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Karteikarten

Urinary System

Molly Hoover Englisch 41 Karten

Urinary System

Karten in diesem Stapel (41)

  1. 1Renal AgenesisUncommon
    occurs unilaterally
    failure of a kidney to develop normally totally
    if less developed than normal then it is known as hypoplasia
    this condition goes hand and hand with renal hyperplasia
  2. 2Renal Hyperplasiaoverdeveloped kidney
    Occurs when opposite kidney is absent or so underdeveloped that its unable to perform the necessary function to maintain a healthy body
    normal kidney enlarges due to working harder
    left kidney has hypoplasia, right kidney has hyperplasia
    Renal Hyperplasia
  3. 3Renal Ectopiamisplaced kidney
    Most commonly found in the pelvis with a shorter ureter
    Renal Ectopia
  4. 4Horseshoe KidneyBoth kidneys are joined at lower poles across the midline
    Renal pelvis is located more anteriorly, at a horizontal angle
    Location causes urine to become stagnant
    Horseshoe Kidney
  5. 5Bifid SystemDouble collecting system
    Can appear: double renal pelvis or ureter in one or both kidneys (upper ureter may insert into bladder below trigone, outside bladder in urethra, or seminal vesicles), or double kidney
    Can cause: obstruction in upper pole collecting system or reflux in the kidney
    Bifid SystemBifid System
  6. 6NephroptosisFloating kidney
    Normally displaced 1 inch inferiorly upon deep inspiration
    When upright can be displaced as much as 2 inches
    Occurs when anterior surface isn't attached to peritoneum
    Length of ureter differentiates from ectopic kidney
    Nephroptosis
  7. 7Polycystic Renal DiseaseAKA polycystic kidney disease or PKD
    inherited condition
    Cysts multiply and enlarge, causing enlarged kidneys
    End stage: kidneys appear smaller and scarred
    Polycystic Renal Disease
  8. 8Glomerulonephritis (left)inflammatory disease of the capillary loops of the renal glomeruli (within nephrons)
    AKA bright disease
    Commonly developed after streptococcal infection due to antigen-antibody deposited in glomerulus
    Commonly causes underdeveloped kidneys
    Characterized by hypertension
    Symptoms: nausea, malaise, and joint pain
    Lab test show: increase in albumin, BUN, and creatinine
    CXR has pulmonary infiltrates
    Radiographic Appearance: can lead to renal failure, if chronic it leads to fibrosis, leading to the kidney shrinkage, scarring, and shrinkage but initially the kidneys are enlarged due to inflammation
    Glomerulonephritis (left)
  9. 9Pyelonephritis (Right)Inflammation of the kidney and renal pelvis
    Acute: escherichia coli invades the renal tissue, commonly enters the kidney through retrograde from the bladder of through blood
    Chronic: chronic reflux occurs of infected urine from the bladder into the renal pelvis
    More common in females
    symptoms: flank pain, bacteriuria, pyuria, dysuria, nocturia, and increased frequency of urination
    Acute: calyces are enlarged, blunting of calyces
    Chronic: decreased kidney size with destruction and scarring due to fibrosis
    Pyelonephritis (Right)
  10. 10DiverticulaAbnormal pouches in variable sizes, urine lies stagnant
    Occurs in the bladder or ureters
    Diverticula
  11. 11UreteroceleCyst-like dilation of the ureter near its opening into the bladder
    Radiographic Appearance: "Cobra head" on an IVU
    Ureter is the body and the dilated ureteric segment is the flared cobra head, if image is taken exactly when contrast is excreted into the bladder it appears as the snake's tongue
    UreteroceleUreterocele
  12. 12Prostatic HypertrophyProstate gland enlargement along with urinary output obstruction
    Common in males over 55 years of age
    Symptoms: reduced urine output along with sensation of full bladder
    Radiographic Appearance: borders of the bladder appear lumpy and irregular, base of the bladder has a notched appearance due to the enlarged prostate pushing up against it
    Prostatic HypertrophyProstatic Hypertrophy
  13. 13HydronephrosisWater in the nephrons of the kidney (there is no actual water though)
    Results from an obstruction of the renal pelvis, calyces, and ureter due to back pressure of urine that can't flow pas the obstruction
    Is NOT an actual disease but results from another
    Occurs bilaterally in pregnancy due to fetus pushing against ureters
    Symptoms: hematuria, pyuria, flank pain, fever
    Radiographic Appearance: IVU-enlargement above the obstruction and no anatomy demonstrated below, calyces are sharp in appearance with enlargement
    Hydronephrosis
  14. 14Renal CalculiSolid masses, consisting of collections of tiny crystals that contain calcium
    Tiny crystals can pass through urine but sometimes they clump and cling to tissue of the kidney, then continue to grow as new crystals are added then harden
    Commonly seen in renal pelvis
    Symptoms: hematuria, severe flank pain radiating to groin or genitals
    Different types: calcium, uric, struvite, staghorn, nephrocalcinosis, and bladder
    Calcium-most common
    Uric-associated with gout, radiolucent, seen as filling defect on x-ray
