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Nervous 1+2

Aufia Z English 120 cards

Nervous 1+2

Cards in this deck (120)

  1. 1—Lobes of the brainLobes of the brain
  2. 2—Basic cellular components of CNSBasic cellular components of CNS
  3. 3—Neurons with round nucleus, prominent nucleolus, and nissl substance surrondingNeurons with round nucleus, prominent nucleolus, and nissl substance surronding
  4. 4—Large purkinje cells in the cerebellumLarge purkinje cells in the cerebellum
  5. 5—NeuromelaninNeuromelanin
  6. 6—Astrocytes with GFAP stainAstrocytes with GFAP stain
  7. 7—AstrocytesAstrocytes
  8. 8—OligodendrogliaOligodendroglia
  9. 9—OligodendrogliaOligodendroglia
  10. 10—Ependymal Cells
    Have both epithelial + glial component    Ependymal Cells Have both epithelial + glial component
  11. 11—Choroid Plexus
    Epithelial properties    Choroid Plexus Epithelial properties
  12. 12—Cerebrovascular territoriesCerebrovascular territories
  13. 13—Global Cerebral IschemiaGlobal Cerebral Ischemia
  14. 14—Ischemic Stroke SignsIschemic Stroke Signs
  15. 15—Ischemic stroke 12-24 hoursIschemic stroke 12-24 hours
  16. 16—Ischemic stroke 12-24 hoursIschemic stroke 12-24 hours
  17. 17—Ischemic stroke 24-48 hoursIschemic stroke 24-48 hours
  18. 18—Ischemic stroke 24-48 hoursIschemic stroke 24-48 hours
  19. 19—Ischemic stoke 2 days - 2 weeksIschemic stoke 2 days - 2 weeks
  20. 20—Ischemic stroke 2 days - 2 weeksIschemic stroke 2 days - 2 weeks
  21. 21—Ischemic stroke 3 weeks - monthsIschemic stroke 3 weeks - months
  22. 22—ischemic stroke 3 weeks - monthsischemic stroke 3 weeks - months
  23. 23—Hemorrhagic stroke: 20%Hemorrhagic stroke: 20%
  24. 24—Hemorrhagic stroke (20%)Hemorrhagic stroke (20%)
  25. 25—Intraparenchymal hemorrhageIntraparenchymal hemorrhage
  26. 26—Intraparenchymal hemorrhageIntraparenchymal hemorrhage
  27. 27—types of intracranial hemorrhagetypes of intracranial hemorrhage
  28. 28—Epidural Hemorrhage
    Trauma, lucid interval    Epidural Hemorrhage Trauma, lucid interval
  29. 29—Subdural Hemorrhage
    Rupture of bridging veins
    Trauma w/ old    Subdural Hemorrhage Rupture of bridging veins Trauma w/ old
  30. 30—Subarachnoid hemorrhage
    Berry aneurysms, mb trauma    Subarachnoid hemorrhage Berry aneurysms, mb trauma
  31. 31—Arteriolosclerosis --> occlusion --> lacunar infarct
    One of the two things hypertension causes
    (medial ischemic thickening)    Arteriolosclerosis --> occlusion --> lacunar infarct One of the two things hypertension causes (medial ischemic thickening)
  32. 32—Charcot Bouchard syndrome
    One of the two things hypertension causes
    (Basal ganglia feeder vessels weakening = rupture = intraparenchymal hemorrhage)    Charcot Bouchard syndrome One of the two things hypertension causes (Basal ganglia feeder vessels weakening = rupture = intraparenchymal ...
  33. 33—Holes in the basal ganglia
    Arise as a result of hypertension
    (charcot bouchard syndrome)    Holes in the basal ganglia Arise as a result of hypertension (charcot bouchard syndrome)
  34. 34—Acute bacterial meningitis
    Infection varies with Age
    Neonates = E.Coli, Group B Strep
    Infants = S. Pneumonia, H Influenza
    Adolescents = Neisseria Meningitis
    Elderly = S. Pneumonia, listeria    Acute bacterial meningitis Infection varies with Age Neonates = E.Coli, Group B Strep Infants = S. Pneumonia, H Influenza Adolescents = N...
  35. 35—Acute Bacterial Meningitis
    Causative Bacteria varies with age
    Infants = S. Pneumonia, Group B    Acute Bacterial Meningitis Causative Bacteria varies with age Infants = S. Pneumonia, Group B
  36. 36—Charcot Bouchard Syndrome
    One of two things hypertension causes
    Weakening of basal ganglia walls by little microaneuryms--> vessel rupture --> intraparenchymal hemorrhage    Charcot Bouchard Syndrome One of two things hypertension causes Weakening of basal ganglia walls by little microaneuryms--> vessel rup...
