There are two general ways in which the
body responds to stressors:
SYMPATHOMEDULLARY PATHWAY (
deals with acute stressors) an
PITUITARY-ADRENAL SYSTEM (deals
with chronic stress)
Sympathomedullary
pathway
The sympathetic mervous
system (SNS) and the
sympatheic adrenal
medullary (SAM) up up
this.
Acute stressors activate the autonomic
nervous system(ANS) which has 2 divisions.
1. The SYMPATHETIC
NERVOUS SYSTEM (SNS)
'trouble shooter', It is highly responsive to stimuli and
through activation is responsible for emotional states
and heightened arousal.
2. The PARASYMPATHEIC NERVOUS SYSTEM
'house keeper'. It is responsible for
maintaining equilibrium and calming bodily
responses.
These 2 divisions are opposing forces,
interacting to produce bodily state at any given
time.
When exposed to an acute stressor, the SNS is
activated and, simultaneously, the SAM system
stimulates the release of ADRENALINE into the
bloodstream from adrendal glands in the adrenal medulla.
This hormone then prepares
for fight or flight repsonse by
boosting the supply of oxygen
and glucose to the brain and
muscles and suppressing
non-emergency processes like
digestion.
research to support this
Taylor (2000) found that cute stress produces the
fight-or-flight response in men, but the 'tend or befriend'
response in women. This is because women produce
more OXYTOCIN, a chemical promoting relaxation and
nuturing, suggesting a gender difference in the activation
of the sympathomedullary pathway.
Evaluation
A gender difference in the activation of this may occur due to womens
evolutionary role in caring for offspring. If women fought or fled when faced
with danger, offspring would be placed in danger, reducing reproductive
success. A better policy is to bond with other group members, most probably
females.
Much research into the Sympathomedullary pathway involves animals and thus presents a problem
of extrapolation, as the stress-related behaviour of animals may not reflect the behaviour of
humans. Humans are more likely to have a cognitive element to their stress response.
Pituitary-adrenal system
Prolonged, chronic stress activates the pituitary-adrenal system. This system is harder to initiate than
the sympathomedllary system, being activate by stressors continuous over a period of time. These
stressors alert the hypothalamus within the brain to stimulate the release of the chemical messenger
CORTICOTROPHIN-RELEASING hormone into bloodstream.
This then turn stimulates the pituitary gland to release
ADRENOCORTICOTROPIC hormone (ACTH) which travels in the bloodstream
to the adrenal glands just above the kidneys, triggering the relase of stress
related hormones including (coritsol)
The production of cortisol permits a steady supply of blood sugar, which
provides an individual with a constant source of energy, allowing the body to
deal with the stressor.
Cortisol also releases the capability to tolerate more pain than usual,
but also leads to impaired cognitive ability and reduced immune
system performance.
Research into this
Watson et al (2004) compared the
pituitary-adrenal system functioning of 26 people
with bipolar disorder, 27 people with bipolar
disorder in remission and 28 healthy controls.
They found heightened cortisol levels in bipolar
sufferers, including those currently in remission,
This suggests that pituitary-adrenal system
dysfunction may be involved in the diisease
process underlying bipolar disorder.
Evaluation
There are individual differences with
response levels. Mason (1975) found that
different individuals produce different levels of
stress hormones when exposed to the same
stressors.
People respond in more active ways to
stressors involving cognitive and emotional
factors. Symington et al (1955) found that
conscious terminal cancer patients experienced
more stress than those in a coma, as they
indulged in a more stressful appraisal of their
condition.
The biological explanation of stress allows accurate, objective measures to be made.