Biological treatment of Sz

megan langdon
Mind Map by megan langdon, updated more than 1 year ago
megan langdon
Created by megan langdon almost 5 years ago


Biological treatments of Schizophrenia

Resource summary

Biological treatment of Sz
1 History of Therapy for Sz
1.1 Biological treatments
1.1.1 1930's Pre - frontal lobotomy
1.1.2 1930's onwards - ECT
1.1.3 1950's - Typical anti - psychotics neuroleptics - antagonists
1.1.4 1990 - Atypical anti - psychotics
1.2 Psychological treatments
1.2.1 Not considered to have insight
1.2.2 Therapy done to them not actively involved
1.2.3 Now more responsive due to drugs & employ their own coping strategies to deal w/ pos symptoms - therapists tap into this
1.2.4 Now supportive, collaborative, active involvement
2 Psychosurgery
2.1 Brain damage to bring about behaviour change
2.2 Pre - frontal lobotomy (1930's)
2.2.1 Sever the connections from pre - frontal lobes to rest of brain
2.3 Patients become calmer & reduced / absent symptoms of Sz
2.4 Effectiveness
2.4.1 Whilst some studies may indicate effectiveness, it's like saying alcohol 'cures' anxiety & stress
2.5 Appropriate
2.5.1 Frontal lobe abnormalities in Sz
2.5.2 Surgery may help in severe cases to control symptoms of Sz
2.5.3 Irreversible - can brain damage be justified
2.5.4 Major side effects
3 Chemotherapy (drug treatment)
3.1 Typical antipsychotics
3.1.1 Chlorpromazine
3.1.2 Lower DA (block receptors) Antagonist
3.1.3 Only pos
3.2 Atypical antipsychotics
3.2.1 Clopazine
3.2.2 Temp occupy DA receptors
3.2.3 Also affect serotonin mood / attention / perception
3.2.4 Pos & Neg
3.3 Effectiveness AO2
3.3.1 Typical Relapse rates suggest they are effective Rzewuska 2002 Chlorpromazine helps 80 - 85% Kane 1992 Longer periods of usage needed (side effects) Little effect on neg symptoms
3.3.2 Atypical Pos & neg symptoms deLima (2005) Effect not much greater than typical anti - psychotics For both pos & neg symptoms
3.3.3 Allows treatment in the community for the maj
3.4 Appropriateness AO2
3.4.1 Typical Symptoms similar to Parkinson's disease Tardive Dyskenia (30%) Motivational problems (compliance)
3.4.2 Atypical Fewer side effects Clozapine Fatal immune damage - agranulocytosis
3.4.3 Treat symptoms not the cause
3.4.4 Relapse
3.4.5 Dependency & tolerance Diff people respond to diff drugs in diff ways
4 Aetiology Fallacy
4.1 Just because drugs work, doesn't mean biology is root cause
4.2 They may work in conjunction w/ other treatments / factors
5 Ethical implications AO2 / 3
5.1 Chemical strait jacket
5.2 Inhumane?
5.3 Reduce control & responsibility
5.4 Informed consent
5.5 Depot injections
5.6 Motivational interviewing
5.6.1 Miller & Rollnick 2002
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