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Gastrointestinal protozoa

l.blake Inglés 12 fichas

Fichas del conjunto (12)

  1. 1Gastrointenstinal protozoaENTAMOEBA HOSTOLYTICA
    GIARDIA DUODENALIS
    TOXOPLASMA GONDII
  2. 2Disease caused:E.hostolytica = AMOEBIASIS in large intestine
    G.duodenalis = TRAVELLERS DIARRHOEA
    T.gondii = TOXOPLASMOSIS
  3. 3Entamoeba statsOccurs in large intestine amd causes INTERNAL INFLAMMATION
    50m infected worldwide -> mostly tropical countries
    85-95% asymptomatic
    5-15% asymptomatic
    E.histolytica invasive form, e.dispar uninvasive form
  4. 4Toxoplasma statsTISSUE CYST FORMING COCCIDIAN
    Suggested >30% pop is infected
    >60% people in US said to be affected
    High prevalence in france and germany
    Rare in south korea
    Usually asymptomatic
    Problematic fro weakened immune systems and pregnant women
  5. 5Giardia STATSGlobal disease
    Affects up to 2% of adults and 6-8% of children in developing countries
    Nearly 33% of people in undev countries have already had it
    Infection rates twice as high in summer
    infected by contaminated food or water
  6. 6Entamoeba LIFECYCLEHuman ingests mature cyst by contaminated food or water
    Excystation occurs in small intestine and trophozoites are released
    Trophozoites migrate to large intestine and multiply by binary fission producing cysts
    Cysts excreted in formed stool
    Trophozoites excreted in diarrhoea - uninfective form
    Trophozoutes cannot survive external environment and if ingested woukdnt survive exposure to gastric environment
    Cysts can survive days to weeks in extrenal environment because of its outer shell
    In some, trophozoites invade intenstinal mucus and ingest rbcs = INTESTINAL DISEASE
    Or through bloodstream, extraintestinal sites such as liver, brain and lungs
    EXTRAINTESTINAL DISEASE
  7. 7Giardia LIFECYCLEHuman eats or drinks contaminated food containing cysts
    Cysts excyst in the small intestine and release trophozoites
    Multiply by longitudinal binary fission
    Cysts passed in stool, trophozoute passed in diarrhoea
  8. 8Toxoplasma LIFECYCLECats become infected by eating intermediate hosts affected with BRADYZOITE CYSTS
    Unsporulated OOCYSTS are shed in cats feces (only 7-14 days)
    OOCYSTS take 1-5 days to sporulate in the environment and become infective
    Intermediate hosts in nature become infected after ingesting soil, water or plant
    Oocysts transform into TACHYZOITES shortly after ingestion.
    They localise in neural amd muscle tissue and deveop into tissue cysts BRADYZOITES
    The unsporulated oocysts may also by co sumed by animals bred for human consumption
    Human can consume food or water infected
    Can handle environmental contaminated boxes eg litter tray
    Can lay dormant inhuman as bradyzoite
    Produce cysts in skeletal muscle, eyes, myocardium amd brain
  9. 9Toxoplasma studiesRats fearless to cats
    Strong correlation to schizophrenia
    Women more likely to cheat on husbands
    Men more aggressive
    Slower reaction times
    More likely to be in a car crash
  10. 10Acute toxoplasmosis10-20% patients
    Sore lymph nodes
    Muscle pains
    Last several weeks then go away
    Parasite remains in body as BRADYZOITE
    Reactivate if become immunosuppressed
  11. 11Toxoplasmosis symptomsConfusion
    Headache
    Fever
    Nausea
    Poor coordination
    Seizures
  12. 12Congenital toxoplasmosis can causeEYE DISEASE
    Eye lesions occur in 20-80% of infected individuals when they reach adulthood
    Born with abnormal enlargement or small head

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Descripción
Fichas sobre Gastrointestinal protozoa, creado por l.blake el 12/05/2015.
Creado por
l.blake · may 2015
Actualizado
16 dic 2016
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