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Notes

Secondary Aldosteronism - created from Mind Map

nurtuba98 English 1 page

Secondary Aldosteronism - created from Mind Map

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Page 1

Symptoms

Causes

Definition

Treatment

Aldosterone

Fatigue

Headache

Muscle weakness

Numbness

Paralysis

Hypertension

Hypokalemia

Hypernatraemia

Hypomagnesemia

K+ level in blood higher than

Essential to the function of nerve and muscle cells, including those in your heart.

K+ level is normally 3.6 to 5.2 millimoles per liter (mmol/L).

Juxtaglomerula Cell Tumor

Renal Artery Stenosis

Fibromuscular hyperplasia

Hyporeabsorption of sodium

Reduced blood supply across the juxtaglomerular apparatus stimulates the production of renin. Hepatic blood flow and aldosterone metabolism are reduced, so circulating levels of the hormone are high.

Juxtaglomerular apparatus

Structure formed by distal convoluted tubule & Glomerular afferent arteriole

Regulate blood pressure

Maintain filtration of glomerulus

Characterized by excessive secretion of aldosterone by adrenal glands, which causes increases in sodium reabsorption and loss of potassium (hypokalemia) and hydrogen ions.

Spironolactone

Potassium-sparing diuretic

Drugs

limiting salt intake

Regulate blood pressure

Increase reabsorption of ions and H2O in kidney

causing conversation of Na

Secretion of K+

Increase water retention

Secondary Aldosteronism - created from Mind Map

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Note on Secondary Aldosteronism - created from Mind Map, created by nurtuba98 on 10/10/2015.
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nurtuba98
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  • Created by nurtuba98 · Oct 10, 2015
  • Copied to Note by nurtuba98 · Oct 10, 2015
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