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L26 Clinical pharmacology of hypertension

Deleted user 9 questions Updated 26 May 2019

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Questions in this quiz (9)

  1. 190% of hypertension is ‘primary’ which means of [blank_start]unknown[blank_end] cause. 'Secondary’ is due to kidney disease, Cushing syndrome, thyroid disease, and drug-induced.
    Drug treatments include drugs to: [blank_start]dilate[blank_end] blood vessels, reduce [blank_start]constriction[blank_end] of vessels, reduce blood [blank_start]volume[blank_end], and reduce cardiac load.
    1. Aunknown
    2. Bdilate
    3. Cconstriction
    4. Dvolume
  2. 2If we give a Beta1 blocker then we anticipate a decrease in heart rate.
    1. ATrue
    2. BFalse
  3. 3If we give an alpha blocker then we anticipate vasoconstriction.
    1. ATrue
    2. BFalse
  4. 4 Non-selective β−blockers (e.g. Propanolol) should be avoided in asthmatics as they may constrict the airways.
    1. ATrue
    2. BFalse
  5. 5The RAAS system maintains fluids and electrolyte balances.
    Renin is released from the arterioles near the [blank_start]glomerulus[blank_end], in response to reduced [blank_start]blood pressure[blank_end] or reduced renal blood [blank_start]flow[blank_end].
    Renin:
    - Converts [blank_start]angiotensinogen[blank_end] to [blank_start]angiotensin[blank_end]
    - Angiotensin is converted to [blank_start]angiotensin II[blank_end] by the [blank_start]ACE[blank_end]
    ATII causes [blank_start]vasoconstriction[blank_end] and aldosterone release from the adrenals. Aldosterone promotes [blank_start]K+[blank_end] excretion in exchange for [blank_start]Na+ and water[blank_end] reabsorption to maintain plasma volume and cardiac output (ie to retain volume and raise BP.)
    1. Aglomerulus
    2. Bblood pressure
    3. Cflow
    4. Dangiotensin
    5. Eangiotensinogen
    6. Fangiotensin II
    7. GACE
    8. Hvasoconstriction
    9. IK+
    10. JNa+ and water
  6. 6If we give an ACE inhibitor or an angiotensin II receptor blocker (ARB) we anticipate vasodilation.
    1. ATrue
    2. BFalse
  7. 7Side effects of ACE inhibitors and ARBs:
    - dizziness/postural [blank_start]hypotension[blank_end]
    - [blank_start]hyper[blank_end]kalaemia (increase K+)
    - ACEI can cause [blank_start]cough[blank_end] due to build up of bradykinin (broken down by ACE)

    [blank_start]Hypotension and hyperkalemia[blank_end] are also side effects of thiazide diuretics.
    1. Ahypotension
    2. Bhyper
    3. Chypo
    4. Dcough
    5. EHypotension and hyperkalemia
  8. 8Which of these is not a side effect of calcium channel blockers?
    1. APeripheral oedema
    2. BConstipation
    3. CDizziness/postural hypotension
    4. DFlushing
    5. EHeadache
    6. FNausea
  9. 9Non-dihydropyridine calcium channel blockers can cause bradycardia (Caution if patient also on a β-blocker).
    1. ATrue
    2. BFalse

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User has deleted their subject information Quiz on L26 Clinical pharmacology of hypertension, created by Deleted user on 14/04/2019.
Author
Deleted user
Language
English
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48
History
Created on 14 Apr 2019 · updated on 26 May 2019
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