    Struvite- composed of magnesium ammonium phosphate
    AKA "infection stones"
    Common in females
    Found with UTI's
    Staghorn Calculus
    Occurs when a stone begins formation in renal pelvis and continues to grow there, may take that shape
    Appears as horns on a deer head, blocking the flow of urine
    Renal CalculiRenal Calculi
  15. 15NephrocalcinosisNumerous irregular spots of calcium in the renal parenchyma
    There may be few or many
    Nephrocalcinosis
  16. 16Bladder StonesSingle or multiple varying in size
    Commonly formed from stagnant urine that is unable to pass due to obstruction
    Bladder Stones
  17. 17Renal FailureCondition where kidneys fail to function properly with a decrease in GFR
    Acute and Chronic
    Acute: rapid, severe deterioration in kidney function, dialysis may be necessary
    Chronic: Slow, progressive, and irreversible loss of renla function, dialysis is undergone on a regular basis until a kidney transplant takes place
    Ultrasound is modality of choice due to contrast detrimental with renal failure
  18. 18Benign NeoplasmRenal Cyst: acquired condition, commonly occurs in cortex of lower pole, asymptomatic unless larger causing pain or obstruction
    Radiographic Appearance: Calyce spreading, does not show irregular opacification after contrast injection, commonly seen on ultrasound, CT, and MRI
    Benign Neoplasm
  19. 19Malignant (Hypernephroma)Most common
    Linked with the next two types of malignancy
  20. 20Grawitz TumorKidney Cancer
    Linked with renal tumor and renal cell carcinoma
  21. 21Renal Cell CarcinomaNormal
    Idiopathic, related to cigarette
    Can be caused by long term dialysis
    Develops from the renal tubules, destroying the kidney while it invades the blood vessels (IVC and renal vein), allowing spreading of malignant cells
    Symptoms: hematuria
    Common in males over 40 years old
    Radiographic Appearance: Angiography and ultrasound modality of choice, angiography: abnormal vascular ultrasound: composition of tumor along with invasion of renal vessels
    Renal Cell CarcinomaRenal Cell Carcinoma
  22. 22NephroblastomaAKA Wilm's Tumor
    Most common abdominal neoplasm in children
    Common at age 3
    Cause unknown but associated with urinary tract birth defects
    Symptoms: palpable abdominal mass, blood in urine, fever
    Radiographic Appearance: abdominal mass that displaces the kidney, CT identifies stage and prognosis of tumor
    Radiographic Appearance: Filling defect during IVU, CT is most reliable due to tumors commonly being small and located in trigone
    Nephroblastoma
  23. 23What do the kidneys do?Balance the fluid in your body, detect waste in your blood, and know when to release the vitamins, minerals, and hormones
  24. 24Main Function of the kidneys?dispose of waste products and turn them into urine
  25. 25Your body has8 liters of blood that pass through the kidneys between 20-25 times a day, these organs filter 180 liters every 24 hours
  26. 26Ingredients in your blood change due to?food and drink, that's why the kidneys need to be on duty
  27. 27Blood enters kidneys through what?Arteries which branch and form tiny internal modules called nephrons
  28. 281 million of the nephrons form what?A powerful array of filters and sensors to carefully sift the blood
  29. 29To filter the blood what two powerful filters do you need?Tube like filter called the glomerulus, string like structure called a tubule
  30. 30The glomerulus acts like a sift allowingcertain ingredients such as vitamins and minerals to pass into the tubule
  31. 31What are the vessels jobto detect whether any of these ingredients are needed in the body
  32. 32Blood doesn't just contain useful ingredients it also containswaste products too and the nephrons have to figure out what to do with them
  33. 33The tubulesSense compounds that the body doesn't need like uria left over from the break down of proteins and redirects them as urine out of the kidneys through two long sewers called ureters
  34. 34What do the ureters do?Empty contents into the bladder ridding your body from that waste
  35. 35What is in your urine?Water and if the kidney detects too much of it in your blood it send it the extra water to the bladder
  36. 36What happens if there is low levels of water in the blood?It prompts the kidneys to distribute water back into the blood, meaning that less water makes it into the urine
  37. 37Why does urine appear yellowerit is When your less hydrated, by controlling water your kidneys stabilize fluid levels
  38. 38What do the kidneys activate?Vitamin D, to secrete a hormone called renin, it raises blood pressure
  39. 39Another hormoneErthropoietin which produces red blood cells
  40. 40Without the kidneysOur urine would spiral out of control, every time we eat our blood would receive another load of unsifted ingredients
  41. 41Buildup of ingredients would do whatOverload our systems and we would expire, our kidneys keep us alive

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Karteikarten am Urinary System , erstellt von Molly Hoover am 02/03/2017.
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Molly Hoover
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