¡GoConqr estrena diseño! Cuéntanos qué te parece.

Fichas

Module Two

Nina James Inglés 115 fichas

Fichas del conjunto (115)

  1. 1SensationA stimulus detection process involving sensory organs translating environmental stimuli into electrical impulses (receiving sensor info)
  2. 2PerceptionActive processing and organising of stimuli and assigning it meaning (making sense of the sensation)
  3. 3Neural Implementation1. Sensory Organ absorbs energy
    2. Energy is transduced into a neural signal (action potential)
    3. The neural signal is sent throughout the brain for further processing
  4. 4Parts of the eye for protection:Sclera
    Cornea
  5. 5Aqueous HumourNutrition/structural
  6. 6Extraocular MusclesMovement
  7. 7ChoroidProduces light scatter
  8. 8Iris/PupilIris reacts to light sensation
  9. 9LensFocuses the light ----> fovea
  10. 10Ciliary Body/MusclesStretches
  11. 11Vitreous HumourJelly
  12. 12RetinaAbsorbs energy into electricity
  13. 13Optic NerveSends information into brain
  14. 14The visible spectrum is a ______ portion of the electromagnetic spectrumSmall
  15. 15Wavelength =Colour
  16. 16Amplitude =Brightness
  17. 17Layers of the RetinaPhotoreceptors (rods and cones)
    Bipolar Cells
    Retinal Ganglion Cell
  18. 18PhotoreceptorsTransduce light into neural activity using opsin proteins
  19. 19Two types of photoreceptorsRods
    Cones
  20. 20RodsBlack and White vision
    Use at night
    Low resolution
    High sensitivity
    More amount than cones
    Spread around the peripheral of retina
    Rhodopsin
  21. 21ConesColour Vision
    Use during the day
    High resolution
    Low sensitivity
    Less amount than rods
    Around fovea
    Cone opsin
  22. 22Bipolar CellsRelay activity to retinal ganglion cells. Half also complete signal inversion
  23. 23Retinal Ganglion Cells (RGC)Relay sensory information to brain. Begin processing visual information
  24. 24Process of Seeing:Light > Cornea > Lens > Photoreceptors > Bipolar Cells > Ganglion Cells > Optic Nerve > LGN > V1
  25. 25Is information from visual space is processed on the opposite or same side of the brain?Opposite
  26. 26LGN and V1 are organised via ________ mappingRetinotopic
  27. 27Retinotopic MappingPoint to point mapping of the external world onto the retina
  28. 28What type of processing occurs after V1?Divergent - visual information takes two streams
  29. 29Mishkin, Ungerleider & Macko ExperimentTraining of monkeys in two tasks
    Object Discrimination and Landmark Discrimination
  30. 30Temporal Lobe damage affects...Object discrimination tasks (knew where not what)
  31. 31Parietal Lobe damage effects...Landmark discrimination tasks (what, not where)
  32. 32Ventral Stream= Temporal cortex = what (objects, colour, patterns etc)
  33. 33Dorsal Stream= Parietal Cortex = where (spatial location and movement)
  34. 34The 'Grandmother Cell' hypothesis:There is a single neuron in your temporal lobe which responds to every different complex visual stimulus
  35. 35Is the Grandmother Cell Hypothesis true or false?False
  36. 36Receptive FieldsEarlier visual neurons (before V1 diverges) respond only to stimuli in a particular location in visual space
  37. 37Rods and Cones preferred stimuli:Diffused darkness
  38. 38Bipolar Cells preferred stimuli:Diffused darkness
  39. 39Retinal Ganglion Cells preferred stimuli:Spots of dark/light
  40. 40LGN Neurons preferred stimuli:Spots of dark/light
  41. 41V1 neurons preferred stimuli:Angled Lines
  42. 42Centre-Surround StructureEnhances visual contrast
  43. 43For on centre cells, stimuli that lands on the centre _____ it, while stimuli that hits the surround, _______ it.Activates, Inhibits.
  44. 44Lateral Inhibition:The capacity of an excited neuron to reduce the activity of its neighbours . The spread of APs from excited neurons in lateral directions is inhibited. This enhances contrast, allowing for increased sensory perceptions.
  45. 45What creates the Herman Grid IllusionLateral Inhibition
  46. 46High contrast stimuli are enhanced by:Lateral Inhibition between cells (horizontal connections)
    Centre surround structure of a single cell (vertical connections: bipolar-RGCs)
  47. 47AnopiaBlindness - caused by damage to the visual pathways
  48. 48BlindsightCan localise objects, but swear they cannot see.
    Damage to V1
    LGN cells bypass V1 and synapse directly onto V2 or later
