches of profunda femoris
  7. 7Describe the superficial muscles of the posterior compartment of the leg-3 muscles: gastrocnemius, soleus and plantaris
    -Share a common tendon: tendon calcaneous (AKA. Archilles Tendon)
    -Tendon inserts into calcaneous of the foot
    -Main action is to plantar flex
  8. 8What are the 4 deep muscles of the posterior compartment of the leg?-Popliteus
    -Flexor halucis longus
    -Tibialis posterior
    -Flexor digitorum longus
  9. 9What are perforating veins?-Connect deep veins to superficial veins
    -Allows venous blood to flow from superficial to deep veins
  10. 10What is compartment syndrome? Treatment?-Limb and life threatening condition
    -Perfusion pressure falls below tissue pressure in a closed anatomical compartment (ischaemia)
    -Blood loss is lost at a capillary level
    -Can lead to necrosis and death
    -Consequence of decreased compartment size or increased fluid content
    Treatment: fasciotomy (relieves tension/pressure)
  11. 11What are the different types of consequences in trauma?Anatomical ie. fracture, dislocation, soft tissue injury
    Physiological ie. bleeding/shock, coagulopathy, multiple organ failure
  12. 12What type of energy and anatomy is involved in pelvic fractures?Energy: high
    Anatomy: strong bone and ligaments
  13. 13What are the consequences of pelvic fractures?Anatomical: pubic rami fractures, sacro-iliac dislocation, urethral injury
    Physiological: haemorrhage and shock, coagulopathy, multiple organ failure
  14. 14What ligaments are associated with the pubic symphysis?Superior pubic ligament
    Arcuate ligament
  15. 15What ligaments control sagittal tilt of the spine?Iliolumbar
    Sacrospinous
    Sacrotuberous
  16. 16What ligaments are associated with the sacroiliac joint?-Anterior sacroiliac ligament
    -Interosseous sacroiliac ligament
    -Posterior sacroiliac ligament
  17. 17Why may haemorrhage occur in a pelvic fracture?-Pelvic floor is covered by major vascular structures
    -Vessels within fractured bones
    -Risk of mortality
  18. 18Describe the energy and anatomy needed for a hip fracture What age group commonly gets these?Low energy eg. fall from standing height
    Weak bone
    The elderly
  19. 19Describe the anatomical and physiological consequences of a hip fractureAnatomical: fracture needing surgery to restore. Mobility is encourage as there is a high risk with bed rest
    Physiological: frail patients with multiple comorbidities and have little physiological reserve. Cause of fall?
  20. 20What are the types of hip fracture?Blue- intracapsular
    Red- trochanteric
    Pink- subtrochanteric
    What are the types of hip fracture?
  21. 21What does the affected leg look like in a hip fracture?The leg is shorter and externally rotated (hip is less painful in this position)
  22. 22Which artery is vulnerable in knee dislocation? Why?Popliteal artery
    Shearing effect is created in the artery damaging the tunica intima
  23. 23What is a compartment? (ie. in a leg/arm)Group of muscles enclosed by unyielding tissues ie. bone or fascia
  24. 24What are the consequences of untreated compartment syndrome? What are the complications of fasciotomies?-Muscle wastage
    -Myoglobin in urine (from renal failure)
    Fractures become open and infected
  25. 25What infections affect the respiratory system?-Bronchitis
    -Pneumonia
    -TB
    -Bronchiectasis
    -CF
    -Acute exacerbations of COPD/asthma
  26. 26Describe acute bronchitis-Occurs in previously well people
    -Cough with(out) sputum
    -Viral
    -Comes with preceding symptoms
  27. 27Describe recurrent bronchitis-Cough with sputum for more than 2 out of 12 months
    -Bacterial
    -Underlying sinus infection (facial pain)
    -Post-nasal drip
    -Bronchiectasis
  28. 28Describe typical community acquired pneumonia (CAP)-Pre-existing disease eg. COPD
    -Abrupt onset (1-2 days)
    -Purulent sputum (dark green)
    -Pleuritic pain (pain in inhalation)
    -Marked systemic upset (eg. low BP)
    -Localised signs eg. coarse crackles/dullness in lungs
    -Responds to beta-lactams eg. penicillin
  29. 29Describe atypical CAP-Previously well
    -Insidious onset (7-10 days)
    -Dry cough, SoB, wheeze (small airways affected)
    -Mild systemic upset
    -No response to beta-lactams
  30. 30Describe the aetiology of CAP-Caused by mycoplasma (bacteria without a cell wall) or leigonella
    -Has winter peaks
    -Can be epidemic