  37. 37—Hole in the basal ganglia as a result of Charcot Bouchard Syndrome
    One of the two things hypertension causes
    Hypertension --> weakens walls of basal ganglia feeder vessels --> microinfarcts --> eventul intraparenchymal hemorrhage    Hole in the basal ganglia as a result of Charcot Bouchard Syndrome One of the two things hypertension causes Hypertension --> weakens ...
  38. 38—Acute Bacterial Meningitis
    Causative Bacteria vary with Age
    Neonates = E.Coli, Group B strep
    Infants = Strep Pneumonia, H. Influenza
    Adolescents = Neisseria Meningitdis
    Elderly = Strep Pneumonia, Listeria    Acute Bacterial Meningitis Causative Bacteria vary with Age Neonates = E.Coli, Group B strep Infants = Strep Pneumonia, H. Influenza Adol...
  39. 39—Acute Bacterial Meningitis
    Causative bacteria vary with age
    Neonates = E.Coli, group B strep
    Infants = Strep Pneumonia, H. Influenza (:))
    Adolescents = Neisseria Meningitis
    Elderly = S. Pneumonia, Listeria    Acute Bacterial Meningitis Causative bacteria vary with age Neonates = E.Coli, group B strep Infants = Strep Pneumonia, H. Influenza (:))...
  40. 40—Acute Viral Meningitis
    Self limiting
    Most commonly due to enteroviruses [echoviruses, coxsackie virus, nonparalytic polio]    Acute Viral Meningitis Self limiting Most commonly due to enteroviruses [echoviruses, coxsackie virus, nonparalytic polio]
  41. 41—Acute Viral Meningitis
    Self limiting
    Caused by enteroviruses [echovirus, coxsackie virus, nonparalytic poliomyelitis]
    Microglia nodules = nuclei of microglia form nodules --> can see them!!!!    Acute Viral Meningitis Self limiting Caused by enteroviruses [echovirus, coxsackie virus, nonparalytic poliomyelitis] Microglia nodules =...
  42. 42—Viral Encephalitis: HSV
    Hemorrhagic temporal lobes, glassy nuclear inclusions    Viral Encephalitis: HSV Hemorrhagic temporal lobes, glassy nuclear inclusions
  43. 43—Viral Encephalitis = HSV
    Bitemporal lobe hemorrhagic
    Glassy nuclear inclusions    Viral Encephalitis = HSV Bitemporal lobe hemorrhagic Glassy nuclear inclusions
  44. 44—VIral Encephalitis: Rabies
    Neari bodies in Purkinje cells    VIral Encephalitis: Rabies Neari bodies in Purkinje cells
  45. 45—Viral Encephalitis: Measles
    Chronic = Subacute Sclerosing Panencephalitis --> may emerge years later
    Glassy inclusions like HSV, but multinucleated    Viral Encephalitis: Measles Chronic = Subacute Sclerosing Panencephalitis --> may emerge years later Glassy inclusions like HSV, but m...
  46. 46—Progressive Multifocal Leukoencephalopathy (PML)
    Myelinating disorder
    Common in immunocompromised people (organ transplant, HIV)
    JC virus --> PML    Progressive Multifocal Leukoencephalopathy (PML) Myelinating disorder Common in immunocompromised people (organ transplant, HIV) JC virus...
  47. 47—Abcess/Abscess Like Lesions
    Bacterial Abscess = "normal" multifocal with neutrophils    Abcess/Abscess Like Lesions Bacterial Abscess = "normal" multifocal with neutrophils
  48. 48—Abscess/Abscess like lesions
    Toxoplasmosis = encysted bradyzoite --> breaks out --> tachyzoite    Abscess/Abscess like lesions Toxoplasmosis = encysted bradyzoite --> breaks out --> tachyzoite
  49. 49—Abscess/Abscess like lesions
    Cryptococcus = Bubbly gross brain, space around blood vessels expands
    India Ink stain    Abscess/Abscess like lesions Cryptococcus = Bubbly gross brain, space around blood vessels expands India Ink stain
  50. 50—Creutzfeldt-Jakob Disease
    Chromosome 20, abnormal folding (now beta sheet)
    Rapid dementia, spongiform encephalopathy, amyloid plaques    Creutzfeldt-Jakob Disease Chromosome 20, abnormal folding (now beta sheet) Rapid dementia, spongiform encephalopathy, amyloid plaques
  51. 51—Contusion in the brain (bruise)
    Damage to crests of gyri (brain surface) most common    Contusion in the brain (bruise) Damage to crests of gyri (brain surface) most common
  52. 52—Contusion (bruise in brain)
    Commonly affects surface of gyri (crests)
    But deep white matter contusions can occur    Contusion (bruise in brain) Commonly affects surface of gyri (crests) But deep white matter contusions can occur
  53. 53—Cerebral Contusion
    (Bruise)
    Coup = bruise at site of impact
    Counter-Coup = bruise on opposite site (larger, inferior frontal + temporal lobe)    Cerebral Contusion (Bruise) Coup = bruise at site of impact Counter-Coup = bruise on opposite site (larger, inferior frontal + temporal l...
