Test
Autonomous Nervous System
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Preguntas del test (40)
1Neurons;
- AMajority of neurons are interneurons (50 billion)
- BThe glial cells are in a lesser amount than the interneurons
- CA couple of the neurons are efferent motor neurons
- DPost-ganglionic and pre-ganglionic neurons are afferent neurons
2Division of Nervous system
- ASomatic afference - All types of realised senses
- BSomatic efference - All types of realised senses
- CEfference - Autonomic/vegetative senses (visceral sensation)
- DEfference - Autonomic system (sympathetic & parasympathetic)
3Types of receptors;
- AThe specialised cells are divided into central & peripheral receptors
- BEyes, ears, nose and tongue are peripheral receptors
- CEyes, ears, nose and tongue are central receptors
- DChemo, osmo, thermo, baro, mechano and proprioceptors are central receptors
4Afferent part of ANS;
- AThe afferent peripheral motor portions of the autonomic nervous system are made up of preganglionic and postganglionic neurons.
- BAfferent pathway consist mostly of motor neurons
- CAutonomic centres are located in cortex, subcortical regions, hypothalamus, medulla oblongata and spinal cord
- DAfferent fibers begin with receptor located in internal organs, forming visceral sensation
5Efferent part of ANS;
- AThe peripheral motor portions of the autonomic nervous system are made up of preganglionic and postganglionic neurons.
- BThe cell bodies of the preganglionic N. are located in the visceral efferent intermediolateral gray column (IML) of the spinal cord or the homologous motor nuclei of the cranial nerves.
- CPreganglionic axons are umyelinated C fibers.
- DPre-ganglionic n. synapse on the cell bodies of postganglionic neurons that are located inside the CNS.
6Efferent part of ANS;
- AThe axons of the postganglionic neurons, mostly unmyelinated C fibers, end on the visceral effectors.
- BEach preganglionic axon diverges to eight or nine postganglionic neurons. Autonomic output diffuses.
- CThe axons of the postganglionic neurons, mostly myelinated B fibers, end on the visceral effectors.
- DEach preganglionic axon diverges to five postganglionic neurons. Autonomic output diffuses.
7Choose the wrong part of ANS
- ASympathetic
- BEnteric nervous system
- CParasympathetic
- DPeripheral nervous system (PNS)
8Sympathetic system;
- AHas thoracolumbar outflow
- BHas craniosacral outflow
- CPreganglionic fibers leave the spinal cord through anterior horn
- DSpinal nerve, Post-ganglionic neurons and fibers leave through the dorsal horn
9Parasympathetic system;
- APreganglionic neurons in lateral horn of the spinal cord
- BPreganglionic fibers – in cranial and splanchnic nerves
- CHas craniosacral outflow
- DPostganglionic neurons in plexuses located deep in viscera and post-ganglonic fibers in short viscera
- EPostganglionic neurons in paravertebral and prevertebral ganglions
10Efferent ANS - chemical divison; Cholinergic component
- AUses epinephrine for transduction
- BFound in all preganglionic sympathetic and parasympathetic fibers
- CMajority of pre-ganglionic parasympathetic fibers
- DSympathetic postganglionic neurons innervating sweat glands
- ESympathetic postganglionic neurons innervating blood vessels to skeletal muscles and external sex organs (producing vascular dilatation)
11Co-transmitters and neuromediators;
- APostganglionic adrenergic - NPY, ATP, somatostatin
- BPostganglionic adrenergic fibers - VIP, NO, ATP, encephalins, Substance P, gastrin, CCK, CGRP, serotonin
- CPreganglionic cholinergic fibers have - GnRH, encephalins, Substance P, PACAP
- DPostganglionic cholinergic fibers - NPY, ATP, somatostatin
12Co-transmitters and neuromediators;
- AThey are often collected in large synaptic vesicles. Often released after serial stimulation
- BSlowly inactivated and usually reabsorbed
- CSometimes they modify function of pre-synaptic receptors through modification of action of main transmitter.
- DCommonly diffuse out of synapses – it is so called parasynaptic transmission.
13Transmitters of the sympathetic ganglion;
- AfEPSP – fast excitatory postsynaptic potential – 1st phase of excitation; early, short lasting (ca 30 ms); released by Ach through N-cholinergic receptor
- BsIPSP – slow inhibitory postsynaptic potential – (2 sec.), released by dopamine through D2 receptor from SIF interneuron (activated by Ach through M1 - cholinergic receptor)
- ClsEPSP – slow excitatory postsynaptic potential; 2nd phase of excitation, late, long (several sec.); released by Ach through M1-cholinergic receptor
- DsEPSP – late,slow excitatory postsynaptic potential; 3rd phase of excitation; very late and slow (minutes), released by GnRH.
14Pre-ganglionic to post-ganglionic;
- AfEPSP, N-cholinergic = Ach
- BsIPSP, N-cholinergic = Ach
- CsEPSP, M1-cholinergic =Ach
- DIsEPSP, M1-cholinergic = GnRH
15Choose correct matches of receptors & transmitter:
- ANicotinic - Ach
- BM1 = dopamine
- CM2 = Ach
- DM3 & M5 = Dopamine
- EM4 = Ach
16Choose correct matches of receptors & transmitter:
- AD1 = Ach
- BD5 = norepinephrine
- CD2 & D3 = Dopamine
- DD4 = Adenosine
17Choose correct receptors to 2nd messenger activity
- AM2 = decreased cAMP
- BM1, M3, M4 = Increased IP3 & DAG
- CM5 = Decreased IP3 & DAG
- DM1, M3, M4 = increased cAMP
18Which of these receptor groups are activated by norepinephrine?
- AM1- M5
- BAlpha-1A, Alpha-1B, Alpha-1D
- CGABA(A), GABA(B)
- DBeta-1, Beta-2, Beta-3
- EAlpha-2A, Alpha-2B, Alpha-2C
19Which of these receptors are NOT activated by glutamate?
- AMetabotropic & ionotropic receptors
- BNicotinic & muscarrinic
- CAMPA, NMDA & Kainate
- DGABA(A)
20Autonomic influence on eye;
- AContraction of the radial muscle of the iris is a cholinergic response
- BRadial muscle of iris is modified by alpha-1 type receptors
- CContraction of sphincter muscle of iris is a cholinergic response
- DContraction for near vision of ciliary muscle is a noradrenergic response
- ERelaxation for far vision is a noradrenergic response through Beta-2 receptors
21Autonomic influence on heart; S-A node
- ACholinergic response; beta 1 receptor = Decrease in heart rate, vagal arrest
- BNoradrenergic response; beta 2 receptor = Decrease in heart rate, vagal arrest
- CCholinergic response; beta 1 receptor = Increase in heart
- DNoradrenergic response; beta 1 = Decreased in heart rate, vagal
22Autonomic influence on heart; Atria
- ANoradrenergic response; Beta-1 = Increase in contractability and conduction velocity
- BCholinergic response; Beta-1 = Decrease in contractility and conduction velocity
- CNoradrenergic response; Beta - 2 receptors = Increase in contractability and conduction velocity
- DCholinergic response; Beta-2 = Decrease in contractility and conduction velocity
23Autonomic influence on heart; A-V node
- ACholinergic response; Beta-1 = Decrease in conduction velocity
- BCholinergic response; Beta-2 = Increase in conduction velocity
- CNoradrenergic response; Beta-2 = Increase conduction velocity
- DNoradrenergic response; Beta-1 = Decrease conduction velocity
24Autonomic influence on heart; His-purkinje system
- ANoradrenergic response; Beta-1 = Decrease in conduction velocity
- BCholinergic response; Beta-1 = Increase conduction velocity
- CNoradrenergic response; Beta-2 = Increase conduction velocity
- DCholinergic response; Beta-1 = Decrease in conduction velocity
25Autonomous influence of heart; Ventricles
- ANoradrenergic response; Beta-1 = decrease in contractibility
- BCholinergic response; Beta-1 = decrease in contractibility
- CNoradrenergic response; Beta-2 = Increase in contractibility
- DNoradrenergic response; Beta-1 = Increase in contractibility
26Autonomous influence of vessels
- ACoronary vessels are constricted by cholinergic and noradrenergic responses by Alpha-2 & Alpha-2 receptors
- BCoronary vessels are dilated through cholinergic response by Beta-1 receptors
- CSystemic veins are dilated through noradrenergic response by Beta-2 receptors.
- DSystemic veins are constricted through noradrenergic response by Beta-2 receptors.
27Autonomous influence of vessels
- ASkeletal muscle is dilated through noradrenergic response by beta-1 receptors
- BSkeletal muscle is constricted by adrenergic response and cholinergic response through Alpha-2
- CSkin and mucosa is constricted through Noradrenergic response Alpha-2
- DSkin and mucosa is dilated through cholinergic response Alpha-1
28Autonomous influence of vessels;
- ACerebral vessels are dilated through cholinergic response by Alpha-1 receptors
- BCerebral vessels are constricted through cholinergic response by beta-2 receptor
- CPulmonary vessels are dilated through cholinergic and noradrenergic responses by beta-2 and Alpha-1 receptors
- DPulmonary vessels are dilated through noradrenergic response by Alpha -1 receptors
29Autonomous influence on vessels
- ASalivary glands are constricted through cholinergic response by Alpha-1 receptors
- BAbdominal viscera is constricted through noradrenergic response by Alpha-1 receptors
- CAbdominal viscera is dilated through noradrenergic response by Beta-2 receptor
- DSalivary glands are dilated through noradrenergic response by Alpha-1 receptors
30Autonomous influence on vessels
- ARenal vessels are constricted by noradrenergic response by Alpha-1 and Alpha-2 receptors
- BRenal vessels are constricted by cholinergic response by Alpha-1 and Alpha-2 receptors
- CRenal vessels are dilated by noradrenergic response by Beta-1 and Beta-2 receptors
- DRenal vessels are dilated by Cholinergic response by Beta-1 and Beta-2 receptors
31Autonomous influence on Gastro-intestinal system; STOMACH;
- AMotility and tone decrease through noradrenergic Beta-2 receptors
- BSphincters relax noradrenically and contract cholinergically by Alpha-1 receptors
- CMotility and tone increase through noradrenergic Alpha-1 & Alpha-2 receptors
- DSecretion is stimulated by cholinergic Alpha-2 and Inhibited by Noradrenergic Alpha-2
32Autonomous influence on Gastro-intestinal system; INTESTINE;
- AMotility and tone increase through cholinergic Alpha-1 and Alpha-2 receptors
- BMotility and tone increase through cholinergic Beta-1 and Beta-2 receptors
- CSphinters relax cholinergically and contract noradrenergically Alpha-1
- DSecretion is stimulated noradrenergically and inhibited cholinergically by Alpha-2 receptors
- EGallbladder is contracted cholenergically and relaxed noradrenergically by Beta-2 receptors
33Autonomous influence on Bronchi/Urinary system
- ABronchial muscle relax through noradrenergic response by beta-2 receptors
- BBronchial glands are stimulated through noradrenergic response by Alpha-1 receptor
- CBronchial glands are inhibited through noradrenergic response by Alpha-1 receptor
- DBronchial muscle contract through noradrenergic response by beta-2 receptors
34Autonomous influence on Urinary bladder;
- ADetrusor is contracted through cholinergic response
- BDetrusor is relaxed through noradrenergic response by Beta-2 receptors
- CTrigone and sphincter relax through cholinergic response by beta-1 receptors
- DTrigone and sphincter contract cholinergic response of Alpha-2
35Autonomous influence;
- AAdrenal Medulla secretes epinephrine and norepinephrine through noradrenergic response
- BUterus relax through noradrenergic response by Alpha-1 (pregnant/non-pregnant)
- CUterus contract through noradrenergic response by Alpha-1 (pregnant)
- DThe liver initiates glycogenolysis through noradrenergic response
36Autonomous influence
- AThe male sex organ is erect through noradrenergic response and ejaculates through cholinergic response
- BPilomotor muscles contract through noradenergic response Alpha-1
- CSweat glands release slight, localised secretion through cholinergic response by Alpha-1 receptors.
- DSpleen capsule is relaxed through noradrenergic Alpha-1 receptor
- ESweat glands release general secretion through cholinergic response by Alpha-1 receptors.
37Autonomic influence of vessel; Pancreas
- AAcini increase secretion through Noradrenergic response by Alpha receptors
- BIslets increase insulin and glucagon secretion through cholinergic response by Alpha-2 receptors
- CIslets decrease insulin and glucagon secretion through noradrenergic response by Alpha-2, and increase secretion by beta-2 receptors
- DSalivary glands secrete profuse and watery secretion through noradrenergic response by Alpha -1 receptor
38Autonomous influence on vessels;
- ASalivary glands secrete amylase through cholinergic response by Beta receptors
- BLacrimal glands secrete by cholinergic and noradrenergic Alpha receptors
- CNasopharyngeal glands secrete through Noradrenergic response
- DAdipose tissue perform lipolysis through noradrenergic response by Alpha-1, Beta-1 & BEta-3
39Autonomous influence on vessels;
- AJuxtaglomerular cells increase renin through noradrenergic response by Beta-1 receptor
- BJuxtaglomerular cells increase renin through cholinergic response by Beta-1 receptor
- CPineal Gland increase melatonin synthesis and secretion through cholinergic response
- DPineal Gland increase melatonin synthesis and secretion through noradrenergic response
40Adrenergic receptors;
- AAlpha-1 = smooth muscle contraction
- BAlpha -2 = Heart muscle contraction
- CBeta - 1 = Smooth muscle relaxation
- DBeta - 3 = Enhance lipolysis
Información
- Descripción
- Based on PUM presentations and own notes
- Autor
- Soheila Amri
- Idioma
- English
- Visitas
- 2
- Etiquetas
- Valoración
- Aún sin valoraciones Valora este test