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AO2

Homeostatic explanation.

as it is an evolutionary adaption it should anticipate and prevent defects in energy rather than reacting too them.eating triggered when glucose low- doesn't fit in with evolutionary perspective.above optimum so prepared for shortage of food.

we consume only what we need for st.- stop us storing fat and remaining agile enough to get food- getting food strenuous not possible if storing extra fat.

LH

Damage to area- also effects to thirst and sex drive. Also, neural circuits have a role- simply not just LH

VMH

LH, VMH alone reductionistic.over simplistic. more going on than theories can account for.

Rats had LM removed could still become hungry- VMH removed- satiety

Supports- that vmh + LH are reductionist theories as still satiety and hunger suggesting other components contribute to the control of eating

NPY

Marie- mice who did not produce NPY.- no change in eating behaviour compared to normal mice.

NPY injection a experimental side effect. wouldnt happen in real life only lab conditions.

Lack external validity- when generalising to real world conditions.

-animal studies- results cant translate in humans- differences in biology- lack internal validity observations in animals may not apply to humans eating behaviour.

IDA

Deterministic- only focuses on the role of nature. override biological urges. e.g dieting.

Real world- Treat obesity e.g leptin- loose weight.

cognitive factors. Determining satiety and when a person s full. people are aware of when they have eaten. conclude when they are full.

Signals sent to hypothalamus, Possible other factors play a role in biological processes.

Rats- eat as darkness decends- caused by another part of the hypothalamus.