  54. 54—Cerebral Contusion (brain bruise)Cerebral Contusion (brain bruise)
  55. 55—Cerebral Contusion (brain bruise)
    Looks like petechial hemorrhage    Cerebral Contusion (brain bruise) Looks like petechial hemorrhage
  56. 56—Cerebral Contusion (brain bruise)
    Petechial hemorrhage
    "Chronic infarc with hemosiderin rust"    Cerebral Contusion (brain bruise) Petechial hemorrhage "Chronic infarc with hemosiderin rust"
  57. 57—Cerebral Contusion (bruise)
    Chronic infarc with hemosiderin rest
    Looks like petechial hemorrhage on outside of brain, subarachnoid hemorrhages common    Cerebral Contusion (bruise) Chronic infarc with hemosiderin rest Looks like petechial hemorrhage on outside of brain, subarachnoid hemorr...
  58. 58—Contusion
    Looks like petechial hemorrhage    Contusion Looks like petechial hemorrhage
  59. 59—Traumatic Axonal Injury
    Blunt force/inertia
    Common sites = Corpus callosum, dorsolateral brainstem, parasagittal cerebral white matter    Traumatic Axonal Injury Blunt force/inertia Common sites = Corpus callosum, dorsolateral brainstem, parasagittal cerebral white matter
  60. 60—Traumatic Axonal Injury
    Blunt force/Inertia
    Petechia = Capillaries rupture in same way as axons    Traumatic Axonal Injury Blunt force/Inertia Petechia = Capillaries rupture in same way as axons
  61. 61Traumatic Axonal InjuryDiffuse
    Does not require direct impact
    Requires rotational/angular acceleration --> sheer stress
  62. 62Contusions and lacerationsLocalized
    Brain impacting with skull
    Coup vs. Countercoup
  63. 63—Damage secondary to trauma
    Edema    Damage secondary to trauma Edema
  64. 64—Consequences of Edema
    Subfalcine herniation = less concerning    Consequences of Edema Subfalcine herniation = less concerning
  65. 65—Consequences of Edema
    Uncle/transtentorial herniation = push on brainstem, fatal
    Tonsillar herniation = out through foramen magnum, push on lower brainstem, fatal
    Central herniation = General edema, whole brain pushes down, pons hemorrhages    Consequences of Edema Uncle/transtentorial herniation = push on brainstem, fatal Tonsillar herniation = out through foramen magnum, push ...
  66. 66—Consequences of Edema
    Subfalcine hemorrhage = falx, least concerning
    Central herniation = whole brain pushes down, general edema, pons hemorrhage
    Transtentorial herniation = push on brainstem, fatal
    Tonsillar hemorrhage = out through foramen magnum, push on lower brainstem, fatal    Consequences of Edema Subfalcine hemorrhage = falx, least concerning Central herniation = whole brain pushes down, general edema, pons he...
  67. 67Demyelinating DisordersThink Multiple Sclerosis
    Usually acquired
    Myelin damage with axon preservation
  68. 68Multiple SclerosisDemyelinating disorder
    Autoimmune = HLA-DR
    W > M but no age predilection
  69. 69Multiple SclerosisRelapsing and remitting (symptoms come and go, myelin, demyelin, myelin....)
