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set8
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Questions in this quiz (20)
1141. Regarding the main functions of plasmatic proteins, the following can be stated:
- AA. They function in blood coagulation
- BB. Albumin has a limited binding ability
- CC. They are transport and carrier compounds
- DD. They function in maintaining the acid-base balance
- EE. Hyperproteinemia results in muscular atrophy
- FF. They form the basis of structure of cells,tissues and organs
- GG. Proteins are not involved in the clotting system
- HH. Proteins maintain colloid-osmotic pressure
- II. Proteins are not involved in maintain blood pressure
2142. Concerning α-globulin fractions, the following can be stated:
- AA. α -lipoproteins migrate as α1
- BB. Haptoglobin is an α1-globulin
- CC. LDL migrates in β-region
- DD. α1 migrates at a slower rate than α2
- EE. HDL migrate as α1
- FF. Pre-β-lipoproteins migrate as α2
- GG. Amiloid A is a chronic phase protein
- HH. α-globulins are synthesized by the kidney
- II. VLDL migrate as α2
3143. Regarding hypoproteinemia by excessive loss the following can be stated:
- AA. It can be caused by starvation or diets poor in proteins
- BB. It is characteristic in nephritic syndrome
- CC. Loss of nutrients is a common hypoproteinemia mechanism
- DD. It is a defining mechanism in multiple myeloma
- EE. Burns are a common cause
- FF. Anorexia nervosa is a common cause
- GG. It is common in acute or chronic inflammations
- HH. It can be caused by protein-losing enteropathy
- IJ. It can be caused by enteric fistula
4144. Regarding the normal albumin: globulin ratio,the following can be stated:
- AA. Simple hypoproteinemia can occur after acute plasma loss
- BB. Excess water intake can cause an increase in serum proteins
- CC. Simple dehydration can cause hyperproteinemia with no albumin:globulin ratio changes
- DD. In simple dehydration all protein fractions increase proportionately
- EE. In simple dehydration only albumin increases
- FF. Overhydration is a common cause of simple hypoproteinemia
- GG. Overhydration can cause hyperproteinemia
- HH. Fluid therapy can cause simple hypoproteinemia
- II. Acute plasma loss can cause hyperproteinemia
5145. The following represent γ-globulins :
- AA. Immunoglobulines E
- BB. Immunoglobulines C
- CC. Immunoglobulines G
- DD. Immunoglobulines B
- EE. Immunoglobulines F
- FF. Immunoglobulines H
- GH. Immunoglobulines M
- HI. Immunoglobulines D
- IJ. Immunoglobulines A
6146. Regarding endocrine impairments in protein-energy malabsorbtion, the following can be stated:
- AA. Delay of puberty due to increased pituitary-gonadal axis can be seen
- BB. Gonadal disfunction can lead to amenorrheea
- CC. Gonadal disfunction can lead to dismenorrheea
- DD. Delay of puberty due to decreased pituitary-gonadal axis can be seen
- EE. Inadequate food intake leads to decreased protein catabolism in the skeletal muscles
- FF. Euthyroid sick syndrome can be present
- GG. Hyperthyroid sick syndrome can be present
- HH. Serum levels of thyroid hormons are decreased
- II. Serum levels of thyroid hormons are decreased
- JJ. Inadequate food intake leads to increased protein catabolism in the skeletal muscles
7147. The following are serum protein functions:
- AA. Major antibodies in response to infections
- BB. Transport
- CC. Complement inhibition
- DD. Hemoglobin transport
- EE. Complement activation
- FF. Protease stimulation
- GG. Coloid-osmotic pressure maintenance
- HH. Thrombin stimulation
- IJ. Fibrinolysis
8148. Regarding protein fractions the following can be stated:
- AA. Alfa2-globulins include: haptoglobin, ceruloplasmin,alfa2 macroglobulin
- BB. Alfa2-globulins include Erythropoietin
- CC. Alfa1-globulins include kininogen
- DD. Gamma-globulins include: IgG, IgA, IgM,IgD,IgE
- EE. Beta-globulins include: transferrin, hemopexin,ferritin and complement fractions
- FF. Hemopexin is a gamma-globulin
- GG. C-reactive protein is a alfa-globulin
- HI. Albumin is the most proeminent of the serum proteins in humans
