apoptosis ✒ =sdfsصدف however is - CRP in body *sometimes failure ot prd underine some disease -active p single c+intact +no inflammatory
physiological :-embryogenesis
pathological :- tumer *liver cells in hepatitis
necrosis:- DT due be.. limp * *always pathological +groups of C *inflammatory repair is common
coagulative N:- 1-c all dead butT.architecture 3faint⬅proteins.C ,TS by fibrosis 5-most organs and common
liquefactive:-N 1-brain 2-T liquefied و heal➡ by glial R
caseous :- 1-cheesy ..casein 2-D T is granular and structureless 3-tuberculosis
Gangrene:-N with putrefaction of T⬅ c.bacteria. -T appear black ⬅dis from D.HG.
fibrinoid.N:- smw.cf سمو كف -NT ➡fibrin in staining. P _Arterioles in case of Vasculitis malignant HT
fat N:- 1-traumatic:D trauma to AD.T▶ extracellular release fat as brast 2-enzymatic :-✂✂of fat ⬅lipase E with form -chalky w.spot like acute pancreatitis
sublethal : " cellular degeneration": *its a morphological cell changes *reversible *but irrversible d if j persistent
hydropic d: - c be pale +swollen etiology :- **H.CP** patho:- a..dmit f ↪Na by water ▶swelling
Ionizing. R:- 1-electro-: xو gamma ray 2-particulate:-A.B.N
mode: Ractive Radical :-Hو oh وh2o2 و O2then interact with m.Lو DNA -D d:- 1-cell death or mitosis 2-just reapair 3-p changein genotype *effect on t: dose radiosenstivity