    Oligoclonal bands + plasma cells in CSF
  70. 70Multiple SclerosisRisk increases 15 fold when relative affected
    HLA DR haplotype
  71. 71—Multiple Sclerosis
    Cortex = Around the ventricles lesions
    Optic tract lesions (vision problems)    Multiple Sclerosis Cortex = Around the ventricles lesions Optic tract lesions (vision problems)
  72. 72—Multiple Sclerosis
    Glassy tan/gray lesions in brainstem    Multiple Sclerosis Glassy tan/gray lesions in brainstem
  73. 73—Multiple Sclerosis Active Plaque
    Hypercellular, many macrophages, maybe astrocytes
    Overall preservation of axons, myelin just breaking down    Multiple Sclerosis Active Plaque Hypercellular, many macrophages, maybe astrocytes Overall preservation of axons, myelin just breaking down
  74. 74—Multiple Sclerosis Active Plaque
    Parenchymal + perivascular lymphocytes
    Oligoclonal banding + plasma cells in CSF    Multiple Sclerosis Active Plaque Parenchymal + perivascular lymphocytes Oligoclonal banding + plasma cells in CSF
  75. 75—Multiple Sclerosis Inactive Plaque (GLIOSIS)
    Hypocellular, fewer macrophages
    Loss of myelin = loss of oligodendrocytes
    Prominent Gliosis = reactive astrocytes
    Loss of axons    Multiple Sclerosis Inactive Plaque (GLIOSIS) Hypocellular, fewer macrophages Loss of myelin = loss of oligodendrocytes Prominent Gliosis ...
  76. 76—Multiple Sclerosis Inactive Plaques
    Gliosis = Reactive Astrocytes
    Loss of mylein - loss of oligodendroycte = loss of axons
    Hypocellular, fewer macrophages + chronic inflammation    Multiple Sclerosis Inactive Plaques Gliosis = Reactive Astrocytes Loss of mylein - loss of oligodendroycte = loss of axons Hypocellular, ...
  77. 77Movement DisordersParkinsons (Hypo) + Huntingtons (Hyper)
  78. 78Dementia DisordersAlzheimers + Lewy Body Dementia
  79. 79Dementia OverviewNot a part of normal aging
    Progressive loss of cognitive function independent of the state of attention
    Causes = primary neurodegenerative diseases (LW + AD) but also infection, vascular, metabolic, etc.
  80. 80—Alzheimer's Disease
    Most common cause of dementia in the elderly
    Incidence increases with age
    Most causes sporadic (but tri 21 correlation)    Alzheimer's Disease Most common cause of dementia in the elderly Incidence increases with age Most causes sporadic (but tri 21 correlation)
  81. 81—Alzheimer's Disease
    Brain shrinks, making ventricles look bigger
    "Fake hydrocephalous"
    Tangles with Tau protein + amyloid plaques    Alzheimer's Disease Brain shrinks, making ventricles look bigger "Fake hydrocephalous" Tangles with Tau protein + amyloid plaques
  82. 82—Alzheimer's Disease
    Deposition of amyloid in blood vessels --> cerebral amyloid amylopathy --> lobar hemorrhage    Alzheimer's Disease Deposition of amyloid in blood vessels --> cerebral amyloid amylopathy --> lobar hemorrhage
  83. 83—Alzheimer's Disease
    Most common reason for dementia in elderly
    Mostly sporadic (tri 21 corr)
    Fake hydrocephalus = brain shrinks, making the ventricles look larger
    Amyloid plaques with Tau proteins
    Deposition of amyloid in blood vessels --> cerebral amyloid amylopathy --> lobar hemorrhage    Alzheimer's Disease Most common reason for dementia in elderly Mostly sporadic (tri 21 corr) Fake hydrocephalus = brain shrinks, making t...
  84. 84—Alzheimer's Disease
    Most common reason for dementia in the elderly
    Mostly sporadic (tri 21 corrl)
    Amyloid plaques + Tau Proteins
    Amyloid plaques deposit in blood vessels --> amyloid amylopathy --> lobal hemorrhage
    Brain shrinks, making ventricles look larger = Fake hydrocephalois    Alzheimer's Disease Most common reason for dementia in the elderly Mostly sporadic (tri 21 corrl) Amyloid plaques + Tau Proteins Amyloid ...
  85. 85Lewy Body DementiaProgressive decline + 2:
    -Fluctuating cognition (lucid intervals, allie in notebook)
    -Visual hallucinations
    -Spontaneous motor features of parkinsonism    Lewy Body Dementia
  86. 86—Lewy Body Dementia
    Progressive congnitive decline + 2:
    -Fluctuating cognition
    -Visual hallucinations
    -Motor features like parkinsons    Lewy Body Dementia Progressive congnitive decline + 2: -Fluctuating cognition -Visual hallucinations -Motor features like parkinsons
  87. 87—Lewy bodies in lewy body dementia
    in both cortex and brainstem
    Lewy bodies have alpha synuclein, so do a stain for this    Lewy bodies in lewy body dementia in both cortex and brainstem Lewy bodies have alpha synuclein, so do a stain for this
  88. 88—Lewy Body dementia
    Loss of pigment in substantia nigra like parkinsons
    Lewy bodies with alpha synnuclein
    Progressive Cognitive Declines + 2:
    -cognition fluctuation
    -visual hallucinations
    -motor problems like parkinsons    Lewy Body dementia Loss of pigment in substantia nigra like parkinsons Lewy bodies with alpha synnuclein Progressive Cognitive Declines +...
