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Ostertagia and Teladorsagia

Caitlin Wringe 14 Fragen

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  1. 1Direct life cycle:
    Adult -- Egg --[blank_start]L1 (hatches from egg)[blank_end] -- [blank_start]L2[blank_end] -- [blank_start]L3 (infective)[blank_end] -- [blank_start]L4[blank_end] -- [blank_start]L5[blank_end]
    1. AL1 (hatches from egg)
    2. BL1 (maturation of egg)
    3. CL2
    4. DL2B
    5. EL3 (infective)
    6. FL1
    7. GL4
    8. HL5
    9. IL5
    10. JL4
  2. 2What is the prepatent period?
    1. A21 days
    2. B14 days
    3. C18 days
    4. D30 days
  3. 3What is unique about the L4 larval stage that aids in survival?
    1. AAbility to enter hypobiosis for several months
    2. BNutrient storage
    3. CRetained cuticle from L3
    4. DTemperature tolerant
  4. 4Once ingested infective L3 [blank_start]exsheaths[blank_end] in the [blank_start]rumen[blank_end] and migrates to the [blank_start]abomasum[blank_end] where it [blank_start]penetrates and encysts[blank_end] in the [blank_start]gastric glands[blank_end]. Bild zu Frage 4
    1. Aexsheaths
    2. Bpenetrates
    3. Crumen
    4. Dabomasum
    5. Eabomasum
    6. Frumen
    7. Gpenetrates and encysts
    8. Hexsheaths and penetrates
    9. Igastric glands
    10. Jgastric epithelial cells
  5. 5What larval stage emerges from the gastric glands and matures in the lumen of the abomasum?
    1. AL5
    2. BL4
    3. CL3
    4. DL2
  6. 6What are the direct impacts the parasite has on the abomasum?
    1. ADilation and erosion of gastric gland
    2. BImpingement on neighbouring cells
    3. CLoss of secretory cells
    4. DIncreases HCl production
    5. EKills/outcompetes other bacteria
  7. 7What are the consequences of the loss of parietal secretory cells (HCl secreting)
    1. AAbomasal pH increases
    2. BAbomasal pH decreases
    3. CBacterial populations decrease
    4. DDecreased protein utilisation
    5. EDecreased appetite
    6. FIncreased appetite
    7. GVomiting
  8. 8What is the consequence of an increased permeability?
    1. AMacromolecules diffuse into abomasum
    2. BPlasma pepsinogen increased
    3. CMacromolecules diffuse out of abomasum
    4. DDecreased plasma albumin concentrations
    5. EHyperproteinemia
    6. FHypoproteinemia
  9. 9An [blank_start]increased[blank_end] abomasa pH leads to;
    Pepsinogen not converted to [blank_start]pepsin[blank_end] - [blank_start]decreased[blank_end] protein utilisation
    [blank_start]Gastrin[blank_end] secretions [blank_start]increase[blank_end] - decreased gut motility and appetite
    Bacterial populations [blank_start]increase[blank_end]
    1. Aincreased
    2. Bdecreased
    3. Cpepsin
    4. DHCl
    5. Edecreased
    6. Fincreased
    7. GGastrin
    8. HHCl
    9. Iincrease
    10. Jdecrease
    11. Kincrease
    12. Ldecrease
  10. 10What are the main clinical signs of Ostertagiasis?
    1. ASevere watery green diarrhoea
    2. BInappetence
    3. CVomiting
    4. DOdema
    5. ECough
    6. FAnorexia

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Beschreibung
Ruminant Nematodes - Abomasal worms
Sprache
English
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