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7033462
Burns
Descrição
year 1 Anatomy Mapa Mental sobre Burns, criado por ahmed king em 18-11-2016.
Sem etiquetas
anatomy
year 1
Mapa Mental por
ahmed king
, atualizado more than 1 year ago
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Criado por
ahmed king
mais de 7 anos atrás
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Resumo de Recurso
Burns
types
Heat burns
Cold temperature burns
Electrical burns
Chemical burns
Radiation burns
Friction burns
skin
epidermis
stratum corneum
sratum granulosum
stratum spinosm
stratum basale
stratum lucedium
dermis
Appendages
Sebaceous glands (sebum)
Empty into hair follicle
Directly into skin surface
Sweat glands
eccrine
apocrine
Hair and hair follicles
arrector pili
hypodermis
adipose tissue
treatment
onitment
topical therapy
providing occlusion, hydration, and lubrication.
Cooling
Pain management
Dressings
Classification
First-degree
affect only the epidermis
second degree
epidermis and part of the dermis
third degree
epidermis and dermis and may go into the subcutaneous tissue
Fourth degree
damage the underlying bones, muscles, and tendons
antibiotics
Mechanism & types
Mycolic acid synthesis inhibitors
folic acid synthesis inhibitors
cell wall synthesis inhibitors
Penicillin
Types
Penicillins
gram-positiveorganisms
gram-negative cocci
andnon-β-lactamaseproducinganaerobes
Susceptible to hydrolysis by β-lactamases
Antistaphylococcal Penicillins
Resistant to staphylococcal β-lactamases
staphylococci
streptococci
Extended-SpectrumPenicillins
gram-negativeorganisms
Susceptible to hydrolysis by β-lactamases
Mechanism
bind covalently to PBP and inhibit the transpeptidation process leading to cell death
Pharmacokinetics
Stable for years in dry form but loses activity rapidly (24 hrs at 20°C) in solution form.
Elimination
renal excretion
active tubular secretion
Absorption
Impaired by food
Most tissue concentrations are equivalent to those in serum
inhibition of protein synthesis
RNA synthesis inhibitors
DNA synthesis inhibitors
Burn center
Al Wakra Hospital
Microbiology lab
testing, culturing, and research
study micro-organisms
cell death
necrosis
Pathologic death of one or more cells, or of a portion of tissue or organ, resulting from irreversible damage
apoptosis
programmable cell death
inflamation
cardinal signs
Pain
Redness
Immobility
Swelling
Heat
steps
recognizing os the forien bodies by host cells and molecules
recruitment of the Leukocytes and plasma proteins
leukocytes and proteins are activated to eliminate the offending substance
damaged tissue is repaired
infection
Lymph Nodes
Functions
Removal of foreign materials such as bacteria and tumor cells from the lymphatic stream
Production of lymphocytes that function in the specific immune response
Lymphadenitis
enlarged by swelling (inflammation), often in response to bacteria, viruses, or fungi
Treatment
Antibiotics to treat bacterial infections
Analgesics (painkillers) to control pain
Anti-inflammatory medicines to reduce inflammation
Immunity
Innate
protects the body from any pathogens.
Adaptive
acts against specific foreign substances.
healing
phases
Inflammatory
Hemostasis
1-Vasoconstriction 2-Platelet plug formation 3-Coagulation
Proliferative
Epithelization
basal cell proliferation and epithelial migration occurring in the fibrin bridgework inside a clot.
Fibroplasia
fibroblast proliferation, accumulation of ground substance, and collagen production
Angiogenesis
formation of new blood vessels
Maturational
collagen cross-linking, collagen remodeling, wound contraction and repigmentation
Classification of Wounds Closure
Primary Intention
All Layers are closed
Secondary Intention
Deep layers are closed but superficial layers are left to heal from the inside out
Tertiary Intention
delayed primary closure
Risk factors for non healing
Aging
Infection
Diabetes
Nutritional status
Smoking
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