Neuro-Cognitive Disorders

SaraJaarour
Mind Map by SaraJaarour, updated more than 1 year ago
SaraJaarour
Created by SaraJaarour over 4 years ago
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University Level Nursing - Psychiatry Mind Map on Neuro-Cognitive Disorders, created by SaraJaarour on 10/13/2015.
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Resource summary

Neuro-Cognitive Disorders
1 Overview

Annotations:

  • Primary deficit in cognitive functions: LMC
1.1 Shifting principles

Annotations:

  • Organic Mental Disorders Cognitive Disorders Neuro-cognitive Disorders
2 Cognitive Domains
2.1 DSM 5

Annotations:

  • Complex Attention Executive Function Language Perceptual motor problems Social Cognition
2.2 DSM 4

Annotations:

  • Memory impairment Aphasia Apraxia Agnosia Executive Function
3 Classes
3.1 Major NCD (Dementia)

Annotations:

  • Dementia Before for elderly after for younger people: HIV andTBI
3.1.1 Diagnostic Criteria

Annotations:

  • Severe Cognitive Decline Interfere with Independence Not due to 2
3.1.2 Types

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  • Alzheimer's Disease Frontotemporal Lobar Degeneration  Lewy Body Disease Vascular Disease Traumatic Brain Injury Substance/Medication HIV infection Prion Disease Parkinson's Disease Huntington Disease
3.2 Mild NCD
3.2.1 Diagnostic Criteria

Annotations:

  • Moderate Cognitive Decline Do not interefere with independence
3.3 Delirium

Annotations:

  • Absence of the criteria of delirium for Major/Minor NCD is called clear consciousness and can coexist.
  • Disruption of the homeostasis of the brain
3.3.1 Diagnostic Criteria

Annotations:

  • Disturbance in attention (direct,focus,sustain,shift) and awareness(orientation) MAY INCLUDE COGNITION (One at least): Memory deficit, disorientation, language, visuospatial ability, or perceptionDue to something else May resolve Development Acute: hours to few daysPersistent: Weeks to months
  • Sleep/wake cycle changes Emotional State changes Worsening of behavioral problems in the evening
3.3.2 Risk factors

Annotations:

  • Age, Cognitive/Sensory Impairment, Men, Severe illness, Hip fracture, Hyper/Hypo thermia, Hypotension, Malnutrition, Meds, Restraints, Metabolic (Hypo/Hyperglycemia, Azotemia), Depression, Alcoholism, and Pain
3.3.3 Etiologies

Annotations:

  • Organic 95% Multifactorial: Sensory Input, surroundings, and stressors DIMTOP
3.3.4 Types
3.3.4.1 Substance Intoxication
3.3.4.2 Substance Withdrawal
3.3.4.3 Medication Induced
3.3.4.4 Due to another Medical Condition
3.3.4.5 Due to multiple Etiologies
3.4 NCD (not specified)
4 Types and Etiologies
4.1 Major NCD
4.1.1 TBI

Annotations:

  • Neuroimaging is required same as minor
4.1.2 Parkinson
4.2 Minor NCD
4.2.1 TBI

Annotations:

  • Onset: known Loss of consciousnessDisorientationAmnesiaNeuroimaging not required
  • Recovery: Partial or complete Weeks to months
4.2.2 S/M Induced
4.2.2.1 Alcohol Abuse Abstinence 30-40%
4.2.2.2 COSA 1/3
4.3 LBD

Annotations:

  • Onset:Insidious Flactuating cognitiong alertness attention Visual hallucination Cognitive impairment-->1year-->Parkinsonian movements REM sleep disorder Neuroleptic sensitivity
4.3.1 Parkinson

Annotations:

  • Onset: Insidious Tremor, rigidity, bradykinesia, shuffling of posture and gate Dementia happens later on
  • substantia nigra in the brain stem  pigmented lesions called lewy bodies no dopamine is produced Major NCD
4.4 HIV

Annotations:

  • Subcortical Pattern Impaired EF Slowing of processing speed Attention learning new info NO PROBLEM in recalling learned information 25-5%
4.5 Prion Disease

Annotations:

  • Onset:Insidious Fatigue Anxiety Appetite/Sleep/concentration problems then after some weeks incoordenation, altered vision, abnormal gait, Rapidly progressive dementia, myoclonus, ataxia
4.5.1 Creutzfeldt-Jakob

Annotations:

  • Transmitted by corneal grafts, injected crude of growth hormone derived from human pituitary, infected electrodes
  • prion agent spongiform encephalopathy vacuolization of nerve cells
  • Confusion Depression Altered Sensation then after weeks to months Dementia ataxia palsy cortical blindness
4.6 Huntington

Annotations:

  • Onset:Insiduous EF (processing speed, organization, and planning) not learning and memory bradykinesia and chorea(kerking)
4.7 Vascular

Annotations:

  • PR: above 65 second most common complex attention and EF
  • due to large vessel stroke small vessel ischemic disease white matter ischemia
4.8 AD

Annotations:

  • lower beta amyloid protein deposition Elevated tau or phosphorelated  Positron Emission Tomography These appear years before the onset of NCD symptoms
  • Duration 10 years 3-20 years
4.8.1 Major

Annotations:

  • 2 or more cognitive domains impaired IADLS
4.8.1.1 Probable

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  • 1 of the following: Genetic mutation evidence or memory impairment  progressive gradual decline no other possible etiology
4.8.1.2 Possible

Annotations:

  • if none of probable occur
4.8.2 Minor

Annotations:

  • 1 or more cognitive domains IADLS intact
4.8.2.1 Probable

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  • AD gene evidence
4.8.2.2 Possible

Annotations:

  • no evidence of AD gene but: Decline in memory progresive gradual decline in cognition no evidence of sources (Vascular, Lewy, Parkinson)
5 Delirium vs. Dementia

Annotations:

  • Onset attention/short term memory Flactuation during the day Visual hallucinations MMSE clock draw
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