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PHSI3011 11-2 Liver in Metabolic Homeostasis

Usuário eliminado 31 perguntas Atualizado em 10 nov 2017

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1Glycolysis turns [blank_start]__________[blank_end] into [blank_start]__________[blank_end].
1. AGlucose-6-P
2. BPyruvate
3. CGlucose
4. DGlycogen
5. ELactate
6. FGlycerol
7. GAmino Acids
2Glycogenesis turns [blank_start]__________[blank_end] into [blank_start]__________[blank_end].
1. AGlucose-6-P
2. BPyruvate
3. CLactate
4. DGlucose
5. EGlycerol
6. FAmino Acids
7. GGlycogen
3Gluconeogenesis turns [blank_start]__________[blank_end] into [blank_start]__________[blank_end].
1. APyruvate
2. BGlucose-6-P
3. CGlucose
4. DGlycogen
5. EGlucagon
4Glycogenolysis turns [blank_start]__________[blank_end] into [blank_start]__________[blank_end].
1. AGlycogen
2. BGlucose-6-P
3. CPyruvate
4. DGlucose
5. ELactate
6. FGlycerol
7. GGlucagon
8. HAmino Acids
5The three rate-limiting steps of Gluconeogenesis are:
1. AGlucose 6-Phosphatase
2. BFructose 1,6-bisphosphatase
3. CPEP carboxykinase
4. DPyruvate kinase
5. EPhosphofructokinase-1
6. FHexokinase
6The three rate-limiting steps of Glycolysis are:
1. AGlucose 6-Phosphatase
2. BFructose 1,6-bisphosphatase
3. CPEP carboxykinase
4. DPyruvate kinase
5. EPhosphofructokinase-1
6. FHexokinase
7Which one is which: Glycolysis vs Gluconeogenesis
1. AGluconeogenesis
2. BGlycolysis
8Put the three rate-limiting steps (of each side) in the right places.
1. AGlucose 6-Phosphatase
2. BFructose 1,6-bisphosphatase
3. CPEP carboxykinase
4. DHexokinase
5. EPhosphofructokinase-1
6. FPyruvate kinase
9Does insulin STIMULATE or SUPPRESS glucose disposal and glycogen&lipid synthesis?
1. AStimulate
2. BSuppress
10Does insulin STIMULATE or SUPPRESS glucose production and VLDL secretion?
1. AStimulate
2. BSuppress

11What’s a LIRKO?
1. ALiver-specific Insulin Receptor KnockOut mouse
2. BLoss of Insulin Receptor KnockOut mouse
3. CLiver-specific Insulin Receptor Knockout Organism
4. DLoss of Insulin Receptor Knockout Organism
12What results in a LIRKO in regards to blood&serum glucose levels?
1. ABlood increase; serum increase
2. BBlood increase; serum decrease
3. CBlood decrease; serum increase
4. DBlood decrease; serum decrease
13Loss of PTEN expression in the liver results in increased Liver Triglyceride levels. What’s PTEN?
1. APhosphatase and TENsin homolog
2. BPhosphatase and TENsin ligand
3. CPhosphatase and TENsin binding factor
4. DPhosphatase and TENsin lipase
14Glucose is taken up in a GLUT2-dependent manner. It’s then stored as [blank_start]__________[blank_end] until blood glucose levels drop.
1. AGlycogen
2. BGlucose 6-Phosphate
3. CPyruvate
4. DGlucagon
5. EGlycerol
15The liver contributes to the homeostasis of circulating levels of which?
1. AFatty Acids
2. BCholesterol
16The liver is the main clearance tissue for removal of Fatty Acids from circulation, then repackages them in what?
1. AVLDL-TAG
2. BLDL-TAG
3. CHDL-TAG
4. DChylomicron-TAG
17Stages of Lipoprotein and FA uptake:
[blank_start]_____[blank_end] – [blank_start]__________[blank_end] resides on endothelial cells to hydrolyse VLDL-TAG to FAs for uptake
1. AStage 1
2. BStage 2
3. CStage 3
4. DHepatic lipase (and Lipoprotein lipase)
5. EPeriphery
6. FLiver
18Stages of Lipoprotein and FA uptake:
[blank_start]_____[blank_end] – [blank_start]__________[blank_end]: TAG-rich VLDL interacts with VLDL-R, enters lysosomal pathway for hydrolysis and TAG synthesis.
1. AStage 1
2. BStage 2
3. CStage 3
4. DHepatic lipase (and Lipoprotein lipase)
5. EPeriphery
6. FLiver
19Stages of Lipoprotein and FA uptake:
[blank_start]_____[blank_end] – [blank_start]__________[blank_end]: VLDL-remnants interact with a range of receptors for endocytosis.
1. AStage 1
2. BStage 2
3. CStage 3
4. DHepatic lipase (and Lipoprotein lipase)
5. EPeriphery
6. FLiver
20Fatty Acid metabolism. The major pathways are:
De novo lipogenesis: [blank_start]__________[blank_end]
TriAcylGlycerol metabolism: [blank_start]__________[blank_end]
β-Oxidation: [blank_start]__________[blank_end]
1. ASynthesis of fatty acids from non-lipid
2. BEsterification, Lipoprotein and Lipolysi
3. CFA-CoA >> acetyl-CoA
21Fatty Acid Metabolism: loss of Lipolysis-Stimulated Receptor (LSR) expression does what?
1. AIncrease in Serum TriGlyceride levels
2. BDecrease in Liver TriGlyceride levels
3. CDecrease in Serum TriGlyceride levels
4. DIncrease in Liver TriGlyceride levels
22Loss of CD36 in the liver leads to [blank_start]__________[blank_end] Plasma FA levels. Plasma FAs [blank_start]__________[blank_end] LPL activity. Therefore loss of CD36 [blank_start]__________[blank_end] Chylomicron/VLDL-TAG hydrolysis (breakdown). …Leading to [blank_start]__________[blank_end] of TAG in the circulation.
1. AIncreased
2. BUnaffected
3. CReduced
4. DInhibit
5. EStimulate
6. FReduces
7. GIncreases
8. HAccumulation
9. IShortage
23True or false: The Hepatokine ILGF stands for Insulin-Like Growth Factor.
1. AVerdadeiro
2. BFalso
24What's this bit called?
1. AGlycolysis
2. BGlycogenesis
3. CGluconeogenesis
4. DGlycogenolysis
25What's this bit called?
1. AGlycolysis
2. BGlycogenesis
3. CGluconeogenesis
4. DGlycogenolysis
26What's this bit called?
1. AGlycolysis
2. BGlycogenesis
3. CGluconeogenesis
4. DGlycogenolysis
27What's this bit called?
1. AGlycolysis
2. BGlycogenesis
3. CGluconeogenesis
4. DGlycogenolysis
28What could be here?
1. APyruvate
2. BGlycogen
29What could be here?
1. APyruvate
2. BGlycogen
30What could be here?
1. APyruvate
2. BGlycogen
31What could be here?
1. APyruvate
2. BGlycogen

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Descrição: PHSI3011 11-2 Liver in Metabolic Homeostasis
Autor: Usuário eliminado
Idioma: English
Visualizações: 22
Histórico: Criado em 5 nov 2017 · atualizado em 10 nov 2017
Etiquetas: phsi3011 hoy metabolism liver w11 physiology homeostasis o usuário eliminou as informações de disciplina
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PHSI3011 11-2 Liver in Metabolic Homeostasis

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