- IJ. Alfa1-globulins include : orosmucoid, alfa1-antitrypsin
9149. Regarding albumin the following can be stated:
- AA. It is the most proeminent of the serum proteins in humans
- BB. Its catabolism is inhibited by cortisol
- CC. It is synthetized by the kidneys
- DD. Its synthesis is enhanced by growth hormone and insulin
- EE. Albumin accounts for 60-70 % of the total serum proteins in humans
- FF. Albumin accounts for 20-30 % of the total serum proteins in humans
- GG. It is synthetized by the liver
- HH. It is synthetized by all metabolically active tissues
- II. It is catabolized by all metabolically active tissues
10150. Regarding hypoalbuminemia, the following can be stated:
- AA. Extensive infammation is associated with hypoalbuminemia
- BB. Extensive infammation is associated with increased albumin
- CC. It can be associated with both normoproteinemia and hypoproteinemia
- DD. Kwashiorkor is charactherized by hypoalbuminemia and normoproteinemia
- EE. It can occur in chronic liver disease
- FF. It is a positive acute phase protein
- GG. It is always present in nephritic syndrome
- HI. It can occur due to defective hepatic synthesis
- IJ. Albumin is selectively lost in nephritic syndrome
11151. Regarding fbrinogen, the following are true:
- AA. Hypofbrinogenemia occurs in liver failure
- BB. It is a small protein
- CC. It is a large protein
- DD. Hypofbrinogenemia occurs in disseminated intravascular coagulation
- EE. Hyperfbrinogenemia is a common characteristic of infammatory processes
- FF. Hyperfbrinogenemia occurs in disseminated intravascular coagulation
- GG. It constitutes 5 % of the total plasma proteins
- HH. Hypofbrinogenemia is a common characteristic of infammatory processes
- II. It constitutes 25 % of the total plasma proteins
12152. Regarding cystinuria the following are true:
- AA. Its overexcretion predisposes to the formation of renal, uretheral and bladder stones
- BB. It is charachterized by excessive urinary excretion of dibasic acids
- CC. It is a rare inborn error of aminoacid transport
- DD. Cystine is the most soluble of the naturally occuring aminoacids
- EE. It is charachterized by reduced urinary excretion of dibasic acids
- FF. It is characterized by impaired tubular resorbtion
- GH. It is the most common inborn error of aminoacid transport
- HI. Cystine is the least soluble of the naturally occuring aminoacids
- IJ. Its overexcretion predisposes to the formation of renal, uretheral and bladder stones
13153. Regarding gout the following can be stated:
- AA. Most patients with chronic gout also develop urate nephropathy
- BB. Gout is the common end point of disorders that produce hyperuricemia
- CC. Hyperuricemia is not the sole determinant of gout
- DD. More than 10 % of the population have hyperuricemia but gout develops in less than 0,5 %
- EE. Few patients with chronic gout also develop urate nephropathy
- FF. It is marked by persistent attacks of acute arthritis
- GG. More than 10 % of the population have hypouricemia but gout develops in more than 0,5 %
- HH. Gout is the common end point of disorders that produce hypouricemia
- IJ. It is marked by transient attacks of acute arthritis
14154. Regarding creatinine the following can be stated:
- AA. It is the end product of creatine
- BB. It is a sensitive marker of liver function
- CC. Its synthesis is disproportionate to muscle mass and metabolism
- DD. Plasma levels refect the balance between synthesis and excretion
- EE. Creatinine is a sensitive marker of renal function
- FF. Creatine is a sensitive marker of renal function
- GG. It is formed intermittently
- HH. It is removed from the body by glomerular fltration
- IJ. It is formed at a constant rate
15155. Regarding urea the following are true:
- AA. Fever is associated with increased urea levels
- BB. Normal level in the plasma exceeds the blood concentration
- CC. Urea is the major disposal of amino groups derived from the catabolism of aminoacids
- DD. Pregnancy is associated with increased urea levels
- EE. Fever is associated with decreased urea levels