    -Related to poor AC in a building
  31. 31For pneumonia to be diagnosed, a CXR should be taken. Describe what is seen when the right upper/middle/lower lobe is affectedR upper: opacity
    R middle: loss of R heart border
    R lower: loss of R diaphragm. Heart border can be seen
  32. 32What is cavitation?A black patch within a opacity
  33. 33How is the severity of CAP graded?CURB65 score:
    Confusion
    Urea
    Respiratory rate
    -Bp
    -65+
    -1 point is given for each factor
    -Total score is out of 5 (higher the score, more intensive treatment is needed)
  34. 34What are the complications of CAP?-Respiratory failure
    -Lung abscess
    -Parapneumonic effusion
  35. 35What are the different severities of parapneumonic effusion?Simple: clear fluid like water. Clears on its own
    Complex: opaque, soup consistancy. Chest drain needed
    Empyema: thick liquid (like custard). Surgical intervention necessary
  36. 36What is a loculated effusion?-Effusion is only partially drained
    -Fluid collects due to fibrous scar tissue in the pleural cavity
  37. 37How is empyema managed?Antibiotics eg. penicillins
    Drainage
    Surgery:
    -break adhesions
    -decortication (remove entire pleura)
    -open drainage into a bag
  38. 38Describe TB-Caused by M. Tb
    -Night sweats, fever
    -Weight loss
    -Productive cough ie. coughing up mucus/sputum
    -Haemoptysis
    -Is either latent or active
    -Upper lung zone shadowing is seen
  39. 39How is TB diagnosed?-Test sputum
    -Aspirate lymph nodes if involved
    -Bronchoscopy
    -Test for HIV
    -Test Vit. D levels (deficiency is highly associated with TB)
    Ask about: travel (to S. Asia/Sub-Saharan Africa), social factors, HIV
  40. 40TB can spread via the blood. What other systems can it spread to?-Meninges (headaches, neck stiffness)
    -Musculoskeletal (damage to vertebrae/intervertebral discs)
    -Genitourinary
    -Gastrointestinal (affects peritoneum: ascites, pain, stiffened peritoneum)
    -Pericardial (pericardial effusion)
  41. 41How can TB be controlled?-Public health campaigns
    -Housing/overcrowding
    -Nutrition, child health
    -Immunisation (current vaccine only covers a few strains)
    -Chemoprophylaxis (preventative treatment for those with latent TB who are asymptomatic)
  42. 42What is bronchiectasis?-Chronic bronchial dilation
    -Poor mucus clearance
    -Recurrent/chronic infection
    *cycle repeats
    Associated with:
    -chest crackles on inspiration and expiration
    (-clubbing of fingers)
    -large volume of phlegm coughed up everyday
  43. 43Describe the aetiology of bronchiectasis-Post-infective
    -Aspiration of stomach contents
    -Mechanical deficiency
    -Immune deficiency
    -RA, IBD
    -Poor mucociliary clearance (CF, primary ciliary dyskinesia)
  44. 44Describe the investigations for bronchiectasisCXR/HRCT
    Sputum culture
  45. 45How is bronchiectasis managed?-Physiotherapy (huffing, flutter device, postural drainage)
    -Bronchodilators (airways are prone to collapse on expiration
    -Antibiotics (nebulised, inhaled or oral macrolides)
  46. 46Describe CF-Autosomal recessive (mutation: deletion of F508 gene)
    -Impaired mucus clearance leads to bronchiectasis
    -Obstruction of pancreatic duct leads to diabetes and nutritional abnormalities
    -Infertility due to obstruction of male reproductive tract
    -Focal biliary cirrhosis
  47. 47What investigations are carried out for CF? How is it managed?-Sweat test (saltier than normal)
    -Genetic testing
    -Manage bronchiectasis
    -Pancreatic enzyme suppliments
    -Insulin
    -Lung transplantation if type 2 respiratory failure occurs
  48. 48Describe the exacerbations caused by COPD-Increased breathlessness
    -Green sputum
    -Increased sputum volume
  49. 49In AECOPD, what happens if admitted to the hospital?-CXR
    -Blood gases if saturation < 92% (possible respiratory failure)
    -ECG (higher risk of heart attack)
    -DECAF score (predicts mortality)
  50. 50What happens in exacerbations of asthma?-Often viral or allergic (can be bacterial)
    -Steroids are used as treatment
    -Monitor PEFR
    -If hospitalised, must be off the nebuliser for 24 hours before discharge

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Muscular Fascial Compartments and Venous Drainage Lower limb trauma
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Dolu Falowo
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