  49. 49AchromatopsiaLoss of colour
    Damage to inferior temporal cortex and V4
  50. 50AkinetopsiaLoss of perception of emotion (see the world in screenshots)
    Damage to middle temporal visual cortex (V5)
  51. 51Agnosia (disorder of perception)See fine, but are unable to recognise or perceive visual stimulus
  52. 52Apperceptive AgnosiaGlobal Deficit in perception
    Unable to name objects
    Poor Shape Matching and copying
  53. 53The Peppery Mask HypothesisWidespread, diffuse damage across V1 caused by hundred of little blind spots
    i.e. CO2 poisoning or Alzheimers disease
  54. 54Two forms of SimultanagnosiaDorsal
    Ventral
  55. 55SimultanagnosiaUnable to recognise multiple objects
  56. 56Dorsal Simultanagnosia (worse)Unable to perceive/recognise more than one object at a time
    Damage is typically bilateral and in parietal/occipital regions
  57. 57Ventral SimultanagnosiaUnable to identify more than one object at a time, but aware there are multiple objects
    Damage is typically in the left hemisphere and in inferior temporal/occipital cortex
  58. 58Associative AgnosiaA specific deficit of recognition
    CAN match shapes and copy
    Whilst accurate, copying is slavish
    Degraded perception
    Damage usually involves inferior/posterior temporal lobe
    "The man who mistook his wife for a hat"
  59. 59Bottom Up Theories (direct)Start broken, build up together - build to form the overall understanding
    This is how the visual system works
  60. 60Top Down Theories (indirect)Start with a hypothesis of the big picture, is then broken down into little details
    The visual system forms hypothesis of what it is seeing based on past experiences and prior knowledge
  61. 61Evidence for Top Down TheoriesWe can recognise stimulus faster than we could process information
    We can read faster then we can process, and use contextual information to make sense of misspelled words
    We are able to switch between a bistable percept
    We are able to perceive contours that aren't there but would complete and object
  62. 62Interactive TheoriesIncorporate both theories during perception
    E.g The Perceptual Cycle Theory
  63. 63Evidence for Interactive TheoriesWhen we are exposed to a stimulus longer and when the stimuli is clearer, there tends to be less error.
    The visual system is actually built with up and down pathways
  64. 64Two forms of Depth PerceptionBinocular Cues
    Monocular Cues
  65. 65Binocular Cues (two eyes)Contribute to depth perception via eye vergence
    Cross eyed: convergence
    Wide eyes: divergent
  66. 66Retinal DisparityThe space between the eyes that means the angle of viewing an object is slightly different for each eye
    Processed in V1
    Useful for objects 10m from the eye
    How 3D glasses create the illusion of 3D
  67. 67Monocular Cues (one eye)Contribute to depth perception when viewing 2D images
  68. 686 Types of Monocular Cues1. Linear Perception
    2. Interposition
    3. Height in Plane
    4. Texture Gradient
    5. Relative Size
    6. Light/Shadow Patterns
  69. 69Colour Perception TheoriesThe Young-Helmholtz Trichromatic Theory
    The Opponent- Process Theory
  70. 70The Young-Helmholtz Trichromatic TheorySuggested that colour perception is made up of 3 different types of cones processing 3 wavelengths of light (short, medium, long)
    Problems with this:
    Why does colour blindness occur in pairs?
    Unable to explain negative colour after images
  71. 71The Opponent-Process TheorySuggests that colour perception is controlled by 3 opponent systems that detect the relative amount of light at different wavelengths
  72. 72Colour Perception Current Thoughts:Both retinal trichromacy and colour opponency circuits contribute to our perception of colour
  73. 73Localisation of Function:Different areas of the brain are responsible for different functions
  74. 74The Edwin Smith Surgical PapyrusRecognised the relationship between the location of the brain injury and the resulting specific neurological deficits
  75. 75AristotleThought that brains role was to cool blood
  76. 76AlcmaeonThought that the brain was the 'organ of the mind'
  77. 77150-1500s experts focused on:The substances within the cerebral ventricles and were 'spirit based'
  78. 78DescartesConsidered the Pineal Gland to be the principal seat of the soul
    (not true)