- FF. Urea is the major disposal of amino groups derived from the anabolism of aminoacids
- GG. Pregnancy is associated with decreased urea levels
- HH. Normal range in the plasma is 20-40 g/dL
- II. Normal range in the plasma is 20-40 mg/dL
16156. Regarding hyperphenilalaninemias the following are true:
- AA. Severe mental retardation is characteristic
- BB. They result from impaired conversion of tyrosine to phenylalanine
- CC. Phenilketonuria is rare
- DD. Phenylalanine loss leads to severe brain damage
- EE. Skin hyperpigmentation is common
- FF. Skin hypopigmentation is common
- GG. They result from impaired conversion of phenylalanine to tyrosin
- HH. Phenilketonuria is characteristic
- IJ. Phenylalanine accumulation leads to severe brain damage
17157. Concernig Hartnup disease, the following can be stated:
- AA. Renal and intestinal transport of monoamino-monocarboxylic aminoacids is defective
- BB. Most patients respond to treatment with oral nicotinamide
- CC. Inadequate tryptophan availability limits nicotinamide synthesis
- DD. It is a common disease
- EE. Secondary pellagra is associated with persistent cerebellar ataxia
- F. Secondary pellagra is associated with episodic cerebellar ataxia
- GG. Few patients respond to treatment with oral nicotinamide
- HH. Inadequate tryptophan availability stimulates nicotinamide synthesis
- IIt is a rare disease
18158. Regarding cystinosis the following are true:
- AA. The basic defect involves impaired influx into lysosomes
- BCystine accumulation in the kidneys causes hyperuricemia and hyperkalemia
- CC. Cystine accumulation in the kidneys causes proximal tubular disfunction
- DD. It is characterized by intralysosomal accumulation of free cystine in body tissues
- EE. It is a rare disorder
- FF. Growth stimulation is proeminent
- GG. The basic defect involves impaired efflux from lysosomes
- HH. Cystine accumulation in the kidneys causes proximal tubular disfunction
- I. Cystine accumulation in the kidneys causes hypouricemia and hypokalemia
19159. Regarding respiratory failure the following can be stated:
- AA. Respiratory failure is said to be of type I when there is associated CO2 retention
- BB. Respiratory failure is said to be of type I when there is no associated CO2 retention
- CC. Low oxygen levels in the blood can cause dyspnea and result in a bluish coloration to the skin (cyanosis)
- DD. Decreased partial pressure of oxygen (PaO2) below 50 mm Hg is associated with alkalosis
- EE. It is defned by partial pressure of oxygen (PaO2) below 45 mm Hg with or without increased arterial
pressure of carbon dioxide (PaCO2) above 60 mm Hg - FF. It is defned by partial pressure of oxygen (PaO2) below 60 mm Hg with or without increased arterial
pressure of carbon dioxide (PaCO2) above 45 mm Hg - GG. Bradypneea is characteristic to both types of respiratory failure
- HI. Decreased partial pressure of oxygen (PaO2) below 50 mm Hg is associated with tissue hypoxia
- IJ. Partial pressure of oxygen (PaO2) below 60 mm Hg is seen in both types of respiratory failure
20160. Regarding the stimulation of the respiratory center,the following can be stated :
- AA. A decrease in pH inhibits breathing
- BB. An increase in PaCO2 inhibits breathing
- CC. Peripheral chemoreceptors are located in the jugular bodies
- DD. Glucocorticoid receptors stimulate the respiratory center
- EF. Peripheral chemoreceptors are located in the carotid or aortic bodies
- FG. Central chemoreceptors stimulate the respiratory center
- GH. Pulmonary stretch receptors stimulate the respiratory center
- HI. Movements of joints and muscles stimulate an increase in ventilation
- IJ. Peripheral chemoreceptors stimulate the respiratory center
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- medium fizpat Quiz on set8, created by Smaranda Grigorescu on 24/01/2026.
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- Smaranda Grigorescu
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- English
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- Created on 24 Jan 2026 · updated on 25 Jan 2026
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