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Neurodeg3AD

Mak Sch 12 questions

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Questions in this quiz (12)

  1. 1Cognitive features impaired in AD are:
    1. AAttentional and executive deficits
    2. BAnterograde episodic memory
    3. CSemantics
    4. DAll of the above
  2. 2Question 2
    Working memory is:
    (multiple answers are possible)
    1. AA. a cognitive system that is responsible for the transient holding, processing, and manipulation of information
    2. BB. an executive controller that interacts with separate short-term stores for auditory-verbal and visuo-spatial information
    3. CC. a cognitive system where information can be stored for long periods of time
    4. DD. a limited capacity system that is capable of briefly storing and manage information involved in the performance of complex cognitive tasks such as reasoning, comprehension and certain types of learning
  3. 3Synaptic loss is a prominent early pathological feature of AD, and closely associated to cognitive decline, it is mainly localized:
    Answers:


    1. AA. Hippocampus
    2. BB. Diffuse in all the brain
    3. CC. Mainly in Cortices
    4. DD. Entorhinal cortex
  4. 4Described by Braak and Braak, tangles sequentially appear in the following specific regions as AD progresses:
    1. AA. Limbic-Isocortical-Transentorihnal
    2. BB. Transetorihnal-Limbic-Isocortical
    3. CC. Isocortical-Transentorihnal- Limbic
    4. DD. Transentorihnal- Isocortical-Limbic
  5. 5In Anterograde Amnesia: (multiple answers possible)
    Answers:







    1. AA. Memories created prior to the event that caused the amnesia are lost while new memories can still be created.
    2. BB. There is loss of the ability to create new memories after the event that caused the amnesia, leading to a partial or complete inability to recall the recent past
    3. CC. New memories can still be created.
    4. DD. Long-term memories from before the event remain intact
  6. 6Question 6
    One of the earliest pathological changes on AD is the increase in tangle Tau formation in:
    1. AA. Transenthorhinal region
    2. BB. Striatum
    3. CD. Parietal cortex
    4. DC. Prefrontal cortex
  7. 7The most common familial early onset AD is related to:





    1. AA. Presenilin 2 gene at Chromosome 1
    2. BB. Apolipoprotein E gene at Chromosome 19
    3. CC. Presenilin 1 gene at Chromosome 14
    4. DD. Amyloid protein precursor at Chromosome 21
  8. 8Question 8
    Studies of AD patients with FDG-PET + MRI coregistration (Positron emission tomography with fluoro-2-deoxy-D-glucose in combination to Magnetic Resonance Imaging) revealed the importance of the following structures in the site of early pathology in AD
    (multiple answers possible)
    1. AA. Posterior cingulate
    2. BD. Mediotemporal lobe
    3. CC. Mammillary bodies and thalamus,
    4. DB. Retrosplenial cortex
  9. 9Question 9
    Other early pathological changes on AD is the increase in amyloid plaques in:
    (multiple answer possible)
    1. AA. Transenthorhinal region
    2. BD. Parietal cortex
    3. CB. Posterior cinguate cortex
    4. DC. Frontal and association cortices
  10. 10Question 10
    The greatest risk factors for Alzheimer's are: (multiple answers possible)
    1. AA. Gender
    2. BB. Age
    3. CC. Genetics
    4. DD. Head injury

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Description
Biologie Quiz on Neurodeg3AD, created by Mak Sch on 30/11/2017.
Author
Mak Sch
Language
English
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