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L13 Asthma

Deleted user 10 questions Updated 26 May 2019

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  1. 1Asthma is an [blank_start]immune[blank_end] dysfunction, an [blank_start]inflammatory[blank_end] response to a non-pathogenic antigen, therefore an atopic or allergic immune response.
    • Type [blank_start]I & IV[blank_end] hypersensitivity
    • Antibody – [blank_start]IgE[blank_end]
    • [blank_start]Cells[blank_end] – T cells, B cells, mast cells, eosinophils
    • 2 phases – [blank_start]Sensitisation[blank_end] & inflammation
    1. Aimmune
    2. Binflammatory
    3. CI & IV
    4. DIgE
    5. ECells
    6. Fsensitisation
  2. 2The [blank_start]Atopic March[blank_end] is the relationship between ashtma, atopic dermatitis(AD) and allergic rhinitis(AR) in children. Children with one [blank_start]atopic[blank_end] condition often will also have [blank_start]another[blank_end].
    Food [blank_start]allergy[blank_end] also occurs frequently in children with AD or [blank_start]precedes[blank_end] AD.
    1. AAtopic March
    2. Banother
    3. Catopic
    4. Dallergy
    5. Eprecedes
  3. 3The first stage of asthma pathology is the [blank_start]sensitisation[blank_end]. A [blank_start]dendritic[blank_end] cell in the lungs encounters an allergen and becomes activated, then travels to a [blank_start]lymph[blank_end] node. T cell activation and [blank_start]proliferation[blank_end] occurs, which releases [blank_start]interleukins[blank_end], and activates B cells. B cells proliferate and release [blank_start]IgE[blank_end] antibodies.
    The second stage is inflammation. IgE coated/sensitised [blank_start]mast[blank_end] cells and eosinophils in the lungs encounter the allergen and [blank_start]degranulate[blank_end], releasing histamines, proteases, leukotrienes, and prostaglandins which inflame the airways.
    1. Asensitisation
    2. Bdendritic
    3. Clymph
    4. Dproliferation
    5. Einterleukins
    6. FIgE
    7. Gmast
    8. Hdegranulate
  4. 4Choose the correct statement.
    1. ATh2 cells produce cytokines that drive IgE production by B cells
    2. BEosinophils release bronchoconstrictors & cytokines
    3. CMast cells release mediators that damage cells, and contribute to remodeling
  5. 5Non-atopic asthma, also known as [blank_start]intrinsic[blank_end] asthma, is asthma [blank_start]without[blank_end] atopy. A skin test will be [blank_start]negative[blank_end]. Commonly in women, and usually in adults.
    There are some differences (debated); the presence of [blank_start]innate[blank_end] lymphoid cells (ILCs), and [blank_start]less[blank_end] IgE, for example.
    Neutrophilic asthma is another type. It is usually [blank_start]late[blank_end] onset and [blank_start]more[blank_end] severe, and less reversible. There are some differences; [blank_start]Th17[blank_end] rather than Th2, [blank_start]neutrophils[blank_end] rather than eosinophils. It is corticosteroid [blank_start]insensitive[blank_end].
    Occupational asthma is another type.
    1. Aintrinsic
    2. Bwithout
    3. Cnegative
    4. Dinnate
    5. Eless
    6. Flate
    7. Gmore
    8. HTh17
    9. Ineutrophils
    10. Jinsensitive
  6. 6The pathophysiology of asthma includes inflammation, hyperresponsiveness/bronchoconstriction, oedema, and remodelling. Inflammation is quite complex and impacts [blank_start]treatment[blank_end] decision. Hyperresponsiveness is [blank_start]reversible[blank_end] early on and can be measured. Oedema is due to [blank_start]mucus[blank_end] hypersecretion. Airway remodelling is associated with a progressive, [blank_start]irreversible[blank_end] loss of lung function. It includes the thickening of the [blank_start]basement[blank_end] membrane, fibrosis, airway smooth muscle [blank_start]hypertrophy[blank_end] and hyperplasia, angiogenesis, and mucus gland hyperplasia.
    1. Atreatment
    2. Breversible
    3. Cmucus
    4. Dirreversible
    5. Ebasement
    6. Fhypertrophy
  7. 7There is a small but significant dose related association with antibiotics in pediatrics and the development of asthma.
    1. ATrue
    2. BFalse
  8. 8There is a dose dependent association between paracetamol use in first
    year of life and asthma, i.e. paracetamol increases the risk of asthma.
    1. ATrue
    2. BFalse
  9. 9NZ's rate of asthma in the population is:
    1. Alow and stable
    2. Blow and fluctuating
    3. Chigh and stable
    4. Dhigh and fluctuating
  10. 10In conventional asthma treatment and management, we:
    - Abolish symptoms - [blank_start]Beta agonists[blank_end] (relievers)
    - Restore best possible airway function - anti-inflammatory drugs ([blank_start]preventers[blank_end])
    - Practice allergen [blank_start]avoidance[blank_end]
    1. ABeta agonists
    2. Bpreventers
    3. Cavoidance

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User has deleted their subject information Quiz on L13 Asthma, created by Deleted user on 14/04/2019.
Author
Deleted user
Language
English
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82
History
Created on 14 Apr 2019 · updated on 26 May 2019
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