WCC VCE PE: Aerobic Chronic Adaptions

Description

These flashcards explain how each of the chronic adaptations to an aerobic training program will lead to improved performance in an athlete.
Chelsea B
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Tim Hodge
Created by Tim Hodge over 9 years ago
Chelsea B
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Resource summary

Question Answer
Increased pulmonary diffusion RESPIRATORY More O2 is diffused from the alveoli to the capillaries, resulting in more O2 being delivered to the working muscles
Increased Ventilation RESPIRATORY V=TVxRR Ventilation will increase due to an increase in Tidal Volume (volume per breath) The more air we breath in @ max intensity, the more opportunity we have to diffuse O2 into our blood stream and onto our working muscles
Decreased O2 cost to the ventilatory muscles RESPIRATORY The Intercostal muscles and Diaphragm are more efficient in the contractions and require less O2. More O2 can be utilised at the working muscles
Increased Stroke Volume CARDIOVASCULAR Results from increased volume of the left ventricle. Also cause your HR to be lower at rest. Leads to more blood being circulated around the body @max intensities, meaning more O2 being delivered to the working muscles
Faster recovering heart rate CARDIOVASCULAR When aerobically trained, an athlete will return to their resting heart rate faster, due to their improved ability to deliver O2 to the working muscles and the decreased reliance on the Anaerobic Glycolysis ES, resulting in the build-up of less Metabolic by-products
Capilarisation at the muscles (slow-twitch) CARDIOVASCULAR Capillary density will increase in the muscles, resulting in a greater surface area and more gaseous exchange occurring. (More O2 enters the muscles)
Increased Blood Volume CARDIOVASCULAR Due to an increase in RBC's and Plasma levels
Increased Red Blood Cell (RBC) count CARDIOVASCULAR This also means that there is more hemoglobin in the blood stream. This increases the O2 carrying capacity of the blood, resulting in more O2 being delivered to the working muscles
Decreased Blood Pressure @ rest & sub-max intensities CARDIOVASCULAR Results in long term health benefits, with less pressure being exerted on the artery walls
Increase in oxidative enzymes MUSCULAR Enzymes that aid in the more efficient breakdown of glycogen and fats when O2 is present
Increase in Myoglobin content/levels MUSCULAR Myoglobin carries O2 with the muscle cell. The more myoglobin present, the increased opportunity to breakdown food fuels aerobically
Increased Mitochondria size, number and surface area MUSCULAR Mitochondria are the power stations of the cells. The more we have, the greater our capacity is to break down fuels (CHO, Fats) to produce ATP
Increased a-vO2 difference MUSCULAR Our muscles have a greater capacity to diffuse more O2 from our caterpillaries (due to increase in myoglobin and mitochondria) , resulting in a greater difference in O2 concentration of the arteries and veins
Increased Triglyceride stores MUSCULAR More triglyceride (fat) stores, means that we have a greater potential to produce ATP.
Increased Glycogen stores MUSCULAR More glycogen (CHO) stores, means that we have a greater potential to produce ATP and maintain higher intensities within the Aerobic training zone (70-85% max HR)
Increased glycogen sparing MUSCULAR As our body becomes more efficient at delivering O2 to the working muscles, we will be able to burn more fats at sub-max intensities. This means our glycogen stores will remain higher, allowing us to work at higher intensities at a later stage in the event
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