  89. 89—Clinically diagnosing lewy body dementia from parkinson's
    Lewy body = Movement disorder + dementia same time
    Parkinson's = Movement --> dementia (wait 5 years)    Clinically diagnosing lewy body dementia from parkinson's Lewy body = Movement disorder + dementia same time Parkinson's = Movement --&gt...
  90. 90Parkinson's Disease L-Dopa ResponsiveHypomovement disorder
    Tremor, rigidity, bradykinesia
    Autonomic + Cognitive Findings Uncommon
    Most sporadic, no gender
    Loss of pigmentation in substantia nigra
    Lewy bodies in brainstem only
  91. 91—Parkinson's Disease
    Hypokinetic Disorder
    Bradykinesia, Rigid, Tremor
    Loss of pigment in substantia nigra
    L Dopa Responsive
    Lewy bodies in brainstem only
    Sporadic, no gender etiology, incidence increases w/ age    Parkinson's Disease Hypokinetic Disorder Bradykinesia, Rigid, Tremor Loss of pigment in substantia nigra L Dopa Responsive Lewy bodies in...
  92. 92—Huntington's Disease
    Hyperkinetic, Heritable!!
    Autosomal dominant gene on chromosome 4p16 = protein huntingtin = CAG repeat expansion
    Anticipating/progressive = more repeats with further generations, worse symptoms
    Mean onset = 40
    Loss of caudate, then putamen
    Motor = chorea writhing movements
    Eventually psychiatric syndomes = Irritability, depression, anxitety    Huntington's Disease Hyperkinetic, Heritable!! Autosomal dominant gene on chromosome 4p16 = protein huntingtin = CAG repeat expansion Ant...
  93. 93—Amyotrophic Lateral Sclerosis
    Mean age of onset = 60
    Slightly more in males (1.5:1)
    Muscle atrophy + hyperreflexia
    death within 5 years --> respiratory failure or bronchopneumonia
    10-20% have frontal dementia
    If familial (rare) problem with superoxide dismutase on chromosome 21    Amyotrophic Lateral Sclerosis Mean age of onset = 60 Slightly more in males (1.5:1) Muscle atrophy + hyperreflexia death within 5 years -...
  94. 94—Amyotrophic lateral Sclerosis
    Muscle atrophy and hyperreflexia
    Age of onset is 60
    Males slightly more common than females (1.5:1)
    Death within 5 years commonly because of bronchopneumonia/respiratory failure
    (10-20% have frontal dementia)
    Anterior spinal root atrophy that is worse distally
    if familial, problem with superoxide dismutase enzyme (chromosome 21)    Amyotrophic lateral Sclerosis Muscle atrophy and hyperreflexia Age of onset is 60 Males slightly more common than females (1.5:1) Death w...
  95. 95—Pilocytic Astrocytoma
    WHO Grade 1
    Kids: Posterior fossa + cerebellum    Pilocytic Astrocytoma WHO Grade 1 Kids: Posterior fossa + cerebellum
  96. 96—Diffuse Astrocytoma
    WHO Grade 2
    Hypercellularity + Atypia    Diffuse Astrocytoma WHO Grade 2 Hypercellularity + Atypia
  97. 97—Anaplastic Astrocytoma
    WHO Grade 3
    Mitotic figures    Anaplastic Astrocytoma WHO Grade 3 Mitotic figures
  98. 98—Glioblastoma
    (Astrocytoma WHO grade 4)
    Pseudopalisading necrosis = necrosis + endothelial cell proliferation    Glioblastoma (Astrocytoma WHO grade 4) Pseudopalisading necrosis = necrosis + endothelial cell proliferation
  99. 99—Glioblastoma
    (Astrocytoma WHO grade 4)
    Pseudopalisading necrosis + vascular proliferation    Glioblastoma (Astrocytoma WHO grade 4) Pseudopalisading necrosis + vascular proliferation
  100. 100—Oligodendroglioma
    WHO Grade 2
    Chicken egg = thin capillary vasculature, perinuclear halo, round nuclei    Oligodendroglioma WHO Grade 2 Chicken egg = thin capillary vasculature, perinuclear halo, round nuclei
  101. 101—Oligodendroglioma
    WHO grade 2
    Chicken egg    Oligodendroglioma WHO grade 2 Chicken egg
  102. 102—Anaplastic oligodendroglioma (chicken egg)
    WHO grade 3
    Mitotic increase, necrosis, endothelial proliferation    Anaplastic oligodendroglioma (chicken egg) WHO grade 3 Mitotic increase, necrosis, endothelial proliferation
  103. 103—Anaplastic oligodendroglioma (chicken egg)
    WHO Grade 3    Anaplastic oligodendroglioma (chicken egg) WHO Grade 3
  104. 104—Ependymoma WHO grade 2
    Happens at ventricular surface (where ependymal cells line, supratentorial, posterior fossa, spinal....)
