Pathoana 1-2

Description

Medizin Flashcards on Pathoana 1-2, created by Elsa Frenzel on 29/10/2017.
Elsa Frenzel
Flashcards by Elsa Frenzel, updated more than 1 year ago
Elsa Frenzel
Created by Elsa Frenzel over 6 years ago
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Question Answer
interstinal metaplasia
helicobacter pylori infection
inflammation
inflammation
inflammation
acute pneumonia, (hyperemia, hemosiderin-laden macrophages?)
acute pneumonia
acute pneumonia
fungal cerebral abscess
cerebral fungal abscess
fungal cerebral abscess
stetanosis, necrotizing granuloma
stetanosis, necrotizing granuloma
stetanosis, necrotizing granuloma
necrotzing granuloma, caused by TB
Hyperplasia, increased number of epithelial cells, (breast),
hyperplasia, increased number of epithelial cells, (prostate)
kidney normal
not normal kidney, not so regular proliferation
lt: normal thyroid gland, rt: hyperplasia/hypertrophy of graves
hypertrophy of heart
neuropathic atrophy
hypertrophy of skeletal tissue
squamous metaplasia of respiratory epithelium
metaplasia, esophagus
fibrous tissue, osteoblasts in pink
dysplasia, normal lt, increased intensity on rt
dysplasia, basal layer, increased nuclei
dysplasia, lt normal, abnormal nuclei in upper layer
dysplasia, upper layer
?
?
?
?
nuclei are not in basal layer, became stratified, polymorphism, hyper chromatic, mitosis
lt: normal, rt: abnormal, squamous stratified epithelium
enlarged number of cells, layers are not distinctive from each other
?
transition to stratified epithelium, no keratinization, mitosis, invasion through basal membrane
high grade dysplasia
columnar cells, nuclei are stratified
only in basal layer
transition, other something abnormal
dysplasia?
dysplasia ?
very stratified, mitosis, atypical
stratified columnar epithelium, mitosis near lumen, hyper chromatic
gastroesophageal junction (barret´s esophagus)
lamina propria infiltrate with MMN, PMN, glandular epithelium with goblet cells (barred´s esophagus)
barret´s esophagus
1. reflux esophagitis, 2. intestinal metaplasia type III, 3. dysplasia, 4. adenocarnimona
dense arrangement of glands lined by a double layered epithelium, prostatic hyperplasia
hyperplastic glands with intraluminal papillary formations, prostatic hyperplasia
basal aller hyplerplasia
chronic gastritis
goblet cells in epithelium, chronic gastritis
ABPAS, chronic gastritis, intestinal metaplasia
metaplasia
squamous cell metaplasia endocervical glands
endometrial hyperplasia
endometrium with distorted, irregular and mystically dilated glands (endometrial hyperplasia)
dilated glands separated by stroma (endometrial hyperplasia)
glandular lining reminiscent of proliferative endometrial gland lining (endometrial hyperplasia)
?
?
?
differentiation and anaplasia
different sized and shaped - variation, hyper chromatic nuclei visible
?
chromatin, clump, melanoma
cytologic atypia, inclusions
two different sizes of nuclei
dots in nuclei - malignant
mitosis, increased mitotic cells
mitotic cells: atypical mitotic figures
mitotic cells: atypical mitotic figures
leiomyoma
normal endometrium
normal endometrium
endometrioid adenocarcinoma: well differentiated G1
endometrioid adenocarninoma: moderately differentiated G2
endometrioid adenocarcinoma: poorly differentiated G3
papillary urothelial (transitional cell) carcinoma
patterns
normal colonic mucosa
first recognizable manifestation of epithelial alteration (colorectal tumor)
adenoma, several layers of abnormal cells form on lamina proproa
invasive carcinoma
metastatis, malignant adenocarcinomas
a. normal, b. metaplasia -> squamous, c. dysplasia -> due to accumulation, d. dysplasia -> further maturation, e. full thickness reached, f. invasion
infiltrative growth: at wall of colon
lymphovascular invasion, can spread into lymph system
cytological atypia, increase nuclear size, involves full thickness of epithelium,
infiltrative arrangement of tubular/glandular structures, adenocarcinoma, anaplasia -> increased nuclear size, prominent nucleoli, slight variation in nuclear size and shape (prostate)
tubular/glandular structures state 3 gleans
cribriform structure, state 4 gleason
invasion into surrounding tissue, in prostate, extraprostatic extension
perineurial spread/invasion, growth/malignant behavior
endometrioid adencarcinoma, fragments are densely packed, glandular cribriform structures
endometrioid adenocarcinoma, tubular/glandular with single lumen
endometrioid adenocarcinoma, cribriform - one complex- multiple lumina
heterogeneity
endometrioid adenocarcinoma
endometrioid adenocarcinoma structures
endometrioid adenocarcinoma structures solid sheets - areas of cohesively growing tumor cells
infiltrative growth
retroperitoneal tumor 1, polymorphism
retroperitoneal tumor 2, well differentiated area, not that pronounced polymorphism
squamous cell carcinoma in situ, polymorphism, enlarged nuclei, multiple figures, loss of maturation, thickening of epithelium, changed throughout thickness of epithelium
infiltrative growth
architectural details, trabecular structures-thick (few cells in thickness) tumor cells strands
palisading-perpendicular arrangement of cells at the periphery of tumor complex, typical for basal cell carcinoma
benign papillary tumors in the urinary bladder are rare
minimal variation in nuclear size, minimal distortion of polarity
kidney, clear cell renal cell carcinoma, most common malignant tumor in kidney with copious clear cytoplasm
kidney, oncocytoma, most common benign tumor of the kidney, cells with eosinophilic cytoplasm
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