  79. 79Luigi RolandoWhite matter tracts link brain regions
  80. 80Gall & SpurzheimThe idea of Phrenology
  81. 81FlourensThe idea of cerebral 'equipotentiality'
  82. 82BrocaRealised that speech function is localised within the brain
  83. 83Fritsch & HitzigThe importance of electricity and the motor strip
  84. 84Santiago Ramon y CajalNeuron Doctrine and used Tracing
  85. 85Brodmanns MapDifferent areas of the cortex have different organisations
  86. 86War DoctorsShrapnel injuries allowed them to localise V1 and produce retinotopic maps
  87. 87Wilder PenfieldMapped the primary motor and sensory cortex by being electrically stimulated during surgery. Motor Homunculus
  88. 88Methods of Localising function in later 20th Century:Stimulation
    Anatomical Connectivity
    Recording Neural Activity
    Lesions (brain damage or surgery)
    Behavioural Testing
  89. 8921st Century Localisation of FunctionBrain imaging and genetic methods of stimulation and anatomy
    i.e. Dayu Lin's research using laser stimulation of genetically targeted neurons
  90. 90Current View is more supportive of:Regionalisation than equipotentiality
  91. 91Combining multiple different experiments provides the ____ evidence for localisation of functionBest
  92. 92Damage to the Superior Temporal Gyrus (STG): (hearing)Corticol Deafness (blindsight)
    Auditory Agnosias
  93. 93Left Posterior STG damage:Wernickes Aphasia
    Understanding tone and rhythm, but unable to understand meaning
  94. 94Right Posterior STG damage:Aprosodia
    Cannot understand prosody (rhythm and intonation of speech)
  95. 95Middle and Inferior Temporal Gyri: Visual ImpairmentsAchromatopsia
    Akinetopsia
    Associative Agnosia
    Ventral Simultanagnosia
  96. 96Fusiform GyrusProsopagnosia: face blindness > can see everything EXCEPT FACES
  97. 97Medial Temporal Lobe (MTL)Memory Impairments
  98. 98Right MTLVisual Memory
    - identify and copy visual stimulus, unable to reproduce them
  99. 99Left MTLVerbal Memory
    - Struggle to remember verbal information
  100. 100Damage to S1Vibration, Fine touch, Proprioception are impaired
  101. 101AstereognosiaLoss of touch based sensation
  102. 102Parietal Lobe beyond S1: sensory integration and movement. Deficits in:Abstract Concepts
    Processing Spatial Information
    Directing Attention
    Integrating Sensory Information
    Guiding Movements to a particular point in space
    Object guided hand movements
  103. 103Left Parietal Lobe:Reading and Writing
    -Agraphia (writing)
    -Alexia (reading)
    Acalculia (maths)
    Symptoms:
    Finger anomia
    Left and right confusion
    Unable to draw details
  104. 104Right Parietal Lobe:Visual and Drawing
    Symptoms:
    Unable to draw overall shape of objects
    Struggle to recognise unfamiliar view of objects
  105. 105Contralateral NeglectThe inability to perceive or attend to objects, even one's own body, on the LEFT side (opposite to side of brain damage)
  106. 106Tests of Contralateral NeglectLetter/line cancellation
    Line bisection
    Finger wiggling
  107. 107AnosognosiaA lack of awareness of their own inability
  108. 108Evidence of Contralateral NeglectThe Burning House Anecdote
    The Milan Piazza Experiment
  109. 109Is Contralateral Neglect Ego-Centric or Object-Centered?Both/Either
  110. 110Evidence of Egocentric vs Object CenteredThe Broken Hearts Test
    Draw or Copy Two People Experiment
  111. 111Why is left sided neglect most common?Because the right hemisphere of the brain is suggested to be specialised for global visuospatial processing
  112. 112Damage to the Primary Motor Cortex (M1)Paralysed or partial paralysis on opposite side of the body to the brain damage
    Problems with fine movement, speed, and strength of movement
  113. 113Damage to Premotor CortexImpaired motor planning
    Brocas aphasia
  114. 114Left premotor Cortex damageUnable to speak fluently
  115. 115Damage to prefrontal cortexImpaired divergent thinking
    Impaired response inhibition
    Environmental Dependency Syndrome (imitation and utilisation)
    Changes in personality (picks disease, Phineas Gage)

Información

Descripción
Fichas sobre Module Two, creado por Nina James el 15/06/2018.
Creado por
Nina James · jun 2018
Actualizado
16 jun 2018
Visitas
0
Valoración

Escríbenos

¿Sobre qué es tu mensaje?

¿Qué te parece el nuevo diseño?

¿Te gusta?