    Perivascular rosettes (internal vessels) with the occasional endymomal canal    Ependymoma WHO grade 2 Happens at ventricular surface (where ependymal cells line, supratentorial, posterior fossa, spinal....) Perivascu...
  105. 105—Ependymoma WHO grade 2
    Perivascular rosettes (internal vessels) with the occasional endymomal canal    Ependymoma WHO grade 2 Perivascular rosettes (internal vessels) with the occasional endymomal canal
  106. 106—Ependymoma Grade 2
    Perivascular rosette with the occasional ependymoma canal    Ependymoma Grade 2 Perivascular rosette with the occasional ependymoma canal
  107. 107—Ependymoma Grade 2
    Perivascular pseudorosettes with the occasional ependymoma canal    Ependymoma Grade 2 Perivascular pseudorosettes with the occasional ependymoma canal
  108. 108—Ependymoma
    WHO Grade 3    Ependymoma WHO Grade 3
  109. 109—Medulloblastoma
    70% under 16
    75% arise in vermis
    By definition, GRADE 4
    ---
    distinguish from ependymoma because rosettes don't have vessel in middle    Medulloblastoma 70% under 16 75% arise in vermis By definition, GRADE 4 --- distinguish from ependymoma because rosettes don't have vesse...
  110. 110—Medulloblastoma
    ----
    70% under 16
    75% in vermis
    By definition GRADE 4
    ---
    Different from ependymoma because rosettes dont have vessel in the middle    Medulloblastoma ---- 70% under 16 75% in vermis By definition GRADE 4 --- Different from ependymoma because rosettes dont have vessel in ...
  111. 111—Meningioma = Dura tumor
    dural tail
    Push brain parenchyma away
    psomma bodies    Meningioma = Dura tumor dural tail Push brain parenchyma away psomma bodies
  112. 112—Meningioma = Dura Tumor
    Psomma bodies
    Most grade 1 = if you have a brain tumor, this is the one you want to have!    Meningioma = Dura Tumor Psomma bodies Most grade 1 = if you have a brain tumor, this is the one you want to have!
  113. 113—Craniopharyngioma
    "machine oil"
    "cholesterol crystals"
    Before age 20 :(
    Supra sellar = Above the sella turcica    Craniopharyngioma "machine oil" "cholesterol crystals" Before age 20 :( Supra sellar = Above the sella turcica
  114. 114—Metastatic Tumors
    Well circumscribed, multifocal (multiple nodules), gray:white junction    Metastatic Tumors Well circumscribed, multifocal (multiple nodules), gray:white junction
  115. 115—Metastatic tumors
    Well circumscribed, multifocal (multiple), gray:white junction    Metastatic tumors Well circumscribed, multifocal (multiple), gray:white junction
  116. 116—Metastatic tumors
    Most common primary tumor sites = Lungs, breast, colon    Metastatic tumors Most common primary tumor sites = Lungs, breast, colon
  117. 117—Metastatic tumor
    Most common hemorrhagic metastasis = Renal, melanoma, choriocarcinoma    Metastatic tumor Most common hemorrhagic metastasis = Renal, melanoma, choriocarcinoma
  118. 118—
  119. 119—Schwannoma peripheral nerve tumor (WHO grade 1)
    Verochay bodies    Schwannoma peripheral nerve tumor (WHO grade 1) Verochay bodiesSchwannoma peripheral nerve tumor (WHO grade 1) Verochay bodies
  120. 120—Neurofibroma
    WHO grade 1
    Carrots    Neurofibroma WHO grade 1 Carrots

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