Fichas
Exam 3
Fichas del conjunto (46)
- 1Most common growth of the oral cavityFibroma
- 2Where do fibromas occur?Along sites prone to occlusal trauma
- 3Giant cell fibroma age predilectionSlight predilection for 1st-3rd decades
- 4Nodule with papillary surfaceGiant cell fibroma
- 5Fibrous hyperplasia secondary to ill-fitting dentureEpulis fissuratum
- 6Inflammatory papillary hyperplasia -Site -Treatment-palatal mucosa
-counsel to discontinue continuous denture wearing
-ablation/excision prior to relining or refabricating denture - 7Pyogenic granuloma-purple/red, rapidly growing nodule or papule occuring in skin or mucosa
-history of trauma may be elicited
-represents an impaired wound healing process - 8Pyogenic granuloma site predilection-Head and neck, especially mucous membranes
>>gingiva in 75% of intraoral cases
-Limbs - 9Pyogenic granuloma clinical features-smooth or lobulated nodule
-bright/dark red and yellow (where ulcerated)
-asymptomatic
-may bleed easily - 10Pyogenic granuloma may arise from the socket of a recently extracted tooth...epulis granulomatosa
- 11Pyogenic granuloma may occur in pre-existing...periodontal disease
- 12Pregnancy tumor (aka...)Granuloma gravidarum
-estrogen regulates angiogenic factors such as VEGF, NO (blood vessels have receptors for estrogen; binding can cause angiogenic growth factors)
-Involution/regression upon cessation to hormonal exposure - 13Vascular malformations arise from... They can be...-genetic errors in capillary plexus during embryologic development
-Can be capillary, venous, lymphatic, or combined - 14Vascular malformations -Affected cells retain...increased proliferative capacity throughout the individual's life
- 15Most common cervicofacial vascular malformationVenous malformation
- 16Majority of venous malformations caused by...activating TIE2 mutations
(endothelial cell-specific tyrosine kinase receptor) - 17Clinical Appearance of Venous Malformation-soft, compressible mass
-bluish hue
-no pulsation
-may be complicated by thrombus, phlebolith formation
-blanches upon diascopic examination - 18Sclerotherapeutic agent...damages endothelium, ultimately resulting in shrinkage
- 19Treatment of Venous Malformation-Surgery curative for small lesions
-Large/extensive VMs require sclerotherapy prior to surgery - 20Most volumetric distortion of all types of vascular malformationsLymphatic malformation
- 21What causes lymphatic malformations?PIKSCA mutations
- 22What is a very common location for lymphatic malformations?the tongue
- 23Highly characteristic intraoral presentation of lymphatic malformationclusters of superficial translucent vesicles
- 24Cystic hygromalymphatic malformation of the neck, axilla, inguinal region
- 25What is a common vascular malformation that affects approximately 0.5% of the population?capillary malformation
- 26What are capillary malformations also known as?Port wine stain
- 27What causes capillary malformations?GNAQ mutations
- 28How do capillary malformations present? What is the treatment?-presents as macular area of erythema (longstanding lesions may be verrucous, nodular)
-treatment unnecessary unless cosmetically indicated - 29Facial Capillary malformations typically follow what distribution? CM affecting what nerve branch warrants brain MRI? What does the MRI rule out?-distribution of trigeminal nerve branches
-Ophthalmic branch (V1)
-Rules out Sturge-Weber syndrome - 30Sturge-Weber Syndrome-facial CM along V1
-Ipsilateral capillary/venous malformations of brain/eye
-cerebral atrophy, seizures, intellectual disability, risk of stroke, dystrophic calcifications, glaucoma - 31Most common tumor of infancy and childhoodHemangioma
- 32More common type of hemangiomaInfantile hemangioma
(predilection for head&neck; becomes apparent within first few days/weeks/months of life; slow proliferation/growth over 6-12months; spontaneous involution over 3-9yrs) - 33Mass of hyperplastic fibrous tissue, presumably secondary to plaque/local irritants (AKA fibroma)Inflammatory fibrous hyperplasia
- 34Extraosseous counterpart to cemento-ossifying fibromaPeripheral ossifying fibroma
- 35Clinical appearance of Peripheral ossifying fibroma-young adults
-interdental papilla area
-pink/red; firm; frequently ulcerated - 36Treatment and recurrence rate of peripheral ossifying fibroma-asymptomatic and conservatively excised
-recurrence rate around 5-15%
-likely originates from PDL/periosteum
-Lesion should be excised down to periosteum - 37Extraosseous counterpart to central giant cell granulomaPeripheral giant cell granuloma
- 38Clinical appearance of Peripheral GCG-older adults
-interdental papilla, edentulous alveolar ridge
-dark red/purple
-cupping resorption of underlying bone characteristic - 39Treatment and recurrence rate of peripheral GCG-asymptomatic and conservatively excised
-recurrence rate around 5-15%
-likely originates from PDL/periosteum
-Lesion should be excised down to periosteum - 40Most common soft tissue tumor in adultsLipoma
- 41Lipoma-benign neoplasm of fat cells
-most often occurs as a solitary subcutaneous mass - 42Many so-called lipomas of buccal mucosa represent...herniation of buccal fat pad
- 43Appearance of lipomas and their treatment-intraoral lipomas may present with yellow hue, otherwise present as submucosal nodules with no distinguishing features
-asymptomatic
-Conservatively excised - 44Most common sarcoma of oral cavityAtypical lipomatous tumor
(Well-differentiated liposarcoma, WDLPS) - 45Can WDLPS metastasize?No
- 46What is the difference between: Atypical lipomatous tumor and WDLPS?Atypical lipomatous tumor= superficially located WDLPS and surgically amenable
Información
- Descripción
- NDBE Oral Path Fichas sobre Exam 3, creado por Laura Gennaro el 27/03/2018.
- Creado por
- Laura Gennaro
- Visitas
- 1
- Valoración
Más sobre Oral Mucosal and Soft Tissue Lesions