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biological explanations of anorexia nervosa
- nerual explanations
- neurotransmitters
- seratonin- disturbances in levels of NT
seratonin are a common characteristic in
individuals with eating disorders.
- bailer et al (2007) compared seratonin activity in women
recovering from restricting type anorexia and binge
eating/purging type with healthy controls. they found
significantly higher seratonin activity in the women recovering
from binge eating/purging type anorexia. they also found
highest levels of seratonin activity in women who showed
most anxiety, suggesting that persistent disruption of
seratonin levels may lead to increased anxiety, which may
trigger AN.
- bailer et al (2007) compared seratonin activity in women
recovering from restricting type anorexia and binge
eating/purging type with healthy controls. they found
significantly higher seratonin activity in the women recovering
from binge eating/purging type anorexia. they also found
highest levels of seratonin activity in women who showed
most anxiety, suggesting that persistent disruption of
seratonin levels may lead to increased anxiety, which may
trigger AN.
- dopamine- recent studies suggest a role for
dopamine in AN.
- Kaye et al (2005) used a PET scan to compare dopamine activity in the
brains of 10 women recovering from AN, and 12 healthy women. In the
AN women, they found overactivity in the dopamine receptors in a part
of the brain called the Basal ganglia, where dopamine plays a part in
the interpretation of harm and pleasure. increase dopamine activity in
this part of the brain appears to alter the way people interpret rewards.
individuals with AN find it difficult to associate good feelings with the
things people fnid most pleasureable (food!)
- Kaye et al (2005) used a PET scan to compare dopamine activity in the
brains of 10 women recovering from AN, and 12 healthy women. In the
AN women, they found overactivity in the dopamine receptors in a part
of the brain called the Basal ganglia, where dopamine plays a part in
the interpretation of harm and pleasure. increase dopamine activity in
this part of the brain appears to alter the way people interpret rewards.
individuals with AN find it difficult to associate good feelings with the
things people fnid most pleasureable (food!)
- seratonin- disturbances in levels of NT
seratonin are a common characteristic in
individuals with eating disorders.
- Neurodevelopment
- pregnancy and birth complications
- Lindberg and Hjern (2003) found significant
association between premature birth and
development of AN. birth complications may
lead to brain damage caused by hypoxia (lack of
oxygen), impairing the neurodevelopment of the
child. nutritional factors may be implicated if
mothers have an eating disorder.
- Bulik et al (2005) suggest that mothers with AN expose their
offspring to 'double disadvantage'- transmission of a genetic
vulnerability to AN, and inadequate nutrition during pregnancy
- Bulik et al (2005) suggest that mothers with AN expose their
offspring to 'double disadvantage'- transmission of a genetic
vulnerability to AN, and inadequate nutrition during pregnancy
- Lindberg and Hjern (2003) found significant
association between premature birth and
development of AN. birth complications may
lead to brain damage caused by hypoxia (lack of
oxygen), impairing the neurodevelopment of the
child. nutritional factors may be implicated if
mothers have an eating disorder.
- season of birth
- research also suggests that individuals with AN
are more likely to be born during the spring
months (Eagles et al 2001). explanations for this
association include uterine infections during
pregnancy and temperature at times of
conception.
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- research also suggests that individuals with AN
are more likely to be born during the spring
months (Eagles et al 2001). explanations for this
association include uterine infections during
pregnancy and temperature at times of
conception.
- pregnancy and birth complications
- neurotransmitters
- evolutionary explanations
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