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Respiratory Qs

Etomby Namme last min review - English 79 cards

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  1. 1how long should a peakflow diary be done for?2 weeks
  2. 2what other risk factors for lung cancer are there?asbestos
    chromium - steel worker
    arsenic- agriculture, industrial town
    iron oxides - miners
    radon gas
  3. 3what's the most common type of lung cancer?sqamous cell carcinoma
  4. 4signs and symptoms of lung cancercough
    dyspnea
    haemoptysis
    recurrent and difficult to treat pneumonia
    redflags
    signs
    clubbing
    HPOA wrist pain (hypertrophic osteoarthopathy)
    supraclavocular and axillary nodrs
  5. 5what type of cells are pancoast tumours made of?most commonly squamous fell carcinomas
  6. 6what paraneoplastic sydromes are caused by sclc?SIADH
    hyperparathyroidism
    hyperthyroidism
    EAS/cushings (ectopic ACTH secretion)
    hyperaldosteronism
    ectopic insulin secretion
    ectopic growth hormone
    serotonin syndrome
    lambert-eaton syndrome
  7. 7investigations in lung cancerCXR
    bronchioscopy
    fine needle aspiration and biopsy
    sputum and pleural fluid cytology
    PET scan for staging
  8. 8what is the use of lung function tests in suspected lung cancer?assess suitability for lobectomy
  9. 9wells score criteria-symptoms of dvt
    HR >100
    -recent stasis or major surgery with 4 weeks
    - haemoptysis
    previous dvt or PE
    cancer- active treatment or in last 6 months
  10. 10what electrolyte imbalances might you see in lung cancer?increased CA
    hypokalamia (EAS)
    hyponatraemia(SIADH)
  11. 11side effects of isoniazidperipheral neuropathy
    hepatitis
    agranulocytosis
  12. 12symptoms of hypercalcemialoss of appetite, nausea, vomiting, constipation, numbness, polyurea, polydypsia, and dehydration, while late symptoms include renal failure, nephrocalcinosis, confusion, and coma
  13. 13what other type of lung cancer causes increased Ca levels?squamous cell carninoma
  14. 14side effects pf pyrazinamidehepatitis
    gout
    arthralgia
    myalgia
  15. 15side effects of ethambutoloptic neuritis
  16. 16what are the 3 most common causes of community acquired pneumonia?-streptococcus pneumonia
    -haemophillus influenza
    -mycoplasma pneumonia
  17. 17what presentations might suggest mycoplasma pneumonia?headache, myalgia, DRY cough..
    -cold agglutinin disease
    -haemolytic anaemia
    - Guillian- Barre
    - Erythema Multiforme
  18. 18what are the 3 most common causes of Hospital acquited pneumonia?staphylococcus aureus
    pseudomonas auerginosa
    klebsiella pneumonia
  19. 19how do you test for Legionella?urine antigen test
  20. 20what is used to assess severity of pneumonia?CURB 65
    - Confusion (amts <8/10)
    -Urea (>7mmol/l)
    -Resp rate (>30/min)
    - blood pressure (<90mmHg)
    over age 65
  21. 21when do you manage pneumonia in hospital?CURB 65
    score over 2
  22. 22what is sepsis six?Blood culture
    urine
    fluids
    antibiotics
    lactate
    oxygen
  23. 23what do you typicallt give for moderate severe community acquired pneumonia?amoxicillin + clarythromycin
  24. 24treating sever c.a.pneumiabenpen + clarithromycin/ Doxycyclin
  25. 25treatment for hospital acquired pneumoniaco-amoxiclav or cefuroxime
  26. 26complications of pneumonia- empyema
    - pneumatocelle
    -pneumothorax
    - pleural effusion
    - DVT
    -pneumatocelle
    -lung abcess
    -septicemia
  27. 27complications of pneumonia- empyema
    - pneumatocelle
    -pneumothorax
    - pleural effusion
    - DVT
    -pneumatocelle
    -lung abcess
    -septicemia
  28. 28whats the test for TB?zeihl- nielsen sputum stain- acid fast bacili
  29. 29whats the test for TB?zeihl- nielsen sputum stain- acid fast bacili
  30. 30treatment of TBFirst 2 months:
    RIPE
    rifampicin
    isoniazide
    pyrazinamide
    ethambutol
    continuation:
    rifampicin
    isoniazid
  31. 31treatment of TBFirst 2 months:
    RIPE
    rifampicin
    isoniazide
    pyrazinamide
    ethambutol
    continuation:
    rifampicin
    isoniazid
  32. 32stepwise management of copdSABA
    LABA-LAMA
    (+ ICS if element of reversibility)
    + theophyline
    + carbocysteine (in chronic mucus production)
    +prophylactic azithromycin (in repeated hospitalisation)
    +Roflumilast ( in severe- FEV1 <50%
  33. 33stepwise management of copdSABA
    LABA-LAMA
    (+ ICS if element of reversibility)
    + theophyline
    + carbocysteine (in chronic mucus production)
    +prophylactic azithromycin (in repeated hospitalisation)
    +Roflumilast ( in severe- FEV1 <50%
  34. 34what are the 3 most common causes of Hospital acquired pneumonia?staphylococcus aureus
    pseudomonas auerginosa
    Kiebsiella Pneumonia
  35. 35what are the 3 most common causes of Hospital acquired pneumonia?staphylococcus aureus
    pseudomonas auerginosa
    Kiebsiella Pneumonia
  36. 36risk factors for IPFmale
    50-70
    exposure to environmental toxins
  37. 37risk factors for IPFmale
    50-70
    exposure to environmental toxins
  38. 38what disorders are associated with IPF?-vasculitis:
    wegners
    churg strauss
    Goodpastures
    -inflammatory disorders:
    SLE
    RA
    systemic sclerosis
    -malignancy
    -sarcoidosis
    -amyloidosis
  39. 39what disorders are associated with IPF?-vasculitis:
    wegners
    churg strauss
    Goodpastures
    -inflammatory disorders:
    SLE
    RA
    systemic sclerosis
    -malignancy
    -sarcoidosis
    -amyloidosis
  40. 40what special test diagnosis IPFreduced transfer factor (TLco test)
  41. 41what special test diagnosis IPFreduced transfer factor (TLco test)
  42. 42whats the prognosis of IPF3-5 years survival
  43. 43whats the prognosis of IPF3-5 years survival
  44. 44obstructive picture on lf testsnormal lung capacity
    decreased FEV1
    decreased FEV/FVC
  45. 45obstructive picture on lf testsnormal lung capacity
    decreased FEV1
    decreased FEV/FVC
  46. 46restrictive picture on lung function testreduced lung volume
    normal FEV1/FVC
    reduced Forced vital capacity
    normal or reduced FEV1
  47. 47restrictive picture on lung function testreduced lung volume
    normal FEV1/FVC
    reduced Forced vital capacity
    normal or reduced FEV1
  48. 48what is MEIGs syndrome?Ascites
    Pleural effusion
    Benign Ovarian Tumour
  49. 49what is MEIGs syndrome?Ascites
    Pleural effusion
    Benign Ovarian Tumour
  50. 50transudative vs exudative effusionExudative- protein exits
    inflammatory condition causes leaky vessels
    >35g/l protein
  51. 51transudative vs exudative effusionExudative- protein exits
    inflammatory condition causes leaky vessels
    >35g/l protein
  52. 52examination findings in pleural effusion- tracheal deviation away from effusion
    - bronchial breathing over area of effusion
    -stony dull percussion
    decreased breath sounds
  53. 53examination findings in pleural effusion- tracheal deviation away from effusion
    - bronchial breathing over area of effusion
    -stony dull percussion
    decreased breath sounds
  54. 54what findings in a peakflow diary are indicative of asthma?diurnal variations
  55. 55what findings in a peakflow diary are indicative of asthma?diurnal variations
  56. 56what investigation differentiates asthma from copd?exhaled nitric oxide
  57. 57what investigation differentiates asthma from copd?exhaled nitric oxide
  58. 58asthma managementSaba
    +ics
    +LTRA
    +LABA
    consider MART
    +LAMA or Theophyline
  59. 59asthma managementSaba
    +ics
    +LTRA
    +LABA
    consider MART
    +LAMA or Theophyline
  60. 60signs of severe asthmaPEF: <50% best/ predicted
    HR >110
    RR > 25
    inability to complete sentences
  61. 61signs of severe asthmaPEF: <50% best/ predicted
    HR >110
    RR > 25
    inability to complete sentences
  62. 62signs of life threatening asthmaPEF: <35%
    O2 sats <92
    PaO2: <8kPa
    poor resp effort
    silent chest
    arrythmia
    hypotension
  63. 63signs of life threatening asthmaPEF: <35%
    O2 sats <92
    PaO2: <8kPa
    poor resp effort
    silent chest
    arrythmia
    hypotension
  64. 64causes of clubbingcardiac:
    cyanotic congenital HD, IE,
    respiratory:
    bronchiectasis, IPF, CF, sarcoidosos, TB, lung cancer
    abdominal:
    IBD, liver cirrhosis
    metabolic:
    acromegaly, pregnancy, thyroid acropathy, inherited familial clubbing
    IBD
  65. 65causes of clubbingcardiac:
    cyanotic congenital HD, IE,
    respiratory:
    bronchiectasis, IPF, CF, sarcoidosos, TB, lung cancer
    abdominal:
    IBD, liver cirrhosis
    metabolic:
    acromegaly, pregnancy, thyroid acropathy, inherited familial clubbing
    IBD
  66. 66what sign do you see in histological examination that indicates bronchiectasissignet ring sign:
    the bronchioles are larger than the corresponding vessels
  67. 67what sign do you see in histological examination that indicates bronchiectasissignet ring sign:
    the bronchioles are larger than the corresponding vessels
  68. 68what is tram tracking?a sign seen on xray indicating bronchiole dilation in bronchiectasis
  69. 69what is tram tracking?a sign seen on xray indicating bronchiole dilation in bronchiectasis
  70. 70what is the test for CF?chloride sweat test
  71. 71what is the test for CF?chloride sweat test
  72. 72what might you see on xray with bronchiectasis?> crowded bronchial markings yhat extend to the periphery
    >tram tracking
  73. 73what might you see on xray with bronchiectasis?> crowded bronchial markings yhat extend to the periphery
    >tram tracking
  74. 74what is the most common cause of massive haemoptysisbronchiectasis
  75. 75what is the most common cause of massive haemoptysisbronchiectasis
  76. 76what other nebulised drug do you give in acute asthma?ipratropium
  77. 77what other nebulised drug do you give in acute asthma?ipratropium
  78. 78what else can be given IV in acute asthma?magnesium sulphate
    aminophylline
  79. 79what else can be given IV in acute asthma?magnesium sulphate
    aminophylline

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Random resp